Javier Jimenez‐Feltström

484 total citations
11 papers, 401 citations indexed

About

Javier Jimenez‐Feltström is a scholar working on Surgery, Endocrinology, Diabetes and Metabolism and Physiology. According to data from OpenAlex, Javier Jimenez‐Feltström has authored 11 papers receiving a total of 401 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Surgery, 6 papers in Endocrinology, Diabetes and Metabolism and 6 papers in Physiology. Recurrent topics in Javier Jimenez‐Feltström's work include Pancreatic function and diabetes (10 papers), Adipose Tissue and Metabolism (5 papers) and Diet, Metabolism, and Disease (4 papers). Javier Jimenez‐Feltström is often cited by papers focused on Pancreatic function and diabetes (10 papers), Adipose Tissue and Metabolism (5 papers) and Diet, Metabolism, and Disease (4 papers). Javier Jimenez‐Feltström collaborates with scholars based in Sweden and Japan. Javier Jimenez‐Feltström's co-authors include Albert Salehi, Ingmar Lundquist, Erik Flodgren, Christer Owman, Björn Olde, Jun‐ichi Miyazaki, Niclas Nilsson, Per Olof Nyman, Lena Eliasson and Jenny Vikman and has published in prestigious journals such as PLoS ONE, The FASEB Journal and Endocrinology.

In The Last Decade

Javier Jimenez‐Feltström

11 papers receiving 397 citations

Peers

Javier Jimenez‐Feltström
Javier Jimenez‐Feltström
Citations per year, relative to Javier Jimenez‐Feltström Javier Jimenez‐Feltström (= 1×) peers T. Shirotani

Countries citing papers authored by Javier Jimenez‐Feltström

Since Specialization
Citations

This map shows the geographic impact of Javier Jimenez‐Feltström's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Javier Jimenez‐Feltström with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Javier Jimenez‐Feltström more than expected).

Fields of papers citing papers by Javier Jimenez‐Feltström

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Javier Jimenez‐Feltström. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Javier Jimenez‐Feltström. The network helps show where Javier Jimenez‐Feltström may publish in the future.

Co-authorship network of co-authors of Javier Jimenez‐Feltström

This figure shows the co-authorship network connecting the top 25 collaborators of Javier Jimenez‐Feltström. A scholar is included among the top collaborators of Javier Jimenez‐Feltström based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Javier Jimenez‐Feltström. Javier Jimenez‐Feltström is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
2.
Mosén, Henrik, Claes-Göran Östenson, Ingmar Lundquist, et al.. (2008). Impaired glucose-stimulated insulin secretion in the GK rat is associated with abnormalities in islet nitric oxide production. Regulatory Peptides. 151(1-3). 139–146. 22 indexed citations
3.
Salehi, Albert, et al.. (2008). Excessive Islet NO Generation in Type 2 Diabetic GK Rats Coincides with Abnormal Hormone Secretion and Is Counteracted by GLP-1. PLoS ONE. 3(5). e2165–e2165. 44 indexed citations
5.
Vikman, Jenny, Javier Jimenez‐Feltström, Per Olof Nyman, & Lena Eliasson. (2008). Insulin secretion is highly sensitive to desorption of plasma membrane cholesterol. The FASEB Journal. 23(1). 58–67. 65 indexed citations
6.
Qader, Saleem S., Javier Jimenez‐Feltström, Mats Ekelund, Ingmar Lundquist, & Albert Salehi. (2007). Expression of islet inducible nitric oxide synthase and inhibition of glucose-stimulated insulin release after long-term lipid infusion in the rat is counteracted by PACAP27. American Journal of Physiology-Endocrinology and Metabolism. 292(5). E1447–E1455. 15 indexed citations
7.
Ekelund, Mats, Saleem S. Qader, Javier Jimenez‐Feltström, & Albert Salehi. (2006). Selective induction of inducible nitric oxide synthase in pancreatic islet of rat after an intravenous glucose or intralipid challenge. Nutrition. 22(6). 652–660. 6 indexed citations
8.
Salehi, Albert, Erik Flodgren, Niclas Nilsson, et al.. (2005). Free fatty acid receptor 1 (FFA1R/GPR40) and its involvement in fatty-acid-stimulated insulin secretion. Cell and Tissue Research. 322(2). 207–215. 126 indexed citations
9.
Mosén, Henrik, Albert Salehi, Per Alm, et al.. (2004). Defective Glucose-Stimulated Insulin Release in the Diabetic Goto-Kakizaki (GK) Rat Coincides with Reduced Activity of the Islet Carbon Monoxide Signaling Pathway. Endocrinology. 146(3). 1553–1558. 37 indexed citations
10.
Jimenez‐Feltström, Javier, Ingmar Lundquist, Stefanie Obermüller, & Albert Salehi. (2004). Insulin feedback actions: complex effects involving isoforms of islet nitric oxide synthase. Regulatory Peptides. 122(2). 109–118. 31 indexed citations
11.
Jimenez‐Feltström, Javier, Ingmar Lundquist, & Albert Salehi. (2004). Glucose stimulates the expression and activities of nitric oxide synthases in incubated rat islets: an effect counteracted by GLP-1 through the cyclic AMP/PKA pathway. Cell and Tissue Research. 319(2). 221–230. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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