John C. Ringold

1.3k total citations · 1 hit paper
7 papers, 943 citations indexed

About

John C. Ringold is a scholar working on Pathology and Forensic Medicine, Surgery and Genetics. According to data from OpenAlex, John C. Ringold has authored 7 papers receiving a total of 943 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Pathology and Forensic Medicine, 3 papers in Surgery and 3 papers in Genetics. Recurrent topics in John C. Ringold's work include Genetic factors in colorectal cancer (5 papers), Digestive system and related health (3 papers) and Cholangiocarcinoma and Gallbladder Cancer Studies (3 papers). John C. Ringold is often cited by papers focused on Genetic factors in colorectal cancer (5 papers), Digestive system and related health (3 papers) and Cholangiocarcinoma and Gallbladder Cancer Studies (3 papers). John C. Ringold collaborates with scholars based in United States and Finland. John C. Ringold's co-authors include James R. Howe, Robert W. Summers, Edwin M. Stone, Frank A. Mitros, Lauri A. Aaltonen, Pertti Sistonen, Stina Roth, Steve Bevan, Richard S. Houlston and Heikki Järvinen and has published in prestigious journals such as Science, Gastroenterology and The American Journal of Human Genetics.

In The Last Decade

John C. Ringold

7 papers receiving 906 citations

Hit Papers

Mutations in the SMAD4/DPC4 Gene in Juvenile Polyposis 1998 2026 2007 2016 1998 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John C. Ringold United States 6 491 427 396 217 148 7 943
Efraim H. Rosenberg Netherlands 15 96 0.2× 413 1.0× 235 0.6× 130 0.6× 214 1.4× 27 1.1k
R. Yoshiyuki Osamura Japan 16 79 0.2× 201 0.5× 196 0.5× 71 0.3× 175 1.2× 39 779
Ginette Gunz France 27 54 0.1× 453 1.1× 240 0.6× 156 0.7× 559 3.8× 43 2.3k
Angela Liou United States 9 52 0.1× 296 0.7× 332 0.8× 96 0.4× 246 1.7× 13 723
Jamel Chelly France 11 108 0.2× 905 2.1× 241 0.6× 162 0.7× 77 0.5× 14 1.2k
Óscar Bengoechea Spain 15 193 0.4× 121 0.3× 251 0.6× 25 0.1× 100 0.7× 24 467
Jane S. Green Canada 7 251 0.5× 353 0.8× 173 0.4× 406 1.9× 66 0.4× 7 808
John Garcia United States 10 90 0.2× 597 1.4× 106 0.3× 441 2.0× 60 0.4× 16 1.3k
Rossella Gulli Italy 14 83 0.2× 180 0.4× 99 0.3× 58 0.3× 78 0.5× 21 676
Irene Chicote Spain 7 174 0.4× 522 1.2× 190 0.5× 69 0.3× 30 0.2× 14 729

Countries citing papers authored by John C. Ringold

Since Specialization
Citations

This map shows the geographic impact of John C. Ringold's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John C. Ringold with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John C. Ringold more than expected).

Fields of papers citing papers by John C. Ringold

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John C. Ringold. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John C. Ringold. The network helps show where John C. Ringold may publish in the future.

Co-authorship network of co-authors of John C. Ringold

This figure shows the co-authorship network connecting the top 25 collaborators of John C. Ringold. A scholar is included among the top collaborators of John C. Ringold based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John C. Ringold. John C. Ringold is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Ellingrod, Vicki L., Paul J. Perry, John C. Ringold, et al.. (2005). Weight gain associated with the −759C/T polymorphism of the 5HT2C receptor and olanzapine. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 134B(1). 76–78. 112 indexed citations
2.
Ellingrod, Vicki L., Del D. Miller, John C. Ringold, & Paul J. Perry. (2004). Distribution of the serotonin 2C (5HT2C) receptor gene −759C/T polymorphism in patients with schizophrenia and normal controls. Psychiatric Genetics. 14(2). 93–95. 8 indexed citations
3.
Howe, James R., Brian Wagner, John C. Ringold, et al.. (2002). Common Deletion of SMAD4 in Juvenile Polyposis Is a Mutational Hotspot. The American Journal of Human Genetics. 70(5). 1357–1362. 32 indexed citations
4.
Howe, James R., John C. Ringold, Jonathan H. Hughes, & Robert W. Summers. (1999). Direct genetic testing for Smad4 mutations in patients at risk for juvenile polyposis. Surgery. 126(2). 162–170. 39 indexed citations
5.
Howe, James R., John C. Ringold, Robert W. Summers, et al.. (1998). A Gene for Familial Juvenile Polyposis Maps to Chromosome 18q21.1. The American Journal of Human Genetics. 62(5). 1129–1136. 82 indexed citations
6.
Howe, James R., Stina Roth, John C. Ringold, et al.. (1998). Mutations in the SMAD4/DPC4 Gene in Juvenile Polyposis. Science. 280(5366). 1086–1088. 668 indexed citations breakdown →
7.
Howe, James R., John C. Ringold, R. W. Summers, et al.. (1998). The gene for familial juvenile polyposis maps to chromosome 18q21. Gastroenterology. 114. A613–A613. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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