Joshua A. Boyce

18.5k total citations · 1 hit paper
162 papers, 8.5k citations indexed

About

Joshua A. Boyce is a scholar working on Physiology, Immunology and Surgery. According to data from OpenAlex, Joshua A. Boyce has authored 162 papers receiving a total of 8.5k indexed citations (citations by other indexed papers that have themselves been cited), including 104 papers in Physiology, 101 papers in Immunology and 29 papers in Surgery. Recurrent topics in Joshua A. Boyce's work include Asthma and respiratory diseases (100 papers), Mast cells and histamine (62 papers) and IL-33, ST2, and ILC Pathways (33 papers). Joshua A. Boyce is often cited by papers focused on Asthma and respiratory diseases (100 papers), Mast cells and histamine (62 papers) and IL-33, ST2, and ILC Pathways (33 papers). Joshua A. Boyce collaborates with scholars based in United States, Japan and Italy. Joshua A. Boyce's co-authors include K. Frank Austen, Tanya M. Laidlaw, Yoshihide Kanaoka, Chunli Feng, Michael F. Gurish, Daniel S. Friend, Katherine N. Cahill, Howard R. Katz, Wei Xing and Akiko Maekawa and has published in prestigious journals such as Nature, New England Journal of Medicine and Proceedings of the National Academy of Sciences.

In The Last Decade

Joshua A. Boyce

160 papers receiving 8.3k citations

Hit Papers

Allergic inflammatory memory in human respiratory epithel... 2018 2026 2020 2023 2018 100 200 300

Peers

Joshua A. Boyce
TH Lee United Kingdom
Stephen I. Wasserman United States
Gregory P. Geba United States
Anne Kagey‐Sobotka United States
Lauren Cohn United States
Christoph Walker Switzerland
A B Kay United States
Brian O’Connor United Kingdom
TH Lee United Kingdom
Joshua A. Boyce
Citations per year, relative to Joshua A. Boyce Joshua A. Boyce (= 1×) peers TH Lee

Countries citing papers authored by Joshua A. Boyce

Since Specialization
Citations

This map shows the geographic impact of Joshua A. Boyce's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joshua A. Boyce with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joshua A. Boyce more than expected).

Fields of papers citing papers by Joshua A. Boyce

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joshua A. Boyce. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joshua A. Boyce. The network helps show where Joshua A. Boyce may publish in the future.

Co-authorship network of co-authors of Joshua A. Boyce

This figure shows the co-authorship network connecting the top 25 collaborators of Joshua A. Boyce. A scholar is included among the top collaborators of Joshua A. Boyce based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joshua A. Boyce. Joshua A. Boyce is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Derakhshan, Tahereh, Alexander Perniss, Jonathan Hacker, et al.. (2025). Human intraepithelial mast cell differentiation and effector function are directed by TGF-β signaling. Journal of Clinical Investigation. 135(1). 4 indexed citations
2.
Joulia, Régis, Laura Yates, Simone A. Walker, et al.. (2025). A single-cell spatial chart of the airway wall reveals proinflammatory cellular ecosystems and their interactions in health and asthma. Nature Immunology. 26(6). 920–933. 9 indexed citations
3.
Ualiyeva, Saltanat, Alexander Perniss, Alice Z. Maxfield, et al.. (2024). A nasal cell atlas reveals heterogeneity of tuft cells and their role in directing olfactory stem cell proliferation. Science Immunology. 9(92). eabq4341–eabq4341. 14 indexed citations
4.
Boyce, Joshua A., et al.. (2024). Cysteinyl Leukotrienes in Allergic Inflammation. Annual Review of Pathology Mechanisms of Disease. 20(1). 115–141. 9 indexed citations
5.
Foer, Dinah, Taneem Amin, Jun Nagai, et al.. (2023). Glucagon-like Peptide-1 Receptor Pathway Attenuates Platelet Activation in Aspirin-Exacerbated Respiratory Disease. The Journal of Immunology. 211(12). 1806–1813. 8 indexed citations
6.
Derakhshan, Tahereh, Joshua A. Boyce, & Daniel F. Dwyer. (2022). Defining mast cell differentiation and heterogeneity through single-cell transcriptomics analysis. Journal of Allergy and Clinical Immunology. 150(4). 739–747. 38 indexed citations
7.
Ualiyeva, Saltanat, Juying Lai, Tao Liu, et al.. (2021). Tuft cell–produced cysteinyl leukotrienes and IL-25 synergistically initiate lung type 2 inflammation. Science Immunology. 6(66). eabj0474–eabj0474. 66 indexed citations
8.
Baek, Seung Han, Dinah Foer, Katherine N. Cahill, et al.. (2021). Systems Approaches to Treatment Response to Imatinib in Severe Asthma: A Pilot Study. Journal of Personalized Medicine. 11(4). 240–240. 5 indexed citations
9.
Liu, Tao, Nora A. Barrett, Jun Nagai, et al.. (2020). Leukotriene D4 paradoxically limits LTC4-driven platelet activation and lung immunopathology. Journal of Allergy and Clinical Immunology. 148(1). 195–208.e5. 9 indexed citations
10.
Derakhshan, Tahereh, Sachin K. Samuchiwal, Lora G. Bankova, et al.. (2020). Lineage-specific regulation of inducible and constitutive mast cells in allergic airway inflammation. The Journal of Experimental Medicine. 218(1). 50 indexed citations
11.
Nagai, Jun, Barbara Balestrieri, Laura B. Fanning, et al.. (2019). P2Y6 Signaling Controls an Innate Alveolar Macrophage-NK Cell Axis That Dampens Type 2 Lung Immunopathology. Journal of Allergy and Clinical Immunology. 143(2). AB293–AB293. 1 indexed citations
12.
Liu, Tao, Nora A. Barrett, Yoshihide Kanaoka, et al.. (2017). Type 2 Cysteinyl Leukotriene Receptors Drive IL-33–Dependent Type 2 Immunopathology and Aspirin Sensitivity. The Journal of Immunology. 200(3). 915–927. 58 indexed citations
13.
Laidlaw, Tanya M., Neil Bhattacharyya, Wei Xing, et al.. (2012). Cysteinyl leukotriene overproduction in aspirin-exacerbated respiratory disease is driven by platelet-adherent leukocytes. Blood. 119(16). 3790–3798. 195 indexed citations
14.
Paruchuri, Sailaja, Hiroyuki Tashimo, Chunli Feng, et al.. (2009). Leukotriene E4–induced pulmonary inflammation is mediated by the P2Y12 receptor. The Journal of Experimental Medicine. 206(11). 2543–2555. 201 indexed citations
15.
Abonia, J. Pablo, Jenny Hallgren, T. G. Jones, et al.. (2006). Alpha-4 integrins and VCAM-1, but not MAdCAM-1, are essential for recruitment of mast cell progenitors to the inflamed lung. Blood. 108(5). 1588–1594. 131 indexed citations
16.
Lin, Debby A. & Joshua A. Boyce. (2006). Lysophospholipids as Mediators of Immunity. Advances in immunology. 89. 141–167. 35 indexed citations
17.
Price, Kursteen S., et al.. (2004). Lysophosphatidic acid accelerates the development of human mast cells. Blood. 104(13). 4080–4087. 67 indexed citations
18.
Jameson, Sean C., Blythe Thomson, Margaret H. Collins, et al.. (2004). Anti–interleukin-5 (mepolizumab) therapy for hypereosinophilic syndromes. Journal of Allergy and Clinical Immunology. 113(1). 115–119. 282 indexed citations
19.
Lora, José M., Amal Al‐Garawi, Michael D. Pickard, et al.. (2003). FcεRI-dependent gene expression in human mast cells is differentially controlled by T helper type 2 cytokines. Journal of Allergy and Clinical Immunology. 112(6). 1119–1126. 20 indexed citations
20.
Silverman, Eric S., George T. De Sanctis, Joshua A. Boyce, et al.. (2001). The Transcription Factor Early Growth-response Factor 1 Modulates Tumor Necrosis Factor- α, Immunoglobulin E, and Airway Responsiveness in Mice. American Journal of Respiratory and Critical Care Medicine. 163(3). 778–785. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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