Jonathan Kaye

20.2k citations
85 papers · 6.3k indexed · 2 hit papers · h-index 40

Jonathan Kaye

81 papers receiving 6.0k citations

Hit Papers

Selective development of CD4+ T cells in transgenic mice ...5811983202619972011100200300400500

Peers

Jonathan Kaye
Comparison fields: 5 of 126
  • Immunology 4.4k
  • Physiology 394
  • Oncology 1.2k
  • Immunology and Allergy 198
  • Radiology, Nuclear Medicine and Imaging 628
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C R Maliszewski United States
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Citations per field
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Citations per year

Countries citing papers authored by Jonathan Kaye

Since Specialization
Citations

This map shows the geographic impact of Jonathan Kaye's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan Kaye with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan Kaye more than expected).

Fields of papers citing papers by Jonathan Kaye

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan Kaye. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan Kaye. The network helps show where Jonathan Kaye may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Jonathan Kaye, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jonathan Kaye Line = papers co-authored together Jonathan Kaye links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20250
2 2020125
3 201531
4 2010181
5 20091
6 200946
7 20075
8 200577
9 2004173
10 200394
11 199827
12 19971
13 19956
14 1992152
15 1989102
16 198633
17 1984118
18 198336
19
Both a monoclonal antibody and antisera specific for determinants unique to individual cloned helper T cell lines can substitute for antigen and antigen-presenting cells in the activation of T cells.breakdown →
1983586
20
Lymphocyte Stimulation. Differential Sensitivity to Radiation. Biochemical and Immunological Processes.
19823

About Jonathan Kaye

Jonathan Kaye is a scholar working on Immunology, Oncology and Physiology, having authored 85 papers that have together received 6.3k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (43 papers), T-cell and B-cell Immunology (41 papers), Monoclonal and Polyclonal Antibodies Research (14 papers), Immunotherapy and Immune Responses (13 papers), CAR-T cell therapy research (11 papers), Biochemical and Molecular Research (7 papers), IL-33, ST2, and ILC Pathways (7 papers) and Cytomegalovirus and herpesvirus research (7 papers). The work is most often cited by research in Immunology (4.4k citations), Physiology (394 citations) and Oncology (1.2k citations). Jonathan Kaye has collaborated with scholars based in United States, Switzerland and Canada. Frequent co-authors include Charles A. Janeway, Stephen Μ. Hedrick, Parinaz Aliahmad, J. Edwin Seegmiller, Dennis A. Carson, J P Tite, D A Carson, Benjamin F. Jones, Brian de la Torre and Stephen C. Jameson. Their work appears in journals such as The Journal of Immunology, The Journal of Experimental Medicine, Proceedings of the National Academy of Sciences, Nature Immunology and Nature Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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