Judith Böhringer

887 total citations
21 papers, 526 citations indexed

About

Judith Böhringer is a scholar working on Physiology, Molecular Biology and Epidemiology. According to data from OpenAlex, Judith Böhringer has authored 21 papers receiving a total of 526 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Physiology, 9 papers in Molecular Biology and 8 papers in Epidemiology. Recurrent topics in Judith Böhringer's work include Lysosomal Storage Disorders Research (18 papers), Trypanosoma species research and implications (6 papers) and Cellular transport and secretion (5 papers). Judith Böhringer is often cited by papers focused on Lysosomal Storage Disorders Research (18 papers), Trypanosoma species research and implications (6 papers) and Cellular transport and secretion (5 papers). Judith Böhringer collaborates with scholars based in Germany, Netherlands and United States. Judith Böhringer's co-authors include Ingo Müller, Rupert Handgretinger, Friederike Gieseke, Massimo Dominici, Rita Bussolari, Ingeborg Krägeloh‐Mann, Samuel Groeschel, Christiane Kehrer, Lüdger Schöls and Michaela Döring and has published in prestigious journals such as Blood, Neurology and Journal of Medical Genetics.

In The Last Decade

Judith Böhringer

21 papers receiving 518 citations

Peers

Judith Böhringer
Olivia Lansinger United States
Nicola Pierobon United States
Grigorios Tsaknakis United Kingdom
Anthony J. Burand United States
Namrata Mastey United States
Judith Böhringer
Citations per year, relative to Judith Böhringer Judith Böhringer (= 1×) peers Mariko Yamaoka

Countries citing papers authored by Judith Böhringer

Since Specialization
Citations

This map shows the geographic impact of Judith Böhringer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Judith Böhringer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Judith Böhringer more than expected).

Fields of papers citing papers by Judith Böhringer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Judith Böhringer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Judith Böhringer. The network helps show where Judith Böhringer may publish in the future.

Co-authorship network of co-authors of Judith Böhringer

This figure shows the co-authorship network connecting the top 25 collaborators of Judith Böhringer. A scholar is included among the top collaborators of Judith Böhringer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Judith Böhringer. Judith Böhringer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kern, Jan Marco, Judith Böhringer, Dagmar Timmann, et al.. (2023). Clinical, Imaging, Genetic, and Disease Course Characteristics in Patients With GM2 Gangliosidosis. Neurology. 102(1). e207898–e207898. 2 indexed citations
2.
Böhringer, Judith, Samuel Groeschel, Christiane Kehrer, et al.. (2022). Hematopoietic Stem Cell Transplantation with Mesenchymal Stromal Cells in Children with Metachromatic Leukodystrophy. Stem Cells and Development. 31(7-8). 163–175. 8 indexed citations
3.
Hayer, Stefanie N., et al.. (2022). Chitotriosidase is a biomarker for adult‐onset leukoencephalopathy with axonal spheroids and pigmented glia. Annals of Clinical and Translational Neurology. 9(11). 1807–1812. 3 indexed citations
4.
Laugwitz, Lucia, Laimdota Zizmare, Claire Cannet, et al.. (2022). Identification of neurodegeneration indicators and disease progression in metachromatic leukodystrophy using quantitative NMR‐based urinary metabolomics. JIMD Reports. 63(2). 168–180. 5 indexed citations
5.
Antony, Justin S., Jun-Hoe Lee, Lukas Heumos, et al.. (2021). A Mutation-Agnostic Hematopoietic Stem Cell Gene Therapy for Metachromatic Leukodystrophy. The CRISPR Journal. 5(1). 66–79. 11 indexed citations
6.
Döring, Michaela, Christiane Kehrer, Judith Böhringer, et al.. (2020). Early clinical course after hematopoietic stem cell transplantation in children with juvenile metachromatic leukodystrophy. PubMed. 7(1). 12–12. 20 indexed citations
7.
Beck‐Wödl, Stefanie, Christiane Kehrer, K. Harzer, et al.. (2020). Long‐term disease course of two patients with multiple sulfatase deficiency differs from metachromatic leukodystrophy in a broad cohort. JIMD Reports. 58(1). 80–88. 5 indexed citations
9.
Kehrer, Christiane, Judith Böhringer, Stefanie Beck‐Wödl, et al.. (2020). Association of Age at Onset and First Symptoms With Disease Progression in Patients With Metachromatic Leukodystrophy. Neurology. 96(2). e255–e266. 42 indexed citations
10.
Kehrer, Christiane, Judith Böhringer, Stefanie Beck‐Wödl, et al.. (2019). Phenotypic variation between siblings with Metachromatic Leukodystrophy. Orphanet Journal of Rare Diseases. 14(1). 136–136. 25 indexed citations
11.
Bruchelt, Gernot, et al.. (2019). Measurement of recombinant human arylsulfatase A and leukocyte sulfatase activities by analytical isotachophoresis. Journal of Chromatography B. 1124. 109–113. 4 indexed citations
12.
Reichbauer, Jennifer, et al.. (2018). Generation of two iPSC lines derived from two unrelated patients with Gaucher disease. Stem Cell Research. 35. 101336–101336. 3 indexed citations
13.
Krägeloh-Mann, Inge, K. Harzer, Kevin Rostásy, et al.. (2017). Late onset Krabbe disease due to the new GALC p.Ala543Pro mutation, with intriguingly high residual GALC activity in vitro. European Journal of Paediatric Neurology. 21(3). 522–529. 5 indexed citations
14.
Böhringer, Judith, René Santer, Neele Schumacher, et al.. (2017). Enzymatic characterization of novel arylsulfatase A variants using human arylsulfatase A‐deficient immortalized mesenchymal stromal cells. Human Mutation. 38(11). 1511–1520. 20 indexed citations
15.
Röeben, Benjamin, Rebecca Schüle, Susanne Ruf, et al.. (2017). SERAC1 deficiency causes complicated HSP: evidence from a novel splice mutation in a large family. Journal of Medical Genetics. 55(1). 39–47. 22 indexed citations
16.
Kehrer, Christiane, et al.. (2017). Rare Variant of GM2 Gangliosidosis through Activator-Protein Deficiency. Neuropediatrics. 48(2). 127–130. 5 indexed citations
17.
Groeschel, Samuel, Jörn‐Sven Kühl, Annette Bley, et al.. (2016). Long-term Outcome of Allogeneic Hematopoietic Stem Cell Transplantation in Patients With Juvenile Metachromatic Leukodystrophy Compared With Nontransplanted Control Patients. JAMA Neurology. 73(9). 1133–1133. 90 indexed citations
18.
Groeschel, Samuel, Christine í Dali, Judith Böhringer, et al.. (2012). Cerebral gray and white matter changes and clinical course in metachromatic leukodystrophy. Neurology. 79(16). 1662–1670. 33 indexed citations
19.
Gieseke, Friederike, Judith Böhringer, Rita Bussolari, et al.. (2010). Human multipotent mesenchymal stromal cells use galectin-1 to inhibit immune effector cells. Blood. 116(19). 3770–3779. 211 indexed citations
20.
Böhringer, Judith, B. Kustermann‐Kuhn, Friederike Gieseke, et al.. (2010). Hematopoietic Stem Cell Therapy In Eight Patients with Metachromatic Leukodystrophy – Relevance of Post-Transplant Medication.. Blood. 116(21). 3727–3727. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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