Barbara Balestrieri
- Molecular Biology
- Immunology top 10%
- Physiology top 10%
- Surgery
- Genetics
- Co-authors
- P. JonathanYoshihide KanaokaJoshua A. BoyceAkiko MaekawaWei XingSachin K. SamuchiwalFrancescopaolo GranataMassimo Triggiani
- Topics
- Asthma and respiratory diseases (11 papers)Protein Kinase Regulation and GTPase Signaling (7 papers)IL-33, ST2, and ILC Pathways (6 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryJournal of Clinical Investigation
- Partner nations
- United StatesItalyJapan
In The Last Decade
Barbara Balestrieri
35 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 90
- Molecular Biology 577
- Immunology 411
- Physiology 272
- Surgery 135
- Genetics 128
Countries citing papers authored by Barbara Balestrieri
This map shows the geographic impact of Barbara Balestrieri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Barbara Balestrieri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Barbara Balestrieri more than expected).
Fields of papers citing papers by Barbara Balestrieri
This network shows the impact of papers produced by Barbara Balestrieri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Barbara Balestrieri. The network helps show where Barbara Balestrieri may publish in the future.
Co-authorship network of co-authors of Barbara Balestrieri
This figure shows the co-authorship network connecting the top 25 collaborators of Barbara Balestrieri. A scholar is included among the top collaborators of Barbara Balestrieri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Barbara Balestrieri. Barbara Balestrieri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 9 | |
| 2 | 2 | |
| 3 | 3 | |
| 4 | 17 | |
| 5 | 1 | |
| 6 | 19 | |
| 7 | 40 | |
| 8 | 37 | |
| 9 | 1 | |
| 10 | 6 | |
| 11 | 44 | |
| 12 | 113 | |
| 13 | 65 | |
| 14 | 26 | |
| 15 | 16 | |
| 16 | 52 | |
| 17 | 42 | |
| 18 | 49 | |
| 19 | 49 | |
| 20 | 4 |
About Barbara Balestrieri
Barbara Balestrieri is a scholar working on Immunology, Physiology and Pharmacology, having authored 35 papers that have together received 1.2k indexed citations. Recurring topics across this work include Asthma and respiratory diseases (11 papers), Protein Kinase Regulation and GTPase Signaling (7 papers) and IL-33, ST2, and ILC Pathways (6 papers). The work is most often cited by research in Immunology (411 citations), Physiology (63 citations) and Immunology and Allergy (81 citations). Barbara Balestrieri has collaborated with scholars based in United States, Italy and Japan. Frequent co-authors include P. Jonathan, Yoshihide Kanaoka, Joshua A. Boyce, Akiko Maekawa, Wei Xing, Sachin K. Samuchiwal, Francescopaolo Granata, Massimo Triggiani, Giorgio Giannattasio and K. Frank Austen. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.