John MacFadyen

579 total citations
7 papers, 440 citations indexed

About

John MacFadyen is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, John MacFadyen has authored 7 papers receiving a total of 440 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Immunology. Recurrent topics in John MacFadyen's work include TGF-β signaling in diseases (3 papers), Protease and Inhibitor Mechanisms (2 papers) and Cell Adhesion Molecules Research (2 papers). John MacFadyen is often cited by papers focused on TGF-β signaling in diseases (3 papers), Protease and Inhibitor Mechanisms (2 papers) and Cell Adhesion Molecules Research (2 papers). John MacFadyen collaborates with scholars based in United Kingdom, United States and Spain. John MacFadyen's co-authors include Clare M. Isacke, Dirk Wienke, Kay Savage, Christopher D. Buckley, Oliver Haworth, Howard R. Morris, Mark Sutton‐Smith, Anne Dell, Deborah Hardie and Peter van der Geer and has published in prestigious journals such as FEBS Letters, Molecular Biology of the Cell and EMBO Reports.

In The Last Decade

John MacFadyen

7 papers receiving 424 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John MacFadyen United Kingdom 7 220 117 114 112 112 7 440
Erica Lorenzon Switzerland 7 291 1.3× 78 0.7× 104 0.9× 81 0.7× 114 1.0× 8 468
M. Denijn Netherlands 7 179 0.8× 97 0.8× 117 1.0× 78 0.7× 120 1.1× 10 376
M C van Altena Netherlands 8 319 1.4× 99 0.8× 192 1.7× 53 0.5× 119 1.1× 8 470
Philip G. Hargreaves United Kingdom 8 126 0.6× 183 1.6× 86 0.8× 130 1.2× 110 1.0× 12 608
Christina Sundberg Sweden 10 309 1.4× 126 1.1× 174 1.5× 43 0.4× 96 0.9× 10 607
Elizabeth M. Chislock United States 6 248 1.1× 60 0.5× 197 1.7× 75 0.7× 39 0.3× 6 469
Katherine A. Rybinski United States 9 190 0.9× 58 0.5× 192 1.7× 71 0.6× 62 0.6× 23 464
Bruna Wassermann Italy 11 241 1.1× 130 1.1× 253 2.2× 104 0.9× 124 1.1× 14 553
E Legrand France 8 193 0.9× 53 0.5× 113 1.0× 83 0.7× 70 0.6× 9 368
Yasuyoshi Sohara Japan 10 243 1.1× 103 0.9× 171 1.5× 46 0.4× 164 1.5× 11 519

Countries citing papers authored by John MacFadyen

Since Specialization
Citations

This map shows the geographic impact of John MacFadyen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John MacFadyen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John MacFadyen more than expected).

Fields of papers citing papers by John MacFadyen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John MacFadyen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John MacFadyen. The network helps show where John MacFadyen may publish in the future.

Co-authorship network of co-authors of John MacFadyen

This figure shows the co-authorship network connecting the top 25 collaborators of John MacFadyen. A scholar is included among the top collaborators of John MacFadyen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John MacFadyen. John MacFadyen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Hawkins, Nathaniel M., Robert S. McKelvie, Stephanie Poon, et al.. (2023). Patient Eligibility for Established and Novel Guideline-Directed Medical Therapies After Acute Heart Failure Hospitalization. JACC Heart Failure. 11(5). 596–606. 10 indexed citations
2.
Hardie, Debbie L., Mathew Baldwin, Amy J. Naylor, et al.. (2011). The stromal cell antigen CD248 (endosialin) is expressed on naive CD8+ human T cells and regulates proliferation. Immunology. 133(3). 288–295. 33 indexed citations
3.
Lax, Siân, Tie Zheng Hou, Eric J. Jenkinson, et al.. (2007). CD248/Endosialin is dynamically expressed on a subset of stromal cells during lymphoid tissue development, splenic remodeling and repair. FEBS Letters. 581(18). 3550–3556. 44 indexed citations
4.
MacFadyen, John, Kay Savage, Dirk Wienke, & Clare M. Isacke. (2006). Endosialin is expressed on stromal fibroblasts and CNS pericytes in mouse embryos and is downregulated during development. Gene Expression Patterns. 7(3). 363–369. 74 indexed citations
5.
MacFadyen, John, Oliver Haworth, Deborah Hardie, et al.. (2005). Endosialin (TEM1, CD248) is a marker of stromal fibroblasts and is not selectively expressed on tumour endothelium. FEBS Letters. 579(12). 2569–2575. 137 indexed citations
6.
Wienke, Dirk, John MacFadyen, & Clare M. Isacke. (2003). Identification and Characterization of the Endocytic Transmembrane Glycoprotein Endo180 as a Novel Collagen Receptor. Molecular Biology of the Cell. 14(9). 3592–3604. 118 indexed citations
7.
Rivera-Calzada, Ángel, David Robertson, John MacFadyen, et al.. (2003). Three‐dimensional interplay among the ligand‐binding domains of the urokinase‐plasminogen‐activator‐receptor‐associated protein, Endo180. EMBO Reports. 4(8). 807–812. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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