Federico Innocenti

15.8k total citations · 2 hit papers
211 papers, 7.8k citations indexed

About

Federico Innocenti is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, Federico Innocenti has authored 211 papers receiving a total of 7.8k indexed citations (citations by other indexed papers that have themselves been cited), including 127 papers in Oncology, 101 papers in Molecular Biology and 45 papers in Cancer Research. Recurrent topics in Federico Innocenti's work include Cancer therapeutics and mechanisms (59 papers), Colorectal Cancer Treatments and Studies (55 papers) and Pharmacogenetics and Drug Metabolism (33 papers). Federico Innocenti is often cited by papers focused on Cancer therapeutics and mechanisms (59 papers), Colorectal Cancer Treatments and Studies (55 papers) and Pharmacogenetics and Drug Metabolism (33 papers). Federico Innocenti collaborates with scholars based in United States, Italy and Japan. Federico Innocenti's co-authors include Mark J. Ratain, Jacqueline Ramı́rez, Donna Niedzwiecki, Soma Das, Richard M. Goldberg, Robert J. Mayer, Lalitha Iyer, Richard L. Schilsky, Heinz‐Josef Lenz and Alan P. Venook and has published in prestigious journals such as Nature Communications, Journal of Clinical Oncology and Blood.

In The Last Decade

Federico Innocenti

204 papers receiving 7.7k citations

Hit Papers

Genetic Variants in the UDP-glucuronosyltransferase 1A1 G... 2004 2026 2011 2018 2004 2010 200 400 600

Peers

Federico Innocenti
Comparison fields: 5 of 130
  • Oncology 4.9k
  • Molecular Biology 3.3k
  • Pulmonary and Respiratory Medicine 1.5k
  • Cancer Research 1.2k
  • Pharmacology 1.1k
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Citations per field, relative to Federico Innocenti
Federico Innocenti · 1×
Citations per year, relative to Federico Innocenti
Federico Innocenti · 1×

Countries citing papers authored by Federico Innocenti

Since Specialization
Citations

This map shows the geographic impact of Federico Innocenti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Federico Innocenti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Federico Innocenti more than expected).

Fields of papers citing papers by Federico Innocenti

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Federico Innocenti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Federico Innocenti. The network helps show where Federico Innocenti may publish in the future.

Co-authorship network of co-authors of Federico Innocenti

This figure shows the co-authorship network connecting the top 25 collaborators of Federico Innocenti. A scholar is included among the top collaborators of Federico Innocenti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Federico Innocenti. Federico Innocenti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 0
2 1
3 1
4 2
5 5
6 24
7 25
8 40
9 6
10 13
11 15
12 8
13 4
14 44
15 31
16 65
17 134
18
UGT1A1 genotyping in patients undergoing treatment with irinotecan.
11
19
Irinotecan treatment in cancer patients with UGT1A1 polymorphisms.
39
20
Correspondence re: Raida, M. et al., Prevalence of a Common Point Mutation in the Dihydropyrimidine Dehydrogenase (DPD) Gene within the 5-Splice Donor Site of Intron 14 in Patients with Severe 5-Fluorouracil (5-FU)-related Toxicity Compared with Controls. Clin. Cancer Res., 7: 2832-2839, 2001.
14

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026