John C. Gill

1.5k total citations
18 papers, 802 citations indexed

About

John C. Gill is a scholar working on Reproductive Medicine, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, John C. Gill has authored 18 papers receiving a total of 802 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Reproductive Medicine, 6 papers in Molecular Biology and 3 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in John C. Gill's work include Hypothalamic control of reproductive hormones (10 papers), Fibroblast Growth Factor Research (3 papers) and Ovarian function and disorders (3 papers). John C. Gill is often cited by papers focused on Hypothalamic control of reproductive hormones (10 papers), Fibroblast Growth Factor Research (3 papers) and Ovarian function and disorders (3 papers). John C. Gill collaborates with scholars based in United States, Canada and Switzerland. John C. Gill's co-authors include Pei‐San Tsai, Andrew Fitton, R.C. Heel, Suzanne M. Moenter, Ursula B. Kaiser, Rona S. Carroll, Víctor M. Navarro, Keith Finnie, Michael Gent and Douglas Holder and has published in prestigious journals such as Circulation, PLoS ONE and Endocrinology.

In The Last Decade

John C. Gill

17 papers receiving 776 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John C. Gill United States 12 387 292 157 116 93 18 802
Almorris Lynch United States 16 210 0.5× 190 0.7× 51 0.3× 52 0.4× 211 2.3× 17 815
Nicole H. Rogers United States 12 59 0.2× 277 0.9× 153 1.0× 77 0.7× 50 0.5× 15 1.0k
Maie Al‐Bader Kuwait 16 181 0.5× 290 1.0× 154 1.0× 21 0.2× 57 0.6× 41 797
Huili Zhu China 17 301 0.8× 203 0.7× 31 0.2× 28 0.2× 47 0.5× 38 950
Shuai Yan China 14 36 0.1× 220 0.8× 33 0.2× 52 0.4× 81 0.9× 38 687
L. Schenkel Switzerland 12 120 0.3× 145 0.5× 348 2.2× 101 0.9× 92 1.0× 16 968
Rochelle M. Hanley United States 11 351 0.9× 431 1.5× 33 0.2× 112 1.0× 39 0.4× 17 855
Paula Díaz‐Sylvester United States 18 93 0.2× 371 1.3× 18 0.1× 195 1.7× 40 0.4× 42 671
Vanda Pinto Portugal 14 20 0.1× 373 1.3× 44 0.3× 45 0.4× 57 0.6× 23 630

Countries citing papers authored by John C. Gill

Since Specialization
Citations

This map shows the geographic impact of John C. Gill's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John C. Gill with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John C. Gill more than expected).

Fields of papers citing papers by John C. Gill

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John C. Gill. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John C. Gill. The network helps show where John C. Gill may publish in the future.

Co-authorship network of co-authors of John C. Gill

This figure shows the co-authorship network connecting the top 25 collaborators of John C. Gill. A scholar is included among the top collaborators of John C. Gill based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John C. Gill. John C. Gill is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Singh, Sunita, et al.. (2022). 413.3: Towards a National System of Living Donor Follow-up in Canada. Transplantation. 106(9S). S410–S410. 1 indexed citations
2.
Gill, John C., Katherine Lazarides, Denis Grandgirard, et al.. (2021). Hair Cell Generation in Cochlear Culture Models Mediated by Novel γ-Secretase Inhibitors. Frontiers in Cell and Developmental Biology. 9. 710159–710159. 12 indexed citations
3.
Gill, John C., Hye‐Jeong Ha, Zeli Shen, et al.. (2016). Helicobacter bilisandHelicobacter trogontum: infectious causes of abortion in sheep. Journal of Veterinary Diagnostic Investigation. 28(3). 225–234. 11 indexed citations
4.
Simavlı, Serap, Iain Thompson, John C. Gill, et al.. (2015). Substance P Regulates Puberty Onset and Fertility in the Female Mouse. Endocrinology. 156(6). 2313–2322. 52 indexed citations
5.
Gill, John C., Víctor M. Navarro, Sekoni D. Noel, et al.. (2012). Increased Neurokinin B (Tac2) Expression in the Mouse Arcuate Nucleus Is an Early Marker of Pubertal Onset with Differential Sensitivity to Sex Steroid-Negative Feedback than Kiss1. Endocrinology. 153(10). 4883–4893. 69 indexed citations
6.
7.
Jeong, Kyeong‐Hoon, John C. Gill, Vânia Nosé, et al.. (2009). Expression of a Gonadotropin-Releasing Hormone Receptor-Simian Virus 40 T-Antigen Transgene Has Sex-Specific Effects on the Reproductive Axis. Endocrinology. 150(7). 3383–3391. 2 indexed citations
8.
Gill, John C., Brandon Wadas, Peilin Chen, et al.. (2008). The Gonadotropin-Releasing Hormone (GnRH) Neuronal Population Is Normal in Size and Distribution in GnRH-Deficient and GnRH Receptor-Mutant Hypogonadal Mice. Endocrinology. 149(9). 4596–4604. 30 indexed citations
9.
Moenter, Suzanne M., Stuart Tobet, Ursula B. Kaiser, et al.. (2008). Hypogonadal Mice in Size and Distribution in GnRH-Deficient and GnRH Receptor-Mutant The Gonadotropin-Releasing Hormone (GnRH) Neuronal Population Is Normal. 1 indexed citations
10.
Tsai, Pei‐San & John C. Gill. (2006). Mechanisms of Disease: insights into X-linked and autosomal-dominant Kallmann syndrome. Nature Clinical Practice Endocrinology & Metabolism. 2(3). 160–171. 69 indexed citations
11.
Gill, John C. & Pei‐San Tsai. (2005). Expression of a Dominant Negative FGF Receptor in Developing GNRH1 Neurons Disrupts Axon Outgrowth and Targeting to the Median Eminence1. Biology of Reproduction. 74(3). 463–472. 44 indexed citations
12.
Gill, John C., Suzanne M. Moenter, & Pei‐San Tsai. (2004). Developmental Regulation of Gonadotropin-Releasing Hormone Neurons by Fibroblast Growth Factor Signaling. Endocrinology. 145(8). 3830–3839. 68 indexed citations
13.
Tsai, Pei‐San, Suzanne M. Moenter, Mohammed El Majdoubi, et al.. (2004). Targeted Expression of a Dominant-Negative Fibroblast Growth Factor (FGF) Receptor in Gonadotropin-Releasing Hormone (GnRH) Neurons Reduces FGF Responsiveness and the Size of GnRH Neuronal Population. Molecular Endocrinology. 19(1). 225–236. 85 indexed citations
14.
Tonelli, Marcello, et al.. (2001). Cardiac risk factors and the use of cardioprotective medications in patients with chronic renal insufficiency. American Journal of Kidney Diseases. 37(3). 484–489. 30 indexed citations
15.
Gill, John C., et al.. (1999). Hepatotoxicity Possibly Caused by Amphotericin B. Annals of Pharmacotherapy. 33(6). 683–685. 11 indexed citations
16.
Cairns, John A., John C. Gill, Brian C. Morton, et al.. (1996). Fish Oils and Low-Molecular-Weight Heparin for the Reduction of Restenosis After Percutaneous Transluminal Coronary Angioplasty. Circulation. 94(7). 1553–1560. 116 indexed citations
17.
Gill, John C., R.C. Heel, & Andrew Fitton. (1992). Amiodarone. Drugs. 43(1). 69–110. 133 indexed citations
18.
Gill, John C., et al.. (1975). An Ohio Doctor Views Campaigning on the White River, 1864. The Arkansas Historical Quarterly. 34(4). 333–333. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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