Jana Nekvindová
- Molecular Biology top 10%
- Cancer Research top 2%
- Oncology
- Pulmonary and Respiratory Medicine
- Cell Biology top 10%
- Co-authors
- Ondřej SlabýLenka RadováRostislav VyzulaMarek SvobodaRadek LakomýMartina RédováAlexandr PoprachRobert Iliev
- Topics
- Plant Pathogens and Fungal Diseases (10 papers)MicroRNA in disease regulation (9 papers)Pharmacogenetics and Drug Metabolism (8 papers)
- Partner nations
- CzechiaUnited StatesNetherlands
In The Last Decade
Jana Nekvindová
47 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 92
- Molecular Biology 846
- Cancer Research 672
- Oncology 193
- Pulmonary and Respiratory Medicine 158
- Cell Biology 140
Countries citing papers authored by Jana Nekvindová
This map shows the geographic impact of Jana Nekvindová's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jana Nekvindová with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jana Nekvindová more than expected).
Fields of papers citing papers by Jana Nekvindová
This network shows the impact of papers produced by Jana Nekvindová. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jana Nekvindová. The network helps show where Jana Nekvindová may publish in the future.
Co-authorship network of co-authors of Jana Nekvindová
This figure shows the co-authorship network connecting the top 25 collaborators of Jana Nekvindová. A scholar is included among the top collaborators of Jana Nekvindová based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jana Nekvindová. Jana Nekvindová is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 9 | |
| 2 | 17 | |
| 3 | 38 | |
| 4 | 30 | |
| 5 | 30 | |
| 6 | 7 | |
| 7 | 54 | |
| 8 | 17 | |
| 9 | 18 | |
| 10 | 30 | |
| 11 | 93 | |
| 12 | 19 | |
| 13 | 119 | |
| 14 | 30 | |
| 15 | 37 | |
| 16 | 24 | |
| 17 | 16 | |
| 18 | 4 | |
| 19 | Experimental approaches to studies on drug metabolism and drug interactions in man: interaction of acyclic nucleoside phosphonates with human liver cytochromes P450 and flavin-containing monooxygenase 3. | 4 |
| 20 | 18 |
About Jana Nekvindová
Jana Nekvindová is a scholar working on Pharmacology, Cancer Research and Cell Biology, having authored 48 papers that have together received 1.4k indexed citations. Recurring topics across this work include Plant Pathogens and Fungal Diseases (10 papers), MicroRNA in disease regulation (9 papers) and Pharmacogenetics and Drug Metabolism (8 papers). The work is most often cited by research in Cancer Research (672 citations), Otorhinolaryngology (57 citations) and Molecular Biology (846 citations). Jana Nekvindová has collaborated with scholars based in Czechia, United States and Netherlands. Frequent co-authors include Ondřej Slabý, Lenka Radová, Rostislav Vyzula, Marek Svoboda, Radek Lakomý, Martina Rédová, Alexandr Poprach, Robert Iliev, Martina Réblová and Pavel Fabián. Their work appears in journals such as Blood, PLoS ONE and Biochemical Pharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.