Joseph Saliba

562 total citations
6 papers, 354 citations indexed

About

Joseph Saliba is a scholar working on Genetics, Molecular Biology and Hematology. According to data from OpenAlex, Joseph Saliba has authored 6 papers receiving a total of 354 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Genetics, 4 papers in Molecular Biology and 3 papers in Hematology. Recurrent topics in Joseph Saliba's work include Myeloproliferative Neoplasms: Diagnosis and Treatment (5 papers), Kruppel-like factors research (3 papers) and Chronic Myeloid Leukemia Treatments (2 papers). Joseph Saliba is often cited by papers focused on Myeloproliferative Neoplasms: Diagnosis and Treatment (5 papers), Kruppel-like factors research (3 papers) and Chronic Myeloid Leukemia Treatments (2 papers). Joseph Saliba collaborates with scholars based in France, Belgium and Austria. Joseph Saliba's co-authors include Gérald Massonnet, Christine Chomienne, Jean-Paul Beressi, Benjamin Manéglier, Els Verhoeyen, Stany Chrétien, Francis Relouzat, Philippe Rousselot, Philippe Leboulch and Victoria Raggueneau and has published in prestigious journals such as Nature, Journal of Clinical Investigation and Blood.

In The Last Decade

Joseph Saliba

6 papers receiving 353 citations

Peers

Countries citing papers authored by Joseph Saliba

Since Specialization

Citations

This map shows the geographic impact of Joseph Saliba's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph Saliba with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph Saliba more than expected).

Fields of papers citing papers by Joseph Saliba

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph Saliba. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph Saliba. The network helps show where Joseph Saliba may publish in the future.

Co-authorship network of co-authors of Joseph Saliba

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph Saliba. A scholar is included among the top collaborators of Joseph Saliba based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph Saliba. Joseph Saliba is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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6 of 6 papers shown

#	Work	Indexed citations
1	PPARγ agonists promote the resolution of myelofibrosis in preclinical models 2021 ·Journal of Clinical Investigation ·Juliette Lambert, Joseph Saliba, Carolina Calderón, Karine Sii-Felice, Mohammad Salma, Valérie Edmond, Jean‐Claude Alvarez, Marc Delord, Caroline Marty, Isabelle Plo, Jean‐Jacques Kiladjian, Éric Soler, William Vainchenker, Jean‐Luc Villeval, Philippe Rousselot, Stéphane Prost	6
2	TET2 haploinsufficiency alters reprogramming into induced pluripotent stem cells 2020 ·Stem Cell Research ·(unknown), (unknown), Antonio Di Stefano, Martín Bonamino, Joseph Saliba, Keisuke Kataoka, Stevens K. Rehen, Hana Raslová, Caroline Marty, Seishi Ogawa, William Vainchenker, Bárbara da Costa Reis Monte-Mór, Isabelle Plo	6
3	Erosion of the chronic myeloid leukaemia stem cell pool by PPARγ agonists 2015 ·Nature ·Stéphane Prost, Francis Relouzat, Marc Spentchian, (unknown), Joseph Saliba, Gérald Massonnet, Jean-Paul Beressi, Els Verhoeyen, Victoria Raggueneau, Benjamin Manéglier, Sylvie Castaigné, Christine Chomienne, Stany Chrétien, Philippe Rousselot, Philippe Leboulch	218
4	Germ-line JAK2 mutations in the kinase domain are responsible for hereditary thrombocytosis and are resistant to JAK2 and HSP90 inhibitors 2014 ·Blood ·Caroline Marty, Cécile Saint‐Martin, C. Pecquet, (unknown), Joseph Saliba, Céline Mouton, Emilie Leroy, Ashot S. Harutyunyan, (unknown), Rémi Favier, Aurélie Toussaint, Éric Solary, Róbert Královics, Stefan N. Constantinescu, A Najman, William Vainchenker, Isabelle Plo, Christine Bellanné‐Chantelot	55
5	Heterozygous and Homozygous JAK2V617F States Modeled by Induced Pluripotent Stem Cells from Myeloproliferative Neoplasm Patients 2013 ·PLoS ONE ·Joseph Saliba, Sofiane Hamidi, Gaëlle Lenglet, Thierry Langlois, (unknown), Xénia Cabagnols, (unknown), Céline Legrand, Anne Galy, (unknown), B Benyahia, Éric Solary, Olivier Bernard, Longyun Chen, Najet Debili, Hana Raslová, Françoise Norol, William Vainchenker, Isabelle Plo, Antonio Di Stefano	28
6	Germline JAK2 Mutations In The Kinase Domain Are Responsible For Hereditary Thrombocytosis and Are Resistant To JAK2 and HSP90 Inhibitors 2013 ·Blood ·Caroline Marty, Cécile Saint‐Martin, C. Pecquet, (unknown), Joseph Saliba, Céline Mouton, Emilie Leroy, Ashot S. Harutyunyan, Jean‐François Abgrall, (unknown), Éric Solary, Róbert Královics, Stefan N. Constantinescu, A Najman, William Vainchenker, Isabelle Plo, Christine Bellanné‐Chantelot	41

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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