Jonathan E. Feig

4.0k citations

35 papers · 3.1k indexed · h-index 25

Impact in

Immunology top 2%
- Atherosclerosis and Cardiovascular Diseases
- Immune cells in cancer
Cancer Research top 5%

Papers in ⓘ

Immunology 15
- Atherosclerosis and Cardiovascular Diseases 14
Immunology and Allergy 3

Co-authors: Edward A. Fisher (28 shared papers)Yuliya Vengrenyuk (10 shared papers)Bernd Hewing (3 shared papers)Jonathan D. Smith (2 shared papers)Stanley L. Hazen (2 shared papers)Michael J. Garabedian (6 shared papers)Raanan Shamir (4 shared papers)Kevin Jon Williams (2 shared papers)
Journals: Circulation Research (4 papers)Arteriosclerosis Thrombosis and Vascular Biology (3 papers)Proceedings of the National Academy of Sciences (2 papers)PLoS ONE (2 papers)Coronary Artery Disease (1 paper)
Partner nations: United States United Kingdom France

In The Last Decade

Jonathan E. Feig

34 papers receiving 3.0k citations

Peers

Countries citing papers authored by Jonathan E. Feig

Since Specialization

Citations

This map shows the geographic impact of Jonathan E. Feig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan E. Feig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan E. Feig more than expected).

Fields of papers citing papers by Jonathan E. Feig

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan E. Feig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan E. Feig. The network helps show where Jonathan E. Feig may publish in the future.

Co-authors

The 25 scholars most cited alongside Jonathan E. Feig, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jonathan E. Feig Line = papers co-authored together Jonathan E. Feig links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 35 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Noninvasive detection of macrophages using a nanoparticulate contrast agent for computed tomography Nature Medicine ·Fabien Hyafil, Jean‐Christophe Cornily, Jonathan E. Feig, Ronald Gordon, Esad Vucic, Vardan Amirbekian, Edward A. Fisher, Valentı́n Fuster, Laurent J. Feldman, Zahi A. Fayad	2007	358
2	High-Density Lipoprotein Function, Dysfunction, and Reverse Cholesterol Transport Arteriosclerosis Thrombosis and Vascular Biology ·Edward A. Fisher, Jonathan E. Feig, Bernd Hewing, Stanley L. Hazen, Jonathan D. Smith	2012	299
3	Gene expression changes in foam cells and the role of chemokine receptor CCR7 during atherosclerosis regression in ApoE-deficient mice Proceedings of the National Academy of Sciences ·Eugene Trogan, Jonathan E. Feig, Snjezana Doğan, George H. Rothblat, Véronique Angeli, Frank Tacke, Gwendalyn J. Randolph, Edward A. Fisher	2006	287
4	HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells Proceedings of the National Academy of Sciences ·Jonathan E. Feig, James X. Rong, Raanan Shamir, Marie Sanson, Yuliya Vengrenyuk, Jianhua Liu, Katey J. Rayner, Kathryn J. Moore, Michael J. Garabedian, Edward A. Fisher	2011	250
5	Reversal of Hyperlipidemia With a Genetic Switch Favorably Affects the Content and Inflammatory State of Macrophages in Atherosclerotic Plaques Circulation ·Jonathan E. Feig, Saj Parathath, James X. Rong, Stephanie L. Mick, Yuliya Vengrenyuk, Lisa Grauer, Stephen G. Young, Edward A. Fisher	2011	195
6	Rapid regression of atherosclerosis: insights from the clinical and experimental literature Nature Clinical Practice Cardiovascular Medicine ·Kevin Jon Williams, Jonathan E. Feig, Edward A. Fisher	2008	150
7	LXR promotes the maximal egress of monocyte-derived cells from mouse aortic plaques during atherosclerosis regression Journal of Clinical Investigation ·Jonathan E. Feig, Inès Pineda‐Torra, Marie Sanson, Michelle N. Bradley, Yuliya Vengrenyuk, Dusan Bogunovic, Emmanuel L. Gautier, Daniel Rubinstein, Cynthia Hong, Jianhua Liu, Chaowei Wu, Nico van Rooijen, Nina Bhardwaj, Michael J. Garabedian, Peter Tontonoz, Edward A. Fisher	2010	146
8	High-Density Lipoprotein and Atherosclerosis Regression Circulation Research ·Jonathan E. Feig, Bernd Hewing, Jonathan D. Smith, Stanley L. Hazen, Edward A. Fisher	2014	136
9	Hypoxia Is Present in Murine Atherosclerotic Plaques and Has Multiple Adverse Effects on Macrophage Lipid Metabolism Circulation Research ·Saj Parathath, Stephanie L. Mick, Jonathan E. Feig, Victor Joaquin, Lisa Grauer, David M. Habiel, Max Gassmann, Lawrence B. Gardner, Edward A. Fisher	2011	131
10	Statins Promote the Regression of Atherosclerosis via Activation of the CCR7-Dependent Emigration Pathway in Macrophages PLoS ONE ·Jonathan E. Feig, Yueting Shang, Noemí Rotllán, Yuliya Vengrenyuk, Chaowei Wu, Raanan Shamir, Inès Pineda‐Torra, Carlos Fernández‐Hernando, Edward A. Fisher, Michael J. Garabedian	2011	124
11	Neuroimmune Guidance Cue Semaphorin 3E Is Expressed in Atherosclerotic Plaques and Regulates Macrophage Retention Arteriosclerosis Thrombosis and Vascular Biology ·Amarylis Wanschel, Tara Seibert, Bernd Hewing, Bhama Ramkhelawon, Tathagat Dutta Ray, Janine M. van Gils, Katey J. Rayner, Jonathan E. Feig, Edward R. O’Brien, Edward A. Fisher, Kathryn J. Moore	2013	108
12	Optical coherence tomography assessment of the mechanistic effects of rotational and orbital atherectomy in severely calcified coronary lesions Catheterization and Cardiovascular Interventions ·Annapoorna Kini, Yuliya Vengrenyuk, Jacobo Pena, Sadako Motoyama, Jonathan E. Feig, Omar A. Meelu, Anitha Rajamanickam, Arjun Bhat, Sadik Panwar, Usman Baber, Samin K. Sharma	2015	105
13	Regression of Atherosclerosis Is Characterized by Broad Changes in the Plaque Macrophage Transcriptome PLoS ONE ·Jonathan E. Feig, Yuliya Vengrenyuk, Vladimı́r Reiser, Chaowei Wu, Alexander Statnikov, Constantin Aliferis, Michael J. Garabedian, Edward A. Fisher, Óscar Puig	2012	91
14	KATP channels of primary human coronary artery endothelial cells consist of a heteromultimeric complex of Kir6.1, Kir6.2, and SUR2B subunits Journal of Molecular and Cellular Cardiology ·Hidetada Yoshida, Jonathan E. Feig, Alison Morrissey, Ioana Ghiu, Michael Artman, William A. Coetzee	2004	78
15	LXRα Regulates Macrophage Arginase 1 Through PU.1 and Interferon Regulatory Factor 8 Circulation Research ·Benoît Pourcet, Jonathan E. Feig, Yuliya Vengrenyuk, Adrian J. Hobbs, Diane Kepka‐Lenhart, Michael J. Garabedian, Sidney M. Morris, Edward A. Fisher, Inès Pineda‐Torra	2011	76
16	Atheroprotective Effects of HDL: Beyond Reverse Cholesterol Transport Current Drug Targets ·Edward A. Fisher, Jonathan E. Feig, Raanan Shamir	2008	73
17	Collagen-Specific Peptide Conjugated HDL Nanoparticles as MRI Contrast Agent to Evaluate Compositional Changes in Atherosclerotic Plaque Regression JACC. Cardiovascular imaging ·Wei Chen, David P. Cormode, Yuliya Vengrenyuk, Beatriz Herránz, Jonathan E. Feig, Ahmed Klink, Willem J. M. Mulder, Edward A. Fisher, Zahi A. Fayad	2013	66
18	Phosphorylation of Liver X Receptor α Selectively Regulates Target Gene Expression in Macrophages Molecular and Cellular Biology ·Inès Pineda‐Torra, Naı̈ma Ismaı̈li, Jonathan E. Feig, Chong‐Feng Xu, Claudio N. Cavasotto, Raluca Pancratov, Inez Rogatsky, Thomas A. Neubert, Edward A. Fisher, Michael J. Garabedian	2008	65
19	Multimodality Intravascular Imaging to Predict Periprocedural Myocardial Infarction During Percutaneous Coronary Intervention JACC: Cardiovascular Interventions ·Annapoorna Kini, Sadako Motoyama, Yuliya Vengrenyuk, Jonathan E. Feig, Jacobo Pena, Usman Baber, Arjun Bhat, Pedro Moreno, Jason C. Kovacic, Jagat Narula, Samin K. Sharma	2015	54
20	Regression of Atherosclerosis: Insights from Animal and Clinical Studies Annals of Global Health ·Jonathan E. Feig	2014	52

About Jonathan E. Feig

Jonathan E. Feig is a scholar working on Immunology, Immunology and Allergy, Endocrinology, Diabetes and Metabolism, Surgery and Cancer Research, having authored 35 papers that have together received 3.1k indexed citations. Recurring topics across this work include Atherosclerosis and Cardiovascular Diseases (14 papers), Lipoproteins and Cardiovascular Health (8 papers), Diabetes, Cardiovascular Risks, and Lipoproteins (6 papers), Cancer, Lipids, and Metabolism (5 papers), Cholesterol and Lipid Metabolism (5 papers), Peroxisome Proliferator-Activated Receptors (5 papers), Adipokines, Inflammation, and Metabolic Diseases (5 papers) and Cardiac Imaging and Diagnostics (4 papers). The work is most often cited by research in Immunology (1.2k citations), Cancer Research (410 citations), Surgery (1.0k citations), Endocrinology, Diabetes and Metabolism (337 citations) and Immunology and Allergy (117 citations). Jonathan E. Feig has collaborated with scholars based in United States, United Kingdom and France. Frequent co-authors include Edward A. Fisher, Yuliya Vengrenyuk, Bernd Hewing, Jonathan D. Smith, Stanley L. Hazen, Michael J. Garabedian, Raanan Shamir, Kevin Jon Williams, James X. Rong and Zahi A. Fayad. Their work appears in journals such as Circulation Research, Arteriosclerosis Thrombosis and Vascular Biology, Proceedings of the National Academy of Sciences, PLoS ONE and Coronary Artery Disease.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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