Juana Gonzalez

5.3k total citations · 1 hit paper
45 papers, 3.6k citations indexed

About

Juana Gonzalez is a scholar working on Immunology, Dermatology and Physiology. According to data from OpenAlex, Juana Gonzalez has authored 45 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Immunology, 27 papers in Dermatology and 11 papers in Physiology. Recurrent topics in Juana Gonzalez's work include Dermatology and Skin Diseases (20 papers), Asthma and respiratory diseases (11 papers) and Psoriasis: Treatment and Pathogenesis (10 papers). Juana Gonzalez is often cited by papers focused on Dermatology and Skin Diseases (20 papers), Asthma and respiratory diseases (11 papers) and Psoriasis: Treatment and Pathogenesis (10 papers). Juana Gonzalez collaborates with scholars based in United States, Germany and Denmark. Juana Gonzalez's co-authors include James G. Krueger, Mayte Suárez‐Fariñas, Emma Guttman‐Yassky, Judilyn Fuentes‐Duculan, Michelle A. Lowes, Lisa C. Zaba, Katherine C. Pierson, Inna Novitskaya, Kristine Nograles and Xiuzhong Zheng and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Blood and The Journal of Immunology.

In The Last Decade

Juana Gonzalez

45 papers receiving 3.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Th17 cytokines interleukin (IL)-17 and IL-22 modulate distinct inflammatory and keratinocyte-response pathways

2008 665 citations Kristine Nograles, Lisa C. Zaba et al. British Journal of Dermatology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Juana Gonzalez United States	28	2.1k	2.1k	925	868	497	45	3.6k
Mary Sullivan‐Whalen United States	25	2.1k 1.0×	1.8k 0.9×	784 0.8×	742 0.9×	329 0.7×	32	3.3k
Kristine Nograles United States	20	3.1k 1.5×	3.3k 1.6×	1.1k 1.2×	1.3k 1.5×	678 1.4×	36	5.0k
Patricia Gilleaudeau United States	34	2.9k 1.4×	3.3k 1.6×	950 1.0×	1.0k 1.2×	502 1.0×	50	5.2k
Inna Novitskaya United States	14	1.6k 0.7×	2.5k 1.2×	340 0.4×	691 0.8×	310 0.6×	14	3.1k
Toyoko Kikuchi United States	25	1.7k 0.8×	3.1k 1.5×	377 0.4×	794 0.9×	379 0.8×	34	4.2k
Claire Q. Wang United States	18	1.1k 0.5×	834 0.4×	674 0.7×	498 0.6×	276 0.6×	27	2.2k
Jörg C. Prinz Germany	30	1.8k 0.8×	3.2k 1.6×	221 0.2×	722 0.8×	544 1.1×	64	4.0k
Artemis Khatcherian United States	12	1.2k 0.6×	1.7k 0.8×	280 0.3×	507 0.6×	231 0.5×	13	2.3k
Luis F. Santamaria‐Babí Spain	27	839 0.4×	1.0k 0.5×	441 0.5×	353 0.4×	260 0.5×	68	1.9k
Peter Korošec Slovenia	32	564 0.3×	790 0.4×	1.6k 1.7×	742 0.9×	341 0.7×	154	3.2k

Countries citing papers authored by Juana Gonzalez

Since Specialization

Citations

This map shows the geographic impact of Juana Gonzalez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Juana Gonzalez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Juana Gonzalez more than expected).

Fields of papers citing papers by Juana Gonzalez

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Juana Gonzalez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Juana Gonzalez. The network helps show where Juana Gonzalez may publish in the future.

Co-authorship network of co-authors of Juana Gonzalez

This figure shows the co-authorship network connecting the top 25 collaborators of Juana Gonzalez. A scholar is included among the top collaborators of Juana Gonzalez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Juana Gonzalez. Juana Gonzalez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Renert‐Yuval, Yael, et al.. (2024). Eosinophils in hidradenitis suppurativa patients exhibit pro‐inflammatory traits, implicating a potential pathogenic role in the disease. Experimental Dermatology. 33(7). e15129–e15129. 1 indexed citations

Miura, Shunsuke, Yohei Ichimura, Uri Sela, et al.. (2023). Staphylococcus Aureus and Streptococcus Pyogenes Induce Psoriasis-Related Transcriptomes Augmented by IL-17A and TNF-α. Journal of Investigative Dermatology. 143(8). 1613–1617.e5. 9 indexed citations

Wang, Claire Q., Sokol Haxhinasto, Sandra Garcet, et al.. (2022). Comparison of the Inflammatory Circuits in Psoriasis Vulgaris, Non‒Pustular Palmoplantar Psoriasis, and Palmoplantar Pustular Psoriasis. Journal of Investigative Dermatology. 143(1). 87–97.e14. 12 indexed citations

Miura, Shunsuke, Sandra Garcet, Xuan Li, et al.. (2022). Cathelicidin Antimicrobial Peptide LL37 Induces Toll-Like Receptor 8 and Amplifies IL-36γ and IL-17C in Human Keratinocytes. Journal of Investigative Dermatology. 143(5). 832–841.e4. 21 indexed citations

Miura, Shunsuke, Sandra Garcet, Juana Gonzalez, et al.. (2021). IL-36 and IL-17A Cooperatively Induce a Psoriasis-Like Gene Expression Response in Human Keratinocytes. Journal of Investigative Dermatology. 141(8). 2086–2090. 14 indexed citations

Navrazhina, Kristina, Sandra Garcet, Juana Gonzalez, et al.. (2021). In-Depth Analysis of the Hidradenitis Suppurativa Serum Proteome Identifies Distinct Inflammatory Subtypes. Journal of Investigative Dermatology. 141(9). 2197–2207. 42 indexed citations

Zhou, Lisa, Alexandra Leonard, Ana B. Pavel, et al.. (2019). Age-specific changes in the molecular phenotype of patients with moderate-to-severe atopic dermatitis. Journal of Allergy and Clinical Immunology. 144(1). 144–156. 106 indexed citations

Bissonnette, Robert, François Harel, James G. Krueger, et al.. (2017). TNF-α Antagonist and Vascular Inflammation in Patients with Psoriasis Vulgaris: A Randomized Placebo-Controlled Study. Journal of Investigative Dermatology. 137(8). 1638–1645. 74 indexed citations

Brunner, Patrick M., Mayte Suárez‐Fariñas, Helen He, et al.. (2017). The atopic dermatitis blood signature is characterized by increases in inflammatory and cardiovascular risk proteins. Scientific Reports. 7(1). 8707–8707. 193 indexed citations

10.

Czarnowicki, Tali, Hitokazu Esaki, Juana Gonzalez, et al.. (2016). Alterations in B-cell subsets in pediatric patients with early atopic dermatitis. Journal of Allergy and Clinical Immunology. 140(1). 134–144.e9. 47 indexed citations

11.

Ungar, Benjamin, Sandra Garcet, Juana Gonzalez, et al.. (2016). An Integrated Model of Atopic Dermatitis Biomarkers Highlights the Systemic Nature of the Disease. Journal of Investigative Dermatology. 137(3). 603–613. 167 indexed citations

12.

Czarnowicki, Tali, Hitokazu Esaki, Juana Gonzalez, et al.. (2015). Early pediatric atopic dermatitis shows only a cutaneous lymphocyte antigen (CLA)+ TH2/TH1 cell imbalance, whereas adults acquire CLA+ TH22/TC22 cell subsets. Journal of Allergy and Clinical Immunology. 136(4). 941–951.e3. 171 indexed citations

13.

Ohmatsu, Hanako, Daniel Humme, Nicholas Gulati, et al.. (2014). IL32 Is Progressively Expressed in Mycosis Fungoides Independent of Helper T-cell 2 and Helper T-cell 9 Polarization. Cancer Immunology Research. 2(9). 890–900. 17 indexed citations

14.

Jabbari, Ali, Mayte Suárez‐Fariñas, Judilyn Fuentes‐Duculan, et al.. (2014). Dominant Th1 and Minimal Th17 Skewing in Discoid Lupus Revealed by Transcriptomic Comparison with Psoriasis. Journal of Investigative Dermatology. 134(6). 1780–1780. 4 indexed citations

15.

Mitsui, Hiroshi, Claire Q. Wang, Mayte Suárez‐Fariñas, et al.. (2013). Identification of anaplastic lymphoma kinase as a potential therapeutic target in Basal Cell Carcinoma. Oncotarget. 4(12). 2237–2248. 17 indexed citations

16.

Gonzalez, Juana, Jamie L. Harden, Leanne M. Johnson-Huang, et al.. (2013). TREM-1 as a Potential Therapeutic Target in Psoriasis. Journal of Investigative Dermatology. 133(7). 1742–1751. 40 indexed citations

17.

Fujita, Hideki, Mayte Suárez‐Fariñas, Hiroshi Mitsui, et al.. (2012). Langerhans Cells from Human Cutaneous Squamous Cell Carcinoma Induce Strong Type 1 Immunity. Journal of Investigative Dermatology. 132(6). 1645–1655. 36 indexed citations

18.

Zhang, Yu, et al.. (2010). Raloxifene Modulates Estrogen-mediated B Cell Autoreactivity in NZB/W F1 Mice. The Journal of Rheumatology. 37(8). 1646–1657. 15 indexed citations

19.

Zaba, Lisa C., Judilyn Fuentes‐Duculan, Narat J. Eungdamrong, et al.. (2008). Psoriasis Is Characterized by Accumulation of Immunostimulatory and Th1/Th17 Cell-Polarizing Myeloid Dendritic Cells. Journal of Investigative Dermatology. 129(1). 79–88. 347 indexed citations

20.

Nograles, Kristine, Lisa C. Zaba, Emma Guttman‐Yassky, et al.. (2008). Th17 cytokines interleukin (IL)-17 and IL-22 modulate distinct inflammatory and keratinocyte-response pathways. British Journal of Dermatology. 159(5). ???–???. 665 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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