Claus‐Dieter Langhans
- Molecular Biology
- Biochemistry top 2%
- Clinical Biochemistry top 2%
- Physiology
- Nutrition and Dietetics top 10%
- Co-authors
- Olaf SommerburgWerner SiemsJürgen G. OkunIngrid WiswedelWolfgang AugustinLorenz SchildGeorg F. HoffmannC Crifò
- Topics
- Metabolism and Genetic Disorders (14 papers)Antioxidant Activity and Oxidative Stress (10 papers)Retinoids in leukemia and cellular processes (7 papers)
- Journals
- PLoS ONEHepatologyThe FASEB Journal
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
Claus‐Dieter Langhans
38 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 112
- Molecular Biology 556
- Biochemistry 260
- Clinical Biochemistry 155
- Physiology 117
- Nutrition and Dietetics 106
Countries citing papers authored by Claus‐Dieter Langhans
This map shows the geographic impact of Claus‐Dieter Langhans's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Claus‐Dieter Langhans with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Claus‐Dieter Langhans more than expected).
Fields of papers citing papers by Claus‐Dieter Langhans
This network shows the impact of papers produced by Claus‐Dieter Langhans. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Claus‐Dieter Langhans. The network helps show where Claus‐Dieter Langhans may publish in the future.
Co-authorship network of co-authors of Claus‐Dieter Langhans
This figure shows the co-authorship network connecting the top 25 collaborators of Claus‐Dieter Langhans. A scholar is included among the top collaborators of Claus‐Dieter Langhans based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Claus‐Dieter Langhans. Claus‐Dieter Langhans is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 7 | |
| 3 | 8 | |
| 4 | 23 | |
| 5 | 44 | |
| 6 | 13 | |
| 7 | 20 | |
| 8 | 63 | |
| 9 | 43 | |
| 10 | 4 | |
| 11 | 22 | |
| 12 | 106 | |
| 13 | 22 | |
| 14 | 7 | |
| 15 | 21 | |
| 16 | 72 | |
| 17 | 44 | |
| 18 | 34 | |
| 19 | 3 | |
| 20 | 38 |
About Claus‐Dieter Langhans
Claus‐Dieter Langhans is a scholar working on Clinical Biochemistry, Biochemistry and Nutrition and Dietetics, having authored 38 papers that have together received 1.1k indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (14 papers), Antioxidant Activity and Oxidative Stress (10 papers) and Retinoids in leukemia and cellular processes (7 papers). The work is most often cited by research in Biochemistry (260 citations), Clinical Biochemistry (155 citations) and Molecular Biology (556 citations). Claus‐Dieter Langhans has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Olaf Sommerburg, Werner Siems, Jürgen G. Okun, Ingrid Wiswedel, Wolfgang Augustin, Lorenz Schild, Georg F. Hoffmann, C Crifò, C. Salerno and D. Rating. Their work appears in journals such as PLoS ONE, Hepatology and The FASEB Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.