Jane Palsgaard

506 total citations
7 papers, 327 citations indexed

About

Jane Palsgaard is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Physiology. According to data from OpenAlex, Jane Palsgaard has authored 7 papers receiving a total of 327 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Endocrinology, Diabetes and Metabolism and 3 papers in Physiology. Recurrent topics in Jane Palsgaard's work include Metabolism, Diabetes, and Cancer (5 papers), Muscle Physiology and Disorders (3 papers) and Growth Hormone and Insulin-like Growth Factors (3 papers). Jane Palsgaard is often cited by papers focused on Metabolism, Diabetes, and Cancer (5 papers), Muscle Physiology and Disorders (3 papers) and Growth Hormone and Insulin-like Growth Factors (3 papers). Jane Palsgaard collaborates with scholars based in Denmark, United Kingdom and United States. Jane Palsgaard's co-authors include Pierre De Meyts, Rehannah Borup, Maja Jensen, Brice Emanuelli, Jonathon N. Winnay, Gérard Karsenty, Grzegorz Sumara, C. Ronald Kahn, Audrey E. Brown and Martin Friedrichsen and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Biochemical Journal.

In The Last Decade

Jane Palsgaard

7 papers receiving 325 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jane Palsgaard Denmark 7 222 105 83 42 42 7 327
Daisuke Yamanaka Japan 11 191 0.9× 87 0.8× 80 1.0× 34 0.8× 41 1.0× 24 366
Alfred K. Ramirez United States 6 188 0.8× 103 1.0× 46 0.6× 42 1.0× 50 1.2× 6 305
Tanner Boes United States 5 218 1.0× 162 1.5× 69 0.8× 25 0.6× 83 2.0× 5 385
Amelia Y.I. Viana Japan 7 268 1.2× 79 0.8× 68 0.8× 102 2.4× 91 2.2× 7 418
Carolina Nylén Sweden 9 286 1.3× 163 1.6× 79 1.0× 109 2.6× 50 1.2× 14 450
Maneet Singh United States 9 186 0.8× 164 1.6× 41 0.5× 72 1.7× 52 1.2× 11 392
Evan P. Taddeo United States 11 291 1.3× 131 1.2× 40 0.5× 76 1.8× 71 1.7× 14 411
Seokjeong Yoon South Korea 6 233 1.0× 112 1.1× 47 0.6× 106 2.5× 30 0.7× 8 349
Yunzhe Bai Japan 10 236 1.1× 62 0.6× 56 0.7× 28 0.7× 19 0.5× 11 378

Countries citing papers authored by Jane Palsgaard

Since Specialization
Citations

This map shows the geographic impact of Jane Palsgaard's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jane Palsgaard with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jane Palsgaard more than expected).

Fields of papers citing papers by Jane Palsgaard

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jane Palsgaard. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jane Palsgaard. The network helps show where Jane Palsgaard may publish in the future.

Co-authorship network of co-authors of Jane Palsgaard

This figure shows the co-authorship network connecting the top 25 collaborators of Jane Palsgaard. A scholar is included among the top collaborators of Jane Palsgaard based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jane Palsgaard. Jane Palsgaard is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Brown, Audrey E., Jane Palsgaard, Rehannah Borup, et al.. (2014). p38 MAPK activation upregulates proinflammatory pathways in skeletal muscle cells from insulin-resistant type 2 diabetic patients. American Journal of Physiology-Endocrinology and Metabolism. 308(1). E63–E70. 46 indexed citations
2.
Versteyhe, Soetkin, Rehannah Borup, Jane Palsgaard, et al.. (2013). IGF-I, IGF-II, and Insulin Stimulate Different Gene Expression Responses through Binding to the IGF-I Receptor. Frontiers in Endocrinology. 4. 98–98. 34 indexed citations
3.
Palsgaard, Jane, Brice Emanuelli, Jonathon N. Winnay, et al.. (2012). Cross-talk between Insulin and Wnt Signaling in Preadipocytes. Journal of Biological Chemistry. 287(15). 12016–12026. 79 indexed citations
4.
Palsgaard, Jane, Charlotte Brøns, Martin Friedrichsen, et al.. (2009). Gene Expression in Skeletal Muscle Biopsies from People with Type 2 Diabetes and Relatives: Differential Regulation of Insulin Signaling Pathways. PLoS ONE. 4(8). e6575–e6575. 84 indexed citations
5.
Palsgaard, Jane, Audrey E. Brown, Maja Jensen, et al.. (2008). Insulin-like growth factor I (IGF-I) is a more potent regulator of gene expression than insulin in primary human myoblasts and myotubes. Growth Hormone & IGF Research. 19(2). 168–178. 20 indexed citations
6.
Jensen, Maja, Jane Palsgaard, Rehannah Borup, Pierre De Meyts, & Lauge Schäffer. (2008). Activation of the insulin receptor (IR) by insulin and a synthetic peptide has different effects on gene expression in IR-transfected L6 myoblasts. Biochemical Journal. 412(3). 435–445. 29 indexed citations
7.
Meyts, Pierre De, et al.. (2004). Structural Biology of Insulin and IGF‐1 Receptors. Novartis Foundation symposium. 262. 160–176. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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