Jan S. Schulte

1.1k total citations
41 papers, 758 citations indexed

About

Jan S. Schulte is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Jan S. Schulte has authored 41 papers receiving a total of 758 indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 26 papers in Cardiology and Cardiovascular Medicine and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Jan S. Schulte's work include Cardiac electrophysiology and arrhythmias (22 papers), Ion channel regulation and function (20 papers) and Cardiomyopathy and Myosin Studies (6 papers). Jan S. Schulte is often cited by papers focused on Cardiac electrophysiology and arrhythmias (22 papers), Ion channel regulation and function (20 papers) and Cardiomyopathy and Myosin Studies (6 papers). Jan S. Schulte collaborates with scholars based in Germany, United States and United Kingdom. Jan S. Schulte's co-authors include Frank U. Müller, K. Quitzsch, Stefan Dhein, Frank Müller, Sabine Enders, Matthias D. Seidl, Uwe Kirchhefer, Alexander Heinick, Wilhelm Schmitz and Lars Eckardt and has published in prestigious journals such as Journal of Biological Chemistry, The FASEB Journal and Endocrinology.

In The Last Decade

Jan S. Schulte

37 papers receiving 751 citations

Peers

Jan S. Schulte
Jan S. Schulte
Citations per year, relative to Jan S. Schulte Jan S. Schulte (= 1×) peers Jocelyn Bescond

Countries citing papers authored by Jan S. Schulte

Since Specialization
Citations

This map shows the geographic impact of Jan S. Schulte's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jan S. Schulte with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jan S. Schulte more than expected).

Fields of papers citing papers by Jan S. Schulte

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jan S. Schulte. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jan S. Schulte. The network helps show where Jan S. Schulte may publish in the future.

Co-authorship network of co-authors of Jan S. Schulte

This figure shows the co-authorship network connecting the top 25 collaborators of Jan S. Schulte. A scholar is included among the top collaborators of Jan S. Schulte based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jan S. Schulte. Jan S. Schulte is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schulte, Jan S., Désirée Schnidrig, Sabine Schneider, et al.. (2025). A novel alveoli-on-chip platform for modeling cyclic stretch in patient-derived alveolar epithelial cells cultured from organoids. Lab on a Chip. 25(24). 6688–6702.
2.
Reinhardt, Jürgen, et al.. (2025). Cre recombinase affects calcium dynamics already in young mice. Frontiers in Pharmacology. 16. 1558573–1558573. 1 indexed citations
3.
Pluteanu, Florentina, et al.. (2024). Loss of protein phosphatase 2A regulatory subunit PPP2R5A is associated with increased incidence of stress-induced proarrhythmia. Frontiers in Cardiovascular Medicine. 11. 1419597–1419597.
4.
Schulte, Jan S., et al.. (2023). Lung-on-a-Chip Models of the Lung Parenchyma. Advances in experimental medicine and biology. 1413. 191–211. 2 indexed citations
5.
Heinick, Alexander, Frank U. Müller, Timofey S. Rozhdestvensky, et al.. (2022). Impaired myocellular Ca2+ cycling in protein phosphatase PP2A-B56α KO mice is normalized by β-adrenergic stimulation. Journal of Biological Chemistry. 298(9). 102362–102362. 4 indexed citations
6.
Bögeholz, Nils, Jan S. Schulte, Joshua I. Goldhaber, et al.. (2022). Increased in vivo perpetuation of whole-heart ventricular arrhythmia in heterozygous Na+/Ca2+ exchanger knockout mice. IJC Heart & Vasculature. 44. 101168–101168. 2 indexed citations
7.
Bögeholz, Nils, Jan S. Schulte, Joshua I. Goldhaber, et al.. (2022). Increased in Vivo Perpetuation of Whole-Heart Ventricular Arrhythmia in Heterozygous Na+/Ca2+ Exchanger Knockout Mice. SSRN Electronic Journal. 1 indexed citations
8.
Wiedmann, Felix, Jan S. Schulte, Maria-Patapia Zafeiriou, et al.. (2018). Atrial fibrillation and heart failure-associated remodeling of two-pore-domain potassium (K2P) channels in murine disease models: focus on TASK-1. Basic Research in Cardiology. 113(4). 27–27. 29 indexed citations
9.
Bögeholz, Nils, Dirk G. Dechering, Gerrit Frommeyer, et al.. (2018). Distinct Occurrence of Proarrhythmic Afterdepolarizations in Atrial Versus Ventricular Cardiomyocytes: Implications for Translational Research on Atrial Arrhythmia. Frontiers in Pharmacology. 9. 933–933. 8 indexed citations
10.
Bukowska, Alicja, Carmen Wolke, Jan S. Schulte, et al.. (2017). CREM-transgene mice: An animal model of atrial fibrillation and thrombogenesis. Thrombosis Research. 163. 172–179. 14 indexed citations
11.
Bögeholz, Nils, Jan S. Schulte, Sven Kaese, et al.. (2017). The Effects of SEA0400 on Ca2+ Transient Amplitude and Proarrhythmia Depend on the Na+/Ca2+ Exchanger Expression Level in Murine Models. Frontiers in Pharmacology. 8. 649–649. 8 indexed citations
12.
Schulte, Jan S., Benedikt Fels, Xander H.T. Wehrens, et al.. (2016). Cardiac expression of the CREM repressor isoform CREM-IbΔC-X in mice leads to arrhythmogenic alterations in ventricular cardiomyocytes. Basic Research in Cardiology. 111(2). 15–15. 22 indexed citations
13.
Bögeholz, Nils, Sven Kaese, Jan S. Schulte, et al.. (2016). Triggered activity in atrial myocytes is influenced by Na+/Ca2+ exchanger activity in genetically altered mice. Journal of Molecular and Cellular Cardiology. 101. 106–115. 15 indexed citations
14.
Schulte, Jan S., et al.. (2014). The Role of Transcription Factors in the Formation of an Arrhythmogenic Substrate in Congestive Human Heart Failure. Current Medicinal Chemistry. 21(11). 1281–1298. 1 indexed citations
15.
Stegemann, Agatha, Anca Sindrilaru, Beate Eckes, et al.. (2012). Tropisetron suppresses collagen synthesis in skin fibroblasts via α7 nicotinic acetylcholine receptor and attenuates fibrosis in a scleroderma mouse model. Arthritis & Rheumatism. 65(3). 792–804. 30 indexed citations
16.
Pott, Christian, Nils Bögeholz, Jan S. Schulte, et al.. (2012). Proarrhythmia in a non-failing murine model of cardiac-specific Na+/Ca2+ exchanger overexpression: whole heart and cellular mechanisms. Basic Research in Cardiology. 107(2). 247–247. 34 indexed citations
17.
Schulte, Jan S., et al.. (2008). Neonatal Rat Cardiomyocytes Show Characteristics of Nonhomotypic Gap Junction Channels. Cell Communication & Adhesion. 15(1-2). 13–25. 6 indexed citations
18.
Dhein, Stefan, Nicole Duerrschmidt, Andreas Boldt, et al.. (2008). A new role for extracellular Ca2+ in gap-junction remodeling: studies in humans and rats. Naunyn-Schmiedeberg s Archives of Pharmacology. 377(2). 125–138. 12 indexed citations
19.
Schulte, Jan S., et al.. (2008). Alpha-1-adrenoceptor subtype selective regulation of connexin 43 expression in rat cardiomyocytes. Naunyn-Schmiedeberg s Archives of Pharmacology. 377(1). 77–85. 16 indexed citations
20.
Kochs, E., et al.. (1991). THE EFFECTS OF SEVOFLURANE ON NEUROLOGIC OUTCOME FROM INCOMPLETE ISCHEMIA IN RATS. Journal of Neurosurgical Anesthesiology. 3(3). 237–237.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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