James R. Paulson
- Molecular Biology top 5%
- Plant Science top 5%
- Cell Biology top 2%
- Genetics top 10%
- Ecology top 10%
- Co-authors
- Ulrich K. LaemmliWilliam C. EarnshawLinfeng XieKenneth W. AdolphKumiko SamejimaMasato T. KanemakiN. M. NaumovaAnton Goloborodko
- Topics
- Microtubule and mitosis dynamics (10 papers)Genomics and Chromatin Dynamics (10 papers)DNA and Nucleic Acid Chemistry (8 papers)
- Partner nations
- United StatesUnited KingdomOman
In The Last Decade
James R. Paulson
38 papers receiving 2.5k citations
Hit Papers
Peers
Comparison fields: 5 of 101
- Molecular Biology 2.4k
- Plant Science 596
- Cell Biology 524
- Genetics 300
- Ecology 193
Countries citing papers authored by James R. Paulson
This map shows the geographic impact of James R. Paulson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James R. Paulson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James R. Paulson more than expected).
Fields of papers citing papers by James R. Paulson
This network shows the impact of papers produced by James R. Paulson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James R. Paulson. The network helps show where James R. Paulson may publish in the future.
Co-authorship network of co-authors of James R. Paulson
This figure shows the co-authorship network connecting the top 25 collaborators of James R. Paulson. A scholar is included among the top collaborators of James R. Paulson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James R. Paulson. James R. Paulson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 0 | |
| 3 | 1 | |
| 4 | 53 | |
| 5 | 203 | |
| 6 | A pathway for mitotic chromosome formationbreakdown → | 486 |
| 7 | 44 | |
| 8 | A pathway for mitotic chromosome formation | 1 |
| 9 | 24 | |
| 10 | 9 | |
| 11 | 19 | |
| 12 | 13 | |
| 13 | 1 | |
| 14 | 3 | |
| 15 | 8 | |
| 16 | 4 | |
| 17 | 25 | |
| 18 | 37 | |
| 19 | 171 | |
| 20 | The structure of histone-depleted metaphase chromosomesbreakdown → | 837 |
About James R. Paulson
James R. Paulson is a scholar working on Cell Biology, Molecular Biology and Molecular Medicine, having authored 41 papers that have together received 2.6k indexed citations. Recurring topics across this work include Microtubule and mitosis dynamics (10 papers), Genomics and Chromatin Dynamics (10 papers) and DNA and Nucleic Acid Chemistry (8 papers). The work is most often cited by research in Molecular Biology (2.4k citations), Cell Biology (524 citations) and Structural Biology (29 citations). James R. Paulson has collaborated with scholars based in United States, United Kingdom and Oman. Frequent co-authors include Ulrich K. Laemmli, William C. Earnshaw, Linfeng Xie, Kenneth W. Adolph, Kumiko Samejima, Masato T. Kanemaki, N. M. Naumova, Anton Goloborodko, Leonid A. Mirny and Job Dekker. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.