Heldin Ch

946 total citations
11 papers, 727 citations indexed

About

Heldin Ch is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Oncology. According to data from OpenAlex, Heldin Ch has authored 11 papers receiving a total of 727 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 2 papers in Pathology and Forensic Medicine and 2 papers in Oncology. Recurrent topics in Heldin Ch's work include TGF-β signaling in diseases (3 papers), Genetic factors in colorectal cancer (2 papers) and Protease and Inhibitor Mechanisms (1 paper). Heldin Ch is often cited by papers focused on TGF-β signaling in diseases (3 papers), Genetic factors in colorectal cancer (2 papers) and Protease and Inhibitor Mechanisms (1 paper). Heldin Ch collaborates with scholars based in Sweden, Japan and Netherlands. Heldin Ch's co-authors include Kohei Miyazono, Jorma Keski‐Oja, Jussi Taipale, Daizo Koinuma, Masato Morikawa, Bengt Westermark, Peter ten Dijke, Carmen Herrera Hidalgo, Eleftheria Vasilaki and Anders Sundqvist and has published in prestigious journals such as The Journal of Cell Biology, Oncogene and British Journal of Cancer.

In The Last Decade

Heldin Ch

11 papers receiving 714 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Heldin Ch Sweden	7	479	154	121	113	98	11	727
Roger Lucas United States	6	711 1.5×	223 1.4×	123 1.0×	174 1.5×	102 1.0×	6	972
Ylva Paulsson Sweden	14	501 1.0×	170 1.1×	105 0.9×	75 0.7×	71 0.7×	17	803
Nina Perusinghe United Kingdom	15	507 1.1×	317 2.1×	141 1.2×	57 0.5×	133 1.4×	20	820
K Rubin Sweden	8	380 0.8×	158 1.0×	86 0.7×	143 1.3×	57 0.6×	8	850
Hong-Ming Zhou United States	8	437 0.9×	194 1.3×	108 0.9×	105 0.9×	75 0.8×	8	804
Teresa López‐Rovira Spain	9	656 1.4×	156 1.0×	94 0.8×	39 0.3×	114 1.2×	10	817
T Okadome Japan	9	680 1.4×	157 1.0×	43 0.4×	57 0.5×	76 0.8×	15	846
Shelley N.-M. Thai United States	8	279 0.6×	106 0.7×	240 2.0×	141 1.2×	84 0.9×	8	598
Tanya A. Rege United States	8	339 0.7×	153 1.0×	108 0.9×	90 0.8×	34 0.3×	13	735
Taro Hayakawa Japan	11	263 0.5×	135 0.9×	249 2.1×	75 0.7×	81 0.8×	18	668

Countries citing papers authored by Heldin Ch

Since Specialization

Citations

This map shows the geographic impact of Heldin Ch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heldin Ch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heldin Ch more than expected).

Fields of papers citing papers by Heldin Ch

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Heldin Ch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heldin Ch. The network helps show where Heldin Ch may publish in the future.

Co-authorship network of co-authors of Heldin Ch

This figure shows the co-authorship network connecting the top 25 collaborators of Heldin Ch. A scholar is included among the top collaborators of Heldin Ch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Heldin Ch. Heldin Ch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

11 of 11 papers shown

Morikawa, Masato, Daizo Koinuma, Kohei Miyazono, & Heldin Ch. (2012). Genome-wide mechanisms of Smad binding. Oncogene. 32(13). 1609–1615. 91 indexed citations

Sundqvist, Anders, Agata Zieba, Eleftheria Vasilaki, et al.. (2012). Specific interactions between Smad proteins and AP-1 components determine TGFβ-induced breast cancer cell invasion. Oncogene. 32(31). 3606–3615. 89 indexed citations

Ch, Heldin. (1996). Role of platelet-derived growth factor in vivo. Medical Entomology and Zoology. 249–273. 44 indexed citations

Ch, Heldin & Ken‐ichi Miyazono. (1995). [Transforming growth factor-beta. An interesting candidate for clinical use].. PubMed. 92(15). 1569–72. 3 indexed citations

Nistér, Monica, Per Enblad, G. Bäckström, et al.. (1994). Platelet-derived growth factor (PDGF) in neoplastic and non-neoplastic cystic lesions of the central nervous system and in the cerebrospinal fluid. British Journal of Cancer. 69(5). 952–956. 23 indexed citations

Taipale, Jussi, Kohei Miyazono, Heldin Ch, & Jorma Keski‐Oja. (1994). Latent transforming growth factor-beta 1 associates to fibroblast extracellular matrix via latent TGF-beta binding protein. The Journal of Cell Biology. 124(1). 171–181. 373 indexed citations

Kato, Mitsuyasu, A Gobl, Hidenori Ichijo, et al.. (1994). Enhanced expression of transforming growth factor-beta s and transforming growth factor-beta type II receptor in the synovial tissues of patients with rheumatoid arthritis.. PubMed. 70(5). 620–30. 72 indexed citations

Ch, Heldin & Bengt Westermark. (1991). Platelet-derived growth factor and autocrine mechanisms of oncogenic processes.. PubMed. 2(2). 109–24. 29 indexed citations

Ch, Heldin, Ken‐ichi Miyazono, Lena Claesson‐Welsh, & Bengt Westermark. (1989). Growth regulatory proteins from human platelets.. PubMed. 20. 81–92. 1 indexed citations

10.

Ch, Heldin, Bengt Westermark, & Åke Wasteson. (1984). [Platelet-derived growth factor in normal and malignant cell growth].. PubMed. 99(12). 319–22. 1 indexed citations

11.

Busch, Christian, Heldin Ch, Åke Wasteson, & Bengt Westermark. (1977). Stimulation of endothelial cell proliferation by a factor released from human platelets.. PubMed. 219–22. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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