James S. Woods

6.3k total citations
143 papers, 4.8k citations indexed

About

James S. Woods is a scholar working on Health, Toxicology and Mutagenesis, Molecular Biology and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, James S. Woods has authored 143 papers receiving a total of 4.8k indexed citations (citations by other indexed papers that have themselves been cited), including 60 papers in Health, Toxicology and Mutagenesis, 58 papers in Molecular Biology and 29 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in James S. Woods's work include Mercury impact and mitigation studies (45 papers), Heavy Metal Exposure and Toxicity (42 papers) and Heme Oxygenase-1 and Carbon Monoxide (37 papers). James S. Woods is often cited by papers focused on Mercury impact and mitigation studies (45 papers), Heavy Metal Exposure and Toxicity (42 papers) and Heme Oxygenase-1 and Carbon Monoxide (37 papers). James S. Woods collaborates with scholars based in United States, Portugal and Canada. James S. Woods's co-authors include Bruce A. Fowler, Dennis M. Miller, Bert‐Ove Lund, Michael D. Martin, Diana Echeverria, Nicholas J. Heyer, Maureen E. Ellis, Alvah C. Bittner, Lincoln Polissar and Federico M. Farin and has published in prestigious journals such as Science, Physical Review Letters and JAMA.

In The Last Decade

James S. Woods

141 papers receiving 4.4k citations

Peers

James S. Woods
Louis W. Chang United States
Sam Kacew Canada
John Osterloh United States
Eric J. Sampson United States
David C. Dorman United States
Louis W. Chang United States
James S. Woods
Citations per year, relative to James S. Woods James S. Woods (= 1×) peers Louis W. Chang

Countries citing papers authored by James S. Woods

Since Specialization
Citations

This map shows the geographic impact of James S. Woods's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James S. Woods with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James S. Woods more than expected).

Fields of papers citing papers by James S. Woods

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James S. Woods. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James S. Woods. The network helps show where James S. Woods may publish in the future.

Co-authorship network of co-authors of James S. Woods

This figure shows the co-authorship network connecting the top 25 collaborators of James S. Woods. A scholar is included among the top collaborators of James S. Woods based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James S. Woods. James S. Woods is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Martins, Isabel Pavão, Alexandre Castro‐Caldas, Mário Bernardo, et al.. (2008). Neurological outcomes in children with and without amalgam-related mercury exposure. The Journal of the American Dental Association. 139(2). 138–145. 29 indexed citations
3.
Heyer, Nicholas J., Diana Echeverria, Federico M. Farin, & James S. Woods. (2008). The Association Between Serotonin Transporter Gene Promoter Polymorphism (5-HTTLPR), Self-Reported Symptoms, and Dental Mercury Exposure. Journal of Toxicology and Environmental Health. 71(19). 1318–1326. 16 indexed citations
4.
Woods, James S., Michael D. Martin, Brian G. Leroux, et al.. (2007). The Contribution of Dental Amalgam to Urinary Mercury Excretion in Children. Environmental Health Perspectives. 115(10). 1527–1531. 48 indexed citations
5.
Ellis, Maureen E., et al.. (2007). RhoA regulation of NF-κB activation is mediated by COX-2-dependent feedback inhibition of IKK in kidney epithelial cells. American Journal of Physiology-Cell Physiology. 293(3). C1160–C1170. 10 indexed citations
6.
Martin, Michael D., Tonya Benton, Mário Bernardo, et al.. (2007). The association of dental caries with blood lead in children when adjusted for IQ and neurobehavioral performance. The Science of The Total Environment. 377(2-3). 159–164. 9 indexed citations
7.
Woods, James S., et al.. (2005). The association between genetic polymorphisms of coproporphyrinogen oxidase and an atypical porphyrinogenic response to mercury exposure in humans. Toxicology and Applied Pharmacology. 206(2). 113–120. 55 indexed citations
8.
Ozuah, Philip O., et al.. (2003). Mercury Exposure in an Urban Pediatric Population. Ambulatory Pediatrics. 3(1). 24–26. 27 indexed citations
9.
Farin, Federico M., James S. Woods, Patricia A. Janssen, et al.. (2001). Genetic polymorphisms of superoxide dismutase in Parkinson's disease. Movement Disorders. 16(4). 705–707. 20 indexed citations
11.
Thompson, Sally A., Collin C. White, Cecile M. Krejsa, et al.. (1999). Induction of glutamate-cysteine ligase (γ-glutamylcysteine synthetase) in the brains of adult female mice subchronically exposed to methylmercury. Toxicology Letters. 110(1-2). 1–9. 57 indexed citations
12.
Ponce, Rafael, Scott M. Bartell, Terrance J. Kavanagh, et al.. (1998). Uncertainty Analysis Methods for Comparing Predictive Models and Biomarkers: A Case Study of Dietary Methyl Mercury Exposure. Regulatory Toxicology and Pharmacology. 28(2). 96–105. 20 indexed citations
13.
Maiorino, Richard M., D González-Ramírez, M A Zuñiga-Charles, et al.. (1996). Sodium 2,3-dimercaptopropane-1-sulfonate challenge test for mercury in humans. III. Urinary mercury after exposure to mercurous chloride.. Journal of Pharmacology and Experimental Therapeutics. 277(2). 938–944. 11 indexed citations
14.
Echeverria, Diana, Nicholas J. Heyer, Michael D. Martin, et al.. (1995). Behavioral effects of low-level exposure to Hg∘ among dentists. Neurotoxicology and Teratology. 17(2). 161–168. 117 indexed citations
15.
Woods, James S.. (1993). Quantitative Measurement of Porphyrins in Biological Tissues and Evaluation of Tissue Porphyrins during Toxicant Exposures. Fundamental and Applied Toxicology. 21(3). 291–297. 19 indexed citations
16.
Hooper, Michael, et al.. (1993). Porphyrin profiles in the nestling European starling(sturnus vulgaris):A potential biomarker of field contaminant exposure. Journal of Toxicology and Environmental Health. 40(1). 47–59. 23 indexed citations
17.
Woods, James S., et al.. (1992). Enhancement of γ-glutamylcysteine synthetase mRNA in rat kidney by methyl mercury. Archives of Biochemistry and Biophysics. 296(1). 350–353. 56 indexed citations
18.
Miller, Dennis M., Bert‐Ove Lund, & James S. Woods. (1991). Reactivity of Hg(II) with superoxide: Evidence for the catalytic dismutation of superoxide by HG(II). Journal of Biochemical Toxicology. 6(4). 293–298. 60 indexed citations
19.
Woods, James S., et al.. (1987). Soft Tissue Sarcoma and Non-Hodgkin's Lymphoma in Relation to Phenoxyherbicide and Chlorinated Phenol Exposure in Western Washington. JNCI Journal of the National Cancer Institute. 78(5). 899–910. 186 indexed citations
20.
Woods, James S. & Bruce A. Fowler. (1977). Effects of chronic arsenic exposure on hematopoietic function in adult mammalian liver. Environmental Health Perspectives. 19. 209–213. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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