Jude M. Mulligan

1.0k total citations
14 papers, 555 citations indexed

About

Jude M. Mulligan is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Jude M. Mulligan has authored 14 papers receiving a total of 555 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 5 papers in Oncology and 3 papers in Pathology and Forensic Medicine. Recurrent topics in Jude M. Mulligan's work include BRCA gene mutations in cancer (3 papers), Genetic factors in colorectal cancer (3 papers) and Colorectal Cancer Treatments and Studies (3 papers). Jude M. Mulligan is often cited by papers focused on BRCA gene mutations in cancer (3 papers), Genetic factors in colorectal cancer (3 papers) and Colorectal Cancer Treatments and Studies (3 papers). Jude M. Mulligan collaborates with scholars based in United Kingdom, Ireland and United States. Jude M. Mulligan's co-authors include Patrick G. Johnston, D. Paul Harkin, Paul B. Mullan, Julia J. Gorski, D. Paul Harkin, Colin R. James, Jennifer E. Quinn, Gail E. Stewart, R. H. Wilson and Alison M. Hosey and has published in prestigious journals such as Nucleic Acids Research, Journal of Clinical Oncology and JNCI Journal of the National Cancer Institute.

In The Last Decade

Jude M. Mulligan

14 papers receiving 544 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jude M. Mulligan United Kingdom	9	346	200	176	148	76	14	555
Samantha E. Boyle Australia	11	406 1.2×	156 0.8×	143 0.8×	224 1.5×	75 1.0×	11	611
Julia J. Gorski United Kingdom	15	732 2.1×	312 1.6×	270 1.5×	212 1.4×	28 0.4×	20	931
Marc Payton United States	10	429 1.2×	269 1.3×	138 0.8×	112 0.8×	15 0.2×	17	613
Yong Xian United States	9	473 1.4×	210 1.1×	302 1.7×	76 0.5×	63 0.8×	9	726
Philip C. Schouten Netherlands	12	325 0.9×	326 1.6×	169 1.0×	243 1.6×	94 1.2×	26	649
Curtis Balch United States	13	714 2.1×	93 0.5×	59 0.3×	283 1.9×	66 0.9×	14	836
Terry Hurst Australia	11	276 0.8×	157 0.8×	57 0.3×	63 0.4×	202 2.7×	18	477
Andrea Milić Canada	9	275 0.8×	221 1.1×	124 0.7×	71 0.5×	24 0.3×	10	557
Huaizeng Chen China	16	514 1.5×	280 1.4×	42 0.2×	165 1.1×	124 1.6×	44	788
Patricia F. van Kuijk Netherlands	10	580 1.7×	60 0.3×	101 0.6×	450 3.0×	50 0.7×	11	769

Countries citing papers authored by Jude M. Mulligan

Since Specialization

Citations

This map shows the geographic impact of Jude M. Mulligan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jude M. Mulligan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jude M. Mulligan more than expected).

Fields of papers citing papers by Jude M. Mulligan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jude M. Mulligan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jude M. Mulligan. The network helps show where Jude M. Mulligan may publish in the future.

Co-authorship network of co-authors of Jude M. Mulligan

This figure shows the co-authorship network connecting the top 25 collaborators of Jude M. Mulligan. A scholar is included among the top collaborators of Jude M. Mulligan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jude M. Mulligan. Jude M. Mulligan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Niedzwiecki, Donna, Wendy L. Frankel, Alan P. Venook, et al.. (2016). Association Between Results of a Gene Expression Signature Assay and Recurrence-Free Interval in Patients With Stage II Colon Cancer in Cancer and Leukemia Group B 9581 (Alliance). Journal of Clinical Oncology. 34(25). 3047–3053. 35 indexed citations

Huang, Shuguang, Dakun Wang, Tianhua Wang, et al.. (2015). Analytical Performance of a 15-Gene Prognostic Assay for Early-Stage Non–Small-Cell Lung Carcinoma Using RNA-Stabilized Tissue. Journal of Molecular Diagnostics. 17(4). 438–445. 9 indexed citations

Huang, Shuguang, et al.. (2014). Analytical Performance of a Formalin-fixed Paraffin-embedded Tissue-based 634-probe Prognostic Assay for Predicting Outcome of Patients With Stage II Colon Cancer. Applied immunohistochemistry & molecular morphology. 22(4). 308–316. 2 indexed citations

Niedzwiecki, Donna, Wendy L. Frankel, Alan P. Venook, et al.. (2014). Association between ColDx assay result and recurrence-free interval in stage II colon cancer patients on CALGB (Alliance) 9581.. Journal of Clinical Oncology. 32(3_suppl). 455–455. 5 indexed citations

Hill, Laura A., Jude M. Mulligan, Steve Deharo, et al.. (2013). DNA damage response deficiency signature predicts response to platinum-based therapy in ovarian cancer. Research Portal (Queen's University Belfast). 49(2). 1 indexed citations

Gorski, Julia J., Kienan I. Savage, Jude M. Mulligan, et al.. (2011). Profiling of the BRCA1 transcriptome through microarray and ChIP-chip analysis. Nucleic Acids Research. 39(22). 9536–9548. 39 indexed citations

Gourley, Charlie, Caroline O. Michie, Katherine E. Keating, et al.. (2011). Establishing a molecular taxonomy for epithelial ovarian cancer (EOC) from 363 formalin-fixed paraffin embedded (FFPE) specimens.. Journal of Clinical Oncology. 29(15_suppl). 5000–5000. 5 indexed citations

Oliver, Gavin R., Vadim Farztdinov, Richard D. Kennedy, et al.. (2008). Generation of a non-small cell lung cancer transcriptome microarray. BMC Medical Genomics. 1(1). 20–20. 18 indexed citations

Quinn, Jennifer E., Colin R. James, Gail E. Stewart, et al.. (2007). BRCA1 mRNA Expression Levels Predict for Overall Survival in Ovarian Cancer after Chemotherapy. Clinical Cancer Research. 13(24). 7413–7420. 160 indexed citations

10.

Hosey, Alison M., Julia J. Gorski, J. E. Quinn, et al.. (2007). Molecular Basis for Estrogen Receptor Deficiency in BRCA1-Linked Breast Cancer. JNCI Journal of the National Cancer Institute. 99(22). 1683–1694. 159 indexed citations

11.

Buckley, Niamh E., Alison M. Hosey, Julia J. Gorski, et al.. (2007). BRCA1 Regulates IFN-γ Signaling through a Mechanism Involving the Type I IFNs. Molecular Cancer Research. 5(3). 261–270. 45 indexed citations

12.

Mulligan, Jude M., Lisa M. Greene, Suzanne M. Cloonan, et al.. (2006). Identification of Tubulin as the Molecular Target of Proapoptotic Pyrrolo-1,5-benzoxazepines. Molecular Pharmacology. 70(1). 60–70. 52 indexed citations

13.

Greene, Lisa M., Marina N. Fleeton, Jude M. Mulligan, et al.. (2005). The pyrrolo-1,5-benzoxazepine, PBOX-6, inhibits the growth of breast cancer cells in vitro independent of estrogen receptor status and inhibits breast tumour growth in vivo. Oncology Reports. 14(5). 1357–63. 18 indexed citations

14.

Mulligan, Jude M., Giuseppe Campiani, Anna Ramunno, Vito Nacci, & Daniela M. Zisterer. (2003). Inhibition of G1 cyclin-dependent kinase activity during growth arrest of human astrocytoma cells by the pyrrolo-1,5-benzoxazepine, PBOX-21. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1639(1). 43–52. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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