James M. Mullin

5.1k total citations
111 papers, 4.0k citations indexed

About

James M. Mullin is a scholar working on Molecular Biology, Neurology and Oncology. According to data from OpenAlex, James M. Mullin has authored 111 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 56 papers in Molecular Biology, 54 papers in Neurology and 27 papers in Oncology. Recurrent topics in James M. Mullin's work include Barrier Structure and Function Studies (54 papers), Drug Transport and Resistance Mechanisms (21 papers) and Connexins and lens biology (14 papers). James M. Mullin is often cited by papers focused on Barrier Structure and Function Studies (54 papers), Drug Transport and Resistance Mechanisms (21 papers) and Connexins and lens biology (14 papers). James M. Mullin collaborates with scholars based in United States, United Kingdom and Canada. James M. Mullin's co-authors include Alejandro Peralta Soler, G. Feldman, Colleen Marano, Thomas G. O’Brien, Mary Carmen Valenzano, K. V. Laughlin, Arnošt Kleinzeller, Hilary Clarke, Katherine M. DiGuilio and Leila Diamond and has published in prestigious journals such as Science, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

James M. Mullin

108 papers receiving 3.9k citations

Peers

James M. Mullin
Hua Yang China
Rudolf Lucas United States
Beth Kelly United States
B F Trump United States
Dirk Brenner Germany
Sandra Barth Germany
James M. Mullin
Citations per year, relative to James M. Mullin James M. Mullin (= 1×) peers Tetsuaki Hirase

Countries citing papers authored by James M. Mullin

Since Specialization
Citations

This map shows the geographic impact of James M. Mullin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James M. Mullin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James M. Mullin more than expected).

Fields of papers citing papers by James M. Mullin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James M. Mullin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James M. Mullin. The network helps show where James M. Mullin may publish in the future.

Co-authorship network of co-authors of James M. Mullin

This figure shows the co-authorship network connecting the top 25 collaborators of James M. Mullin. A scholar is included among the top collaborators of James M. Mullin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James M. Mullin. James M. Mullin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Howes, Simon R., et al.. (2025). Anesthesia and EMI: Ensuring Safe CIED Management During Surgical Procedures. Journal of Cardiothoracic and Vascular Anesthesia. 39(6). 1534–1537.
2.
DiGuilio, Katherine M., et al.. (2023). Quercetin improves and protects Calu-3 airway epithelial barrier function. Frontiers in Cell and Developmental Biology. 11. 1271201–1271201. 3 indexed citations
3.
Mullin, James M., et al.. (2018). Accuracy of phase-contrast velocimetry in systems with skewed intravoxel velocity distributions. Journal of Magnetic Resonance. 296. 121–129. 5 indexed citations
4.
DiGuilio, Katherine M., et al.. (2018). Cobalt chloride compromises transepithelial barrier properties of CaCo-2 BBe human gastrointestinal epithelial cell layers. BMC Gastroenterology. 18(1). 2–2. 7 indexed citations
5.
Pawlikowski, Jeff S., Claire Brock, Sheau‐Chiann Chen, et al.. (2015). Acute Inhibition of MEK Suppresses Congenital Melanocytic Nevus Syndrome in a Murine Model Driven by Activated NRAS and Wnt Signaling. Journal of Investigative Dermatology. 135(11). 2902–2902. 4 indexed citations
6.
Thomas, Sunil, et al.. (2015). Novel Colitis Immunotherapy Targets Bin1 and Improves Colon Cell Barrier Function. Digestive Diseases and Sciences. 61(2). 423–432. 11 indexed citations
7.
Chang, Mee Young, Janette Boulden, Mary Carmen Valenzano, et al.. (2012). Bin1 Attenuation Suppresses Experimental Colitis by Enforcing Intestinal Barrier Function. Digestive Diseases and Sciences. 57(7). 1813–1821. 9 indexed citations
8.
Ramalingam, Arivudainambi, et al.. (2010). Dietary methionine restriction improves colon tight junction barrier function and alters claudin expression pattern. American Journal of Physiology-Cell Physiology. 299(5). C1028–C1035. 39 indexed citations
9.
Rajasekaran, Sigrid A., Daniel Wolle, Cromwell E. Espineda, et al.. (2010). Na,K-ATPase Subunits as Markers for Epithelial-Mesenchymal Transition in Cancer and Fibrosis. Molecular Cancer Therapeutics. 9(6). 1515–1524. 67 indexed citations
10.
Mullin, James M., et al.. (2009). Modification of Tight Junction Structure and Permeability by Nutritional Means. Annals of the New York Academy of Sciences. 1165(1). 99–112. 17 indexed citations
11.
Valenzano, Mary Carmen, et al.. (2009). Omeprazole Induces Gastric Permeability to Digoxin. Digestive Diseases and Sciences. 55(5). 1255–1263. 12 indexed citations
12.
Mullin, James M., Mary Carmen Valenzano, Deborah Lurie, et al.. (2008). Esomeprazole induces upper gastrointestinal tract transmucosal permeability increase. Alimentary Pharmacology & Therapeutics. 28(11-12). 1317–1325. 78 indexed citations
13.
Bain, Maureen, et al.. (2007). Noninvasive monitoring of chick development in ovo using a 7T MRI system from day 12 of incubation through to hatching. Journal of Magnetic Resonance Imaging. 26(1). 198–201. 63 indexed citations
14.
Mullin, James M., Nicole M Agostino, Erika P. Rendón‐Huerta, & Joseph Thornton. (2005). Keynote review: Epithelial and endothelial barriers in human disease. Drug Discovery Today. 10(6). 395–408. 97 indexed citations
15.
Mullin, James M., James M. Leatherman, Mary Carmen Valenzano, et al.. (2005). RasMutation Impairs Epithelial Barrier Function to a Wide Range of Nonelectrolytes. Molecular Biology of the Cell. 16(12). 5538–5550. 35 indexed citations
16.
Mullin, James M., et al.. (2002). Age- and Diet-Related Increase in Transepithelial Colon Permeability of Fischer 344 Rats. Digestive Diseases and Sciences. 47(10). 2262–2270. 40 indexed citations
17.
Clarke, Hilary, Nicole Ginanni, Alejandro Peralta Soler, & James M. Mullin. (2000). Regulation of protein kinase C-δ and -ɛ isoforms by phorbol ester treatment of LLC-PK1 renal epithelia. Kidney International. 58(3). 1004–1015. 16 indexed citations
18.
Rosson, Dan, Thomas G. O’Brien, Zoltán Szállási, et al.. (1997). Protein Kinase C-α Activity Modulates Transepithelial Permeability and Cell Junctions in the LLC-PK1 Epithelial Cell Line. Journal of Biological Chemistry. 272(23). 14950–14953. 83 indexed citations
19.
Soler, Alejandro Peralta, et al.. (1995). Cell Division Does Not Increase Transepithelial Permeability of LLC-PK1 Cell Sheets. Experimental Cell Research. 220(2). 446–455. 20 indexed citations
20.
Mullin, James M. & Thomas G. O’Brien. (1987). Spontaneous reversal of polarity of the voltage across LLC‐PK1 renal epithelial cell sheets. Journal of Cellular Physiology. 133(3). 515–522. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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