Johan Luthman

6.6k total citations · 1 hit paper
91 papers, 3.3k citations indexed

About

Johan Luthman is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Johan Luthman has authored 91 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Cellular and Molecular Neuroscience, 31 papers in Molecular Biology and 22 papers in Physiology. Recurrent topics in Johan Luthman's work include Neuroscience and Neuropharmacology Research (26 papers), Neuropeptides and Animal Physiology (9 papers) and Alzheimer's disease research and treatments (9 papers). Johan Luthman is often cited by papers focused on Neuroscience and Neuropharmacology Research (26 papers), Neuropeptides and Animal Physiology (9 papers) and Alzheimer's disease research and treatments (9 papers). Johan Luthman collaborates with scholars based in Sweden, United States and United Kingdom. Johan Luthman's co-authors include Camilla Nilsberth, Lars Lannfelt, Margaret M. Condron, Steven G. Younkin, Karin Axelman, Jan Näslund, Christopher B. Eckman, Anita Westlind‐Danielsson, Charlotte Forsell and Charlotte Stenh and has published in prestigious journals such as Nature Neuroscience, The Journal of Comparative Neurology and Neuroscience.

In The Last Decade

Johan Luthman

89 papers receiving 3.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced Aβ protofibril formation

2001 943 citations Camilla Nilsberth, Anita Westlind‐Danielsson et al. Nature Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Johan Luthman Sweden	27	1.4k	1.2k	1.1k	526	411	91	3.3k
Gareth W. Roberts United Kingdom	27	1.8k 1.3×	1.5k 1.2×	775 0.7×	466 0.9×	602 1.5×	49	3.6k
Hoau-Yan Wang United States	30	1.9k 1.3×	2.0k 1.7×	1.6k 1.4×	397 0.8×	282 0.7×	50	4.6k
Kaoru Yamada Japan	30	1.5k 1.1×	1.4k 1.2×	904 0.8×	218 0.4×	400 1.0×	133	3.8k
Thomas G. Ohm Germany	35	2.2k 1.6×	1.4k 1.2×	1.1k 1.0×	583 1.1×	320 0.8×	86	3.9k
Nobuya Matsuoka Japan	32	722 0.5×	1.1k 1.0×	826 0.7×	374 0.7×	197 0.5×	96	2.9k
John J. LaFrancois United States	24	2.6k 1.9×	1.4k 1.2×	1.4k 1.2×	338 0.6×	453 1.1×	36	4.5k
Rajesh N. Kalaria United States	35	1.6k 1.2×	1.3k 1.1×	870 0.8×	507 1.0×	576 1.4×	65	3.9k
Manuela Polydoro United States	23	1.9k 1.4×	926 0.8×	960 0.8×	295 0.6×	514 1.3×	28	3.2k
Toshiaki Irie Japan	28	748 0.5×	699 0.6×	560 0.5×	448 0.9×	493 1.2×	96	2.7k
Fang Cai China	32	1.7k 1.3×	1.8k 1.5×	905 0.8×	209 0.4×	333 0.8×	91	4.2k

Countries citing papers authored by Johan Luthman

Since Specialization

Citations

This map shows the geographic impact of Johan Luthman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Johan Luthman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Johan Luthman more than expected).

Fields of papers citing papers by Johan Luthman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Johan Luthman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Johan Luthman. The network helps show where Johan Luthman may publish in the future.

Co-authorship network of co-authors of Johan Luthman

This figure shows the co-authorship network connecting the top 25 collaborators of Johan Luthman. A scholar is included among the top collaborators of Johan Luthman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Johan Luthman. Johan Luthman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Merchant, Kalpana, Jesse M. Cedarbaum, Patrik Brundin, et al.. (2018). A Proposed Roadmap for Parkinson’s Disease Proof of Concept Clinical Trials Investigating Compounds Targeting Alpha-Synuclein. Journal of Parkinson s Disease. 9(1). 31–61. 38 indexed citations

Johansson, Sara, Ann‐Cathrin Radesäter, Richard F. Cowburn, Johan Thyberg, & Johan Luthman. (2005). Modelling of amyloid β-peptide induced lesions using roller-drum incubation of hippocampal slice cultures from neonatal rats. Experimental Brain Research. 168(1-2). 11–24. 15 indexed citations

Nilsberth, Camilla, et al.. (2002). Changes in APP, PS1 and other factors related to Alzheimer’s disease pathophysiology after trimethyltin-induced brain lesion in the rat. Neurotoxicity Research. 4(7-8). 625–636. 39 indexed citations

Nilsberth, Camilla, Anita Westlind‐Danielsson, Christopher B. Eckman, et al.. (2001). The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced Aβ protofibril formation. Nature Neuroscience. 4(9). 887–893. 943 indexed citations breakdown →

Luthman, Johan. (2000). Anticonvulsant effects of the 3-hydroxyanthranilic acid dioxygenase inhibitor NCR-631. Amino Acids. 19(1). 325–334. 7 indexed citations

Nilsberth, Camilla, Johan Luthman, Lars Lannfelt, & Marianne Schultzberg. (1999). Expression of Presenilin 1 mRNA in Rat Peripheral Organs and Brain. The Histochemical Journal. 31(8). 515–523. 10 indexed citations

Luthman, Johan & Christian Humpel. (1997). Pentylenetetrazol kindling decreases N-methyl-d-aspartate and kainate but increases gamma-aminobutyric acid-A receptor binding in discrete rat brain areas. Neuroscience Letters. 239(1). 9–12. 15 indexed citations

Luthman, Johan, et al.. (1997). Functional changes induced by neonatal cerebral 6-hydroxydopamine treatment: effects of dose levels on behavioral parameters. Behavioural Brain Research. 82(2). 213–221. 26 indexed citations

Sandberg, Mats, et al.. (1995). Visualization of the NMDA recognition site in rat and mouse spinal cord by [3H]CGS 19755in vitro autoradiography. Amino Acids. 9(3). 247–263. 2 indexed citations

10.

Andersson, Hans, et al.. (1995). Trimethyltin-induced loss of NMDA and kainate receptors in the rat brain. Amino Acids. 8(1). 23–35. 9 indexed citations

11.

Willson, Christopher A., Johan Luthman, Barry J. Hoffer, & Israel Hanin. (1995). The use of the rat iris as a model system to evaluate the effect of the cholinotoxin, AF64A, in vivo. Journal of Neuroscience Methods. 56(1). 49–55. 1 indexed citations

12.

Leonard, Sherry, et al.. (1993). Acidic and basic fibroblast growth factor mRNAs are increased in striatum following MPTP-induced dopamine neurofiber lesion: assay by quantitative PCR. Molecular Brain Research. 18(4). 275–284. 34 indexed citations

13.

Luthman, Johan, M Friedemann, Paula C. Bickford, et al.. (1993). In vivo electrochemical measurements and electrophysiological studies of rat striatum following neonatal 6-hydroxydopamine treatment. Neuroscience. 52(3). 677–687. 33 indexed citations

14.

Modéer, Thomas, et al.. (1991). Immunohistological study of neuronal markers in inflamed gingiva obtained from children with Down's syndrome. Journal Of Clinical Periodontology. 18(8). 624–633. 14 indexed citations

15.

Luthman, Johan, et al.. (1990). Plant‐Derived Neurotoxic Amino Acids (β‐N‐Oxalylamino‐l‐Alanine and β‐N‐Methylamino‐l‐Alanine): Effects on Central Monoamine Neurons. Journal of Neurochemistry. 55(3). 941–949. 16 indexed citations

16.

Luthman, Johan, et al.. (1990). No apparent difference in the effects of 1 -methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) on the sympathetic system in NMRI and C57 BL/6 mice. Toxicology Letters. 54(1). 83–92. 8 indexed citations

17.

Luthman, Johan, Elisabet Lindqvist, David A. Young, & Richard F. Cowburn. (1990). Neonatal dopamine lesion in the rat results in enhanced adenylate cyclase activity without altering dopamine receptor binding or dopamine- and adenosine 3??5?-monophosphate-regulated phosphoprotein (DARPP-32) immunoreactivity. Experimental Brain Research. 83(1). 85–95. 52 indexed citations

18.

Hedin, Ulf, Bradford A. Bottger, Johan Luthman, Staffan Johansson, & Johan Thyberg. (1989). A substrate of the cell-attachment sequence of fibronectin (Arg-Gly-Asp-Ser) is sufficient to promote transition of arterial smooth muscle cells from a contractile to a synthetic phenotype. Developmental Biology. 133(2). 489–501. 88 indexed citations

19.

Luthman, Johan, Håkan Aldskogius, Mikael Svensson, & G. Jönsson. (1988). Exogenous GM1 ganglioside and thyrotropin-releasing hormone do not affect survival rate of spinal motorneurons and number of ventral root myelinated fibers following early postnatal sciatic nerve transection. Experimental Neurology. 99(1). 166–177. 7 indexed citations

20.

Sundström, Erik, et al.. (1988). Time course of MPTP-induced degeneration of the nigrostriatal dopamine system in C57 BL/6 mice. Brain Research Bulletin. 21(2). 257–263. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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