Jana Löster

1.3k total citations
25 papers, 1.0k citations indexed

About

Jana Löster is a scholar working on Molecular Biology, Genetics and Cell Biology. According to data from OpenAlex, Jana Löster has authored 25 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 8 papers in Genetics and 5 papers in Cell Biology. Recurrent topics in Jana Löster's work include Connexins and lens biology (18 papers), RNA regulation and disease (5 papers) and Yersinia bacterium, plague, ectoparasites research (5 papers). Jana Löster is often cited by papers focused on Connexins and lens biology (18 papers), RNA regulation and disease (5 papers) and Yersinia bacterium, plague, ectoparasites research (5 papers). Jana Löster collaborates with scholars based in Germany, Switzerland and United Kingdom. Jana Löster's co-authors include Jochen Graw, Martin Biel, Stefan Herrmann, Robert Feil, Franz Hofmann, A. Ludwig, Susanne Feil, Juliane Stieber, Norman Klopp and Rudi Balling and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The EMBO Journal and Genetics.

In The Last Decade

Jana Löster

24 papers receiving 996 citations

Peers

Countries citing papers authored by Jana Löster

Since Specialization

Citations

This map shows the geographic impact of Jana Löster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jana Löster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jana Löster more than expected).

Fields of papers citing papers by Jana Löster

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jana Löster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jana Löster. The network helps show where Jana Löster may publish in the future.

Co-authorship network of co-authors of Jana Löster

This figure shows the co-authorship network connecting the top 25 collaborators of Jana Löster. A scholar is included among the top collaborators of Jana Löster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jana Löster. Jana Löster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Developmental genetics in ophthalmology 2003 ·Ophthalmic Genetics ·Jochen Graw, Jana Löster	45
2	The hyperpolarization-activated channel HCN4 is required for the generation of pacemaker action potentials in the embryonic heart 2003 ·Proceedings of the National Academy of Sciences ·Juliane Stieber, Stefan Herrmann, Susanne Feil, Jana Löster, Robert Feil, Martin Biel, Franz Hofmann, A. Ludwig	359
3	V76D mutation in a conserved gD-crystallin region leads to dominant cataracts in mice 2002 ·Mammalian Genome ·Jochen Graw, Jana Löster, Dian Soewarto, Helmut Fuchs, André Reis, Eckhard Wolf, Rudi Balling, Martin Hrabě de Angelis	23
4	Altered aggregation properties of mutant γ-crystallins cause inherited cataract 2002 ·The EMBO Journal ·Aileen Sandilands, Aileen M. Hutcheson, Heather A. Long, Alan R. Prescott, Gijs F.J.M. Vrensen, Jana Löster, Norman Klopp, (unknown), Jochen Graw, Shigeo Masaki, Christopher M. Dobson, Cait E. MacPhee, Roy A. Quinlan	124
5	ALTERED AGGREGATION PROPERTIES OF M-CRYSTALLINS CAUSE INHERITED CATARACT 2002 ·Aileen Sandilands, A. M. Hutcheson, Heather A. Long, (unknown), Gijs F.J.M. Vrensen, Jana Löster, N. Klopp, (unknown), (unknown), (unknown), Cait E. MacPhee, (unknown)	1
6	Mutually regulated expression of Pax6 and Six3 and its implications for the Pax6 haploinsufficient lens phenotype 2002 ·Proceedings of the National Academy of Sciences ·Guy Goudreau, Petros Petrou, Lixing W. Reneker, Jochen Graw, Jana Löster, Peter Gruß	74
7	Characterization of a Mutation in the Lens-specific MP70 Encoding Gene of the Mouse Leading to a Dominant Cataract 2001 ·Experimental Eye Research ·Jochen Graw, Jana Löster, Dian Soewarto, Helmut Fuchs, Birgit Meyer, André Reis, Eckhard Wolf, Rudi Balling, Martin Hrabě de Angelis	47
8	Ethylnitrosourea-Induced Mutation in Mice Leads to the Expression of a Novel Protein in the Eye and to Dominant Cataracts 2001 ·Genetics ·Jochen Graw, Norman Klopp, Jana Löster, Dian Soewarto, Helmut Fuchs, (unknown), André Reis, Eckhard Wolf, Rudi Balling, Martin Hrabě de Angelis	16
9	Characterization of a new, dominant V124E mutation in the mouse alphaA-crystallin-encoding gene. 2001 ·PubMed ·Jochen Graw, Jana Löster, Dian Soewarto, Helmut Fuchs, Birgit Meyer, André Reis, Eckhard Wolf, Rudi Balling, Martin Hrabě de Angelis	38
10	Aey2, a new mutation in the betaB2-crystallin-encoding gene of the mouse. 2001 ·PubMed ·Jochen Graw, Jana Löster, Dian Soewarto, Helmut Fuchs, André Reis, Eckhard Wolf, Rudi Balling, Martin Hrabě de Angelis	42
11	Characterization of a 1-bp deletion in the gammaE-crystallin gene leading to a nuclear and zonular cataract in the mouse. 2001 ·PubMed ·Norman Klopp, Jana Löster, Jochen Graw	16
12	Mutation in the βA3/A1-Crystallin Encoding Gene Cryba1 Causes a Dominant Cataract in the Mouse 1999 ·Genomics ·Jochen Graw, Martin Jung, Jana Löster, (unknown), Dian Soewarto, (unknown), Helmut Fuchs, André Reis, Eckhard Wolf, Rudi Balling, Martin Hrabě de Angelis	39
13	Three Murine Cataract Mutants (Cat2) Are Defective in Different γ-Crystallin Genes 1998 ·Genomics ·(unknown), Jack Favor, Jana Löster, (unknown), A. Neuhäuser-Klaus, Alan R. Prescott, Walter Pretsch, Roy A. Quinlan, Aileen Sandilands, Gijs F.J.M. Vrensen, Jochen Graw	57
14	Aphakia (ak), a mouse mutation affecting early eye development: Fine mapping, consideration of candidate genes and alteredPax6 andSix3 gene expression pattern 1998 ·Developmental Genetics ·Christina Grimm, Bimal Chatterjee, Jack Favor, Thomas Immervoll, Jana Löster, Norman Klopp, Rodica Sandulache, Jochen Graw	33
15	Cat3 vl and Cat3 vao cataract mutations on mouse chromosome 10: phenotypic characterization, linkage studies and analysis of candidate genes 1997 ·Molecular and General Genetics MGG ·Jana Löster, Thomas Immervoll, Thomas Schmitt‐John, Jochen Graw	6
16	Molecular analysis of two new Steel mutations in mice shows a transversion or an insertion 1996 ·Mammalian Genome ·Jochen Graw, Jana Löster, A. Neuhäuser-Klaus, Walter Pretsch, Thomas Schmitt‐John	6
17	Sequence analysis of the βB2-crystallin cDNA of hamster containing a domain conserved among vertebrates 1996 ·Gene ·Konstantinos Zarbalis, Bimal Chatterjee, Jana Löster, Thomas Werner, Jochen Graw	2
18	P 206 CAT3, new cataract gene on mouse chromosome 10: Linkage studies and phenotypic characterization 1995 ·Vision Research ·Jana Löster, A. Neuhäuser-Klaus, Eva Schäffer, Thomas Schmitt‐John, Jochen Graw	1
19	Reduced levels of γ-crystallin transcripts during embryonic development of murine Cat2 nop mutant lenses 1995 ·Graefe s Archive for Clinical and Experimental Ophthalmology ·Sathiyavedu Thyagarajan Santhiya, (unknown), Jana Löster, Jochen Graw	19
20	Close Linkage of the Dominant Cataract Mutations (Cat-2) with Idh-1 and Cryge on Mouse Chromosome 1 1994 ·Genomics ·Jana Löster, Walter Pretsch, Rodica Sandulache, Thomas Schmitt‐John, Mary F. Lyon, Jochen Graw	12

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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