Joseph D. Etlinger

3.6k total citations · 1 hit paper
58 papers, 3.0k citations indexed

About

Joseph D. Etlinger is a scholar working on Molecular Biology, Cell Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Joseph D. Etlinger has authored 58 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 19 papers in Cell Biology and 12 papers in Pathology and Forensic Medicine. Recurrent topics in Joseph D. Etlinger's work include Spinal Cord Injury Research (12 papers), Muscle Physiology and Disorders (11 papers) and Muscle metabolism and nutrition (9 papers). Joseph D. Etlinger is often cited by papers focused on Spinal Cord Injury Research (12 papers), Muscle Physiology and Disorders (11 papers) and Muscle metabolism and nutrition (9 papers). Joseph D. Etlinger collaborates with scholars based in United States, Italy and Israel. Joseph D. Etlinger's co-authors include Alfred L. Goldberg, Richard J. Zeman, Donald A. Fischman, David F. Goldspink, Charles K. Jablecki, Gary Guo, Pravin B. Sehgal, R Zak, Hong Peng and Kenneth M. Lerea and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Joseph D. Etlinger

58 papers receiving 2.8k citations

Hit Papers

A soluble ATP-dependent proteolytic system responsible fo... 1977 2026 1993 2009 1977 100 200 300 400

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Joseph D. Etlinger United States 32 1.7k 969 503 391 367 58 3.0k
Hideo Sugita Japan 34 2.7k 1.6× 879 0.9× 751 1.5× 244 0.6× 155 0.4× 132 3.7k
Douglas J. Mahoney Canada 27 2.2k 1.3× 763 0.8× 609 1.2× 637 1.6× 76 0.2× 64 3.9k
Simon S. Wing Canada 38 3.4k 2.0× 1.1k 1.2× 815 1.6× 616 1.6× 68 0.2× 71 4.8k
Lynn A. Megeney Canada 33 3.6k 2.1× 677 0.7× 684 1.4× 348 0.9× 52 0.1× 66 4.4k
Paul C. Holland Canada 35 2.4k 1.4× 412 0.4× 425 0.8× 279 0.7× 58 0.2× 79 3.3k
R Zak United States 36 2.4k 1.4× 687 0.7× 372 0.7× 82 0.2× 260 0.7× 71 3.7k
Isabelle Richard France 38 4.4k 2.6× 1.6k 1.6× 596 1.2× 149 0.4× 99 0.3× 148 5.4k
Chia‐Ling Tu United States 37 1.8k 1.1× 602 0.6× 412 0.8× 359 0.9× 20 0.1× 69 3.7k
Bronwyn A. Evans Australia 36 1.8k 1.1× 157 0.2× 745 1.5× 316 0.8× 207 0.6× 82 3.4k

Countries citing papers authored by Joseph D. Etlinger

Since Specialization
Citations

This map shows the geographic impact of Joseph D. Etlinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph D. Etlinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph D. Etlinger more than expected).

Fields of papers citing papers by Joseph D. Etlinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph D. Etlinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph D. Etlinger. The network helps show where Joseph D. Etlinger may publish in the future.

Co-authorship network of co-authors of Joseph D. Etlinger

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph D. Etlinger. A scholar is included among the top collaborators of Joseph D. Etlinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph D. Etlinger. Joseph D. Etlinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zeman, Richard J., et al.. (2021). Role of the Polyol Pathway in Locomotor Recovery and Wallerian Degeneration after Spinal Cord Contusion Injury. SHILAP Revista de lepidopterología. 2(1). 411–423. 4 indexed citations
2.
Murakami, Koko & Joseph D. Etlinger. (2018). Role of SMURF1 ubiquitin ligase in BMP receptor trafficking and signaling. Cellular Signalling. 54. 139–149. 20 indexed citations
3.
Peng, Hong, et al.. (2010). Partial functional recovery after complete spinal cord transection by combined chondroitinase and clenbuterol treatment. Pflügers Archiv - European Journal of Physiology. 460(3). 657–666. 29 indexed citations
4.
Zeman, Richard J., et al.. (2009). Improved functional recovery with oxandrolone after spinal cord injury in rats. Neuroreport. 20(9). 864–868. 14 indexed citations
5.
Zeman, Richard J., et al.. (2008). STEREOTACTIC RADIOSURGERY IMPROVES LOCOMOTOR RECOVERY AFTER SPINAL CORD INJURY IN RATS. Neurosurgery. 63(5). 981–988. 11 indexed citations
6.
Guo, Gary, Kirit Patel, Mehul Shah, et al.. (2002). Association of the Chaperone Glucose-Regulated Protein 58 (GRP58/ER-60/ERp57) with Stat3 in Cytosol and Plasma Membrane Complexes. Journal of Interferon & Cytokine Research. 22(5). 555–563. 82 indexed citations
7.
Zeman, Richard J., Hong Peng, Paul Visintainer, et al.. (2001). X-irradiation of the Contusion Site Improves Locomotor and Histological Outcomes in Spinal Cord-Injured Rats. Experimental Neurology. 172(1). 228–234. 31 indexed citations
8.
Zeman, Richard J., Hong Peng, M. J. Danon, & Joseph D. Etlinger. (2000). Clenbuterol reduces degeneration of exercised or aged dystrophic (mdx) muscle. Muscle & Nerve. 23(4). 521–528. 39 indexed citations
9.
Zeman, Richard J., Yong Feng, Hong Peng, & Joseph D. Etlinger. (1999). Clenbuterol, a β2-Adrenoceptor Agonist, Improves Locomotor and Histological Outcomes after Spinal Cord Contusion in Rats. Experimental Neurology. 159(1). 267–273. 37 indexed citations
10.
Patel, Kirit, MacKevin Ndubuisi, Sansar Sharma, et al.. (1997). Proteasome- and p53-dependent Masking of Signal Transducer and Activator of Transcription (STAT) Factors. Journal of Biological Chemistry. 272(8). 4659–4662. 34 indexed citations
11.
Lerea, Kenneth M., et al.. (1996). Phosphorylation of the Proteasome Activator PA28 Is Required for Proteasome Activation. Biochemical and Biophysical Research Communications. 225(3). 855–860. 30 indexed citations
12.
Suárez, Guillaume, et al.. (1995). Fructated Protein Is More Resistant to ATP-Dependent Proteolysis than Glucated Protein Possibly as a Result of Higher Content of Maillard Fluorophores. Archives of Biochemistry and Biophysics. 321(1). 209–213. 25 indexed citations
13.
Etlinger, Joseph D., et al.. (1993). Phosphorylation and Ubiquitination of the 26S Proteasome Complex. PubMed. 47(4-6). 325–329. 15 indexed citations
14.
Etlinger, Joseph D., et al.. (1992). Ubiquitinated proteasome inhibitor is a component of the 26 S proteasome complex. Biochemistry. 31(48). 11963–11967. 18 indexed citations
15.
Gu, Min, et al.. (1991). Isolation and characterization of a novel endogenous inhibitor of the proteasome. Biochemistry. 30(40). 9709–9715. 46 indexed citations
16.
Etlinger, Joseph D., et al.. (1989). Protease/inhibitor mechanisms involved in ATP-dependent proteolysis.. PubMed. 20. 197–216. 2 indexed citations
17.
Zeman, Richard J., et al.. (1987). Clenbuterol, a β2-agonist, retards atrophy in denervated muscles. American Journal of Physiology-Legacy Content. 252(1). 64 indexed citations
18.
Zeman, Richard J., et al.. (1986). Calcium Regulation of Lysosomal Proteolysis in Skeletal Musclea. Annals of the New York Academy of Sciences. 463(1). 247–249. 6 indexed citations
19.
Zeman, Richard J., et al.. (1985). Regulation of protein degradation in muscle by calcium. Journal of Biological Chemistry. 260. 23 indexed citations
20.
Janeczko, Richard & Joseph D. Etlinger. (1984). Inhibition of intracellular proteolysis in muscle cultures by multiplication-stimulating activity. Journal of Biological Chemistry. 259. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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