Jeremy Z. Fields

10.4k total citations · 1 hit paper
143 papers, 8.6k citations indexed

About

Jeremy Z. Fields is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Oncology. According to data from OpenAlex, Jeremy Z. Fields has authored 143 papers receiving a total of 8.6k indexed citations (citations by other indexed papers that have themselves been cited), including 58 papers in Molecular Biology, 29 papers in Cellular and Molecular Neuroscience and 28 papers in Oncology. Recurrent topics in Jeremy Z. Fields's work include Receptor Mechanisms and Signaling (18 papers), Cancer Cells and Metastasis (16 papers) and Neuroscience and Neuropharmacology Research (16 papers). Jeremy Z. Fields is often cited by papers focused on Receptor Mechanisms and Signaling (18 papers), Cancer Cells and Metastasis (16 papers) and Neuroscience and Neuropharmacology Research (16 papers). Jeremy Z. Fields collaborates with scholars based in United States, United Kingdom and Russia. Jeremy Z. Fields's co-authors include Ali Keshavarzian, Bruce M. Boman, Ashkan Farhadi, Ali Banan, Henry I. Yamamura, A. Banan, Terry Reisine, Seema Bhatlekar, Christopher B. Forsyth and Maliha Shaikh and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Journal of Biological Chemistry.

In The Last Decade

Jeremy Z. Fields

139 papers receiving 8.3k citations

Hit Papers

Aldehyde Dehydrogenase 1 Is a Marker for Normal and Malig... 2009 2026 2014 2020 2009 250 500 750

Peers

Jeremy Z. Fields
Daniel C. Anthony United Kingdom
Puttur D. Prasad United States
Feng Li China
Partha Mukhopadhyay United States
Muniswamy Madesh United States
Wendy B. Bollag United States
Jeremy Z. Fields
Citations per year, relative to Jeremy Z. Fields Jeremy Z. Fields (= 1×) peers Mariko Saito

Countries citing papers authored by Jeremy Z. Fields

Since Specialization
Citations

This map shows the geographic impact of Jeremy Z. Fields's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jeremy Z. Fields with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jeremy Z. Fields more than expected).

Fields of papers citing papers by Jeremy Z. Fields

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jeremy Z. Fields. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jeremy Z. Fields. The network helps show where Jeremy Z. Fields may publish in the future.

Co-authorship network of co-authors of Jeremy Z. Fields

This figure shows the co-authorship network connecting the top 25 collaborators of Jeremy Z. Fields. A scholar is included among the top collaborators of Jeremy Z. Fields based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jeremy Z. Fields. Jeremy Z. Fields is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Boman, Bruce M., Vignesh Viswanathan, Caroline O. Facey, Jeremy Z. Fields, & James W. Stave. (2023). The v8-10 variant isoform of CD44 is selectively expressed in the normal human colonic stem cell niche and frequently is overexpressed in colon carcinomas during tumor development. Cancer Biology & Therapy. 24(1). 2195363–2195363. 10 indexed citations
2.
Viswanathan, Vignesh, Shirish Damle, Tao Zhang, et al.. (2017). An miRNA Expression Signature for the Human Colonic Stem Cell Niche Distinguishes Malignant from Normal Epithelia. Cancer Research. 77(14). 3778–3790. 16 indexed citations
3.
Sugimori, Michiya, Yumiko Hayakawa, Bruce M. Boman, et al.. (2015). Discovery of Power-Law Growth in the Self-Renewal of Heterogeneous Glioma Stem Cell Populations. PLoS ONE. 10(8). e0135760–e0135760. 14 indexed citations
4.
Zhang, Tao, Jeremy Z. Fields, Emi Masuda, et al.. (2010). Survivin-Induced Aurora-B Kinase Activation. American Journal Of Pathology. 177(6). 2816–2826. 32 indexed citations
5.
Cifra, Michal, Jeremy Z. Fields, & Ashkan Farhadi. (2010). Electromagnetic cellular interactions. Progress in Biophysics and Molecular Biology. 105(3). 223–246. 239 indexed citations
6.
Singhal, Shashideep, et al.. (2010). The Role of Oral Hygiene in Inflammatory Bowel Disease. Digestive Diseases and Sciences. 56(1). 170–175. 54 indexed citations
7.
Boman, Bruce M., et al.. (2008). How Dysregulated Colonic Crypt Dynamics Cause Stem Cell Overpopulation and Initiate Colon Cancer. Cancer Research. 68(9). 3304–3313. 87 indexed citations
9.
Walton, Kenneth G., et al.. (2004). Lowering Cortisol and CVD Risk in Postmenopausal Women: A Pilot Study Using the Transcendental Meditation Program. Annals of the New York Academy of Sciences. 1032(1). 211–215. 44 indexed citations
10.
Fields, Jeremy Z., et al.. (2003). The Maharishi Vedic Medicine Chronic Disorders Program: Introduction and Case Histories. Alternative and Complementary Therapies. 9(4). 183–190. 1 indexed citations
11.
Keshavarzian, Ali, et al.. (1999). Leaky Gut in Alcoholic Cirrhosis: A Possible Mechanism for Alcohol-Induced Liver Damage. The American Journal of Gastroenterology. 94(1). 200–207. 302 indexed citations
12.
Fields, Jeremy Z., et al.. (1997). Regulation of neutrophils in ulcerative colitis by colonic factors: A possible mechanism of neutrophil activation and tissue damage. Journal of Laboratory and Clinical Medicine. 130(6). 590–602. 21 indexed citations
13.
Winship, Daniel H., et al.. (1996). The Role of Nitric Oxide in Ethanol‐Induced Gastrointestinal Dysfunction. Alcoholism Clinical and Experimental Research. 20(9). 1618–1624. 7 indexed citations
14.
Drucker, George E., et al.. (1994). Prevention and reversal of dopamine receptor supersensitivity by cyclo(leucyl-glycyl) (CLG): Biphasic dose-response curves. Pharmacology Biochemistry and Behavior. 47(1). 141–145. 7 indexed citations
15.
Keshavarzian, A., et al.. (1991). Chronic ethanol (EtOH) feeding increases muscarinic receptor (mAChR) density in esophagus without parallel change in dose response (D-R) to cholinergic agonists. 1 indexed citations
16.
Fields, Jeremy Z., et al.. (1991). Long-lasting dopamine receptor up-regulation in amphetamine-treated rats following amphetamine neurotoxicity. Pharmacology Biochemistry and Behavior. 40(4). 881–886. 9 indexed citations
17.
Keshavarzian, A., A. A. E. Wibowo, J.H. Gordon, & Jeremy Z. Fields. (1990). MPTP-induced duodenal ulcers in rat. Prevention by reuptake blockers for serotonin and norepinephrine, but not dopamine.. PubMed. 98(3). 554–60. 8 indexed citations
18.
Fields, Jeremy Z., et al.. (1982). Modification of morphine-induced change in striatal (3H)-spiroperidol binding and stereotype behavior by cyclo(Leu-Gly). Life Sciences. 30(18). 1573–1580. 24 indexed citations
19.
Fields, Jeremy Z., William R. Roeske, Eugene Morkin, & Henry I. Yamamura. (1977). Biochemical demonstration of muscarinic cholinergic receptors in mammalian heart. Federation Proceedings. 36(3). 2 indexed citations
20.
Roeske, William R., Jeremy Z. Fields, Eugene Morkin, & Henry I. Yamamura. (1977). Myocardial muscarinic cholinergic receptors. Clinical research. 25(3). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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