Hajime Akada

561 total citations
12 papers, 437 citations indexed

About

Hajime Akada is a scholar working on Genetics, Rheumatology and Molecular Biology. According to data from OpenAlex, Hajime Akada has authored 12 papers receiving a total of 437 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Genetics, 7 papers in Rheumatology and 5 papers in Molecular Biology. Recurrent topics in Hajime Akada's work include Myeloproliferative Neoplasms: Diagnosis and Treatment (11 papers), Eosinophilic Disorders and Syndromes (7 papers) and Cytokine Signaling Pathways and Interactions (5 papers). Hajime Akada is often cited by papers focused on Myeloproliferative Neoplasms: Diagnosis and Treatment (11 papers), Eosinophilic Disorders and Syndromes (7 papers) and Cytokine Signaling Pathways and Interactions (5 papers). Hajime Akada collaborates with scholars based in United States. Hajime Akada's co-authors include R. Hutchison, Golam Mohi, Dongqing Yan, M. Golam Mohi, Haiying Zou, Steven Fiering, Yue Yang, Kazuhito Sakamoto, Kay‐Uwe Wagner and Ajeet Gajra and has published in prestigious journals such as Blood, Stem Cells and Leukemia.

In The Last Decade

Hajime Akada

12 papers receiving 436 citations

Peers

Countries citing papers authored by Hajime Akada

Since Specialization

Citations

This map shows the geographic impact of Hajime Akada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hajime Akada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hajime Akada more than expected).

Fields of papers citing papers by Hajime Akada

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hajime Akada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hajime Akada. The network helps show where Hajime Akada may publish in the future.

Co-authorship network of co-authors of Hajime Akada

This figure shows the co-authorship network connecting the top 25 collaborators of Hajime Akada. A scholar is included among the top collaborators of Hajime Akada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hajime Akada. Hajime Akada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

#	Work	Indexed citations
1	Loss of Ezh2 cooperates with Jak2V617F in the development of myelofibrosis in a mouse model of myeloproliferative neoplasm 2016 ·Blood ·Yue Yang, Hajime Akada, (unknown), R. Hutchison, Golam Mohi	66
2	Coordinate Expansion of Murine Hematopoietic and Mesenchymal Stem Cell Compartments by SHIPi 2015 ·Stem Cells ·(unknown), Sonia Iyer, Hajime Akada, Sudha Neelam, Christopher M. Russo, John D. Chisholm, William G. Kerr	21
3	Loss of wild-type Jak2 allele enhances myeloid cell expansion and accelerates myelofibrosis in Jak2V617F knock-in mice 2014 ·Leukemia ·Hajime Akada, Shinji Akada, R. Hutchison, Golam Mohi	11
4	Critical Role of Jak2 in the Maintenance and Function of Adult Hematopoietic Stem Cells 2014 ·Stem Cells ·Hajime Akada, (unknown), R. Hutchison, Kazuhito Sakamoto, Kay‐Uwe Wagner, Golam Mohi	67
5	Loss of EZH2 Inhibits Erythropoiesis and Accelerates the Development of Myelofibrosis in Jak2V617F Knock-in Mice 2014 ·Blood ·Yue Yang, Hajime Akada, (unknown), R. Hutchison, Golam Mohi	1
6	Critical Role Of Jak2 In The Maintenance and Function Of Adult Hematopoietic Stem Cells 2013 ·Blood ·Hajime Akada, (unknown), Golam Mohi	3
7	Erythroid lineage-restricted expression of Jak2V617F is sufficient to induce a myeloproliferative disease in mice 2012 ·Haematologica ·Hajime Akada, (unknown), R. Hutchison, Golam Mohi	4
8	Loss of Wild-Type Jak2 Allele Enhances Myeloid Cell Expansion and Accelerates Myelofibrosis in Jak2V617F Knock-in Mice 2012 ·Blood ·Hajime Akada, (unknown), Dongqing Yan, R. Hutchison, Golam Mohi	4
9	Efficacy of vorinostat in a murine model of polycythemia vera 2012 ·Blood ·Hajime Akada, (unknown), Ajeet Gajra, (unknown), Stephen L. Graziano, R. Hutchison, Golam Mohi	50
10	Conditional expression of heterozygous or homozygous Jak2V617F from its endogenous promoter induces a polycythemia vera–like disease 2010 ·Blood ·Hajime Akada, Dongqing Yan, Haiying Zou, Steven Fiering, R. Hutchison, M. Golam Mohi	195
11	Efficacy of Vorinostat In a Murine Model of Polycythemia Vera 2010 ·Blood ·Hajime Akada, (unknown), M. Golam Mohi	4
12	Conditional Expression of Heterozygous or Homozygous Jak2V617F From Its Endogenous Promoter Induces a Polycythemia Vera-Like Disease. 2009 ·Blood ·Hajime Akada, Dongqing Yan, Haiying Zou, R. Hutchison, M. Golam Mohi	11

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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