Nicole Ginanni

473 total citations
9 papers, 396 citations indexed

About

Nicole Ginanni is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Nicole Ginanni has authored 9 papers receiving a total of 396 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Cancer Research and 2 papers in Oncology. Recurrent topics in Nicole Ginanni's work include Peroxisome Proliferator-Activated Receptors (3 papers), Cancer, Lipids, and Metabolism (3 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Nicole Ginanni is often cited by papers focused on Peroxisome Proliferator-Activated Receptors (3 papers), Cancer, Lipids, and Metabolism (3 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Nicole Ginanni collaborates with scholars based in United States. Nicole Ginanni's co-authors include Gregory J. Opiteck, Ashok Dongre, Stanley A. Hefta, James M. Mullin, K. V. Laughlin, Hilary Clarke, Alejandro Peralta Soler, Colleen Marano, Nancy E. Kohl and Michael R. Tota and has published in prestigious journals such as Cancer Research, Clinical Cancer Research and Annals of the New York Academy of Sciences.

In The Last Decade

Nicole Ginanni

9 papers receiving 392 citations

Peers

Nicole Ginanni

MB

SPECT

CR

NEURO

ONCOL

Maria C. Messner United States

Shaun R. Selness United States

Lingjue Wang United States

David C. Montgomery United States

Adrián Benito Spain

Wataru Obuchi Japan

Wolf Oehrl Germany

Agus Darwanto United States

Elmar Schmid Germany

Renee C. Geck United States

Maria C. Messner United States

Nicole Ginanni

260 ×1.0

MB

60 ×0.5

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19 ×0.2

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12 ×0.2

NEURO

24 ×0.5

ONCOL

Citations per year, relative to Nicole Ginanni Nicole Ginanni (= 1×) peers Maria C. Messner

Countries citing papers authored by Nicole Ginanni

Since Specialization

Citations

This map shows the geographic impact of Nicole Ginanni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicole Ginanni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicole Ginanni more than expected).

Fields of papers citing papers by Nicole Ginanni

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nicole Ginanni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicole Ginanni. The network helps show where Nicole Ginanni may publish in the future.

Co-authorship network of co-authors of Nicole Ginanni

This figure shows the co-authorship network connecting the top 25 collaborators of Nicole Ginanni. A scholar is included among the top collaborators of Nicole Ginanni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nicole Ginanni. Nicole Ginanni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Zhan, Yanai, Nicole Ginanni, Michael R. Tota, et al.. (2008). Control of Cell Growth and Survival by Enzymes of the Fatty Acid Synthesis Pathway in HCT-116 Colon Cancer Cells. Clinical Cancer Research. 14(18). 5735–5742. 53 indexed citations

2.

Ginanni, Nicole, Yanai Zhan, Michael R. Tota, et al.. (2007). Kinetic analysis of the fatty acid synthesis pathway in HCT-116 colon cancer cells: role of FAS and ACC1 in maintaining tumor cell viability and proliferation. Cancer Research. 67. 4473–4473. 2 indexed citations

3.

Rivkin, Alexey, Mark T. Goulet, Nathan Bays, et al.. (2006). 3-Aryl-4-hydroxyquinolin-2(1H)-one derivatives as type I fatty acid synthase inhibitors. Bioorganic & Medicinal Chemistry Letters. 16(17). 4620–4623. 42 indexed citations

4.

Ginanni, Nicole, et al.. (2002). Biomarker Discovery in Urine by Proteomics. Journal of Proteome Research. 1(2). 161–169. 171 indexed citations

5.

Marano, Colleen, et al.. (2001). Phorbol Ester Treatment Increases Paracellular Permeability Across IEC-18 Gastrointestinal Epithelium In Vitro. Digestive Diseases and Sciences. 46(7). 1490–1499. 17 indexed citations

6.

Clarke, Hilary, Nicole Ginanni, Alejandro Peralta Soler, & James M. Mullin. (2000). Regulation of protein kinase C-δ and -ɛ isoforms by phorbol ester treatment of LLC-PK1 renal epithelia. Kidney International. 58(3). 1004–1015. 16 indexed citations

7.

Mullin, James M., K. V. Laughlin, Nicole Ginanni, et al.. (2000). Increased Tight Junction Permeability Can Result from Protein Kinase C Activation/Translocation and Act as a Tumor Promotional Event in Epithelial Cancers. Annals of the New York Academy of Sciences. 915(1). 231–236. 60 indexed citations

8.

Clarke, Hilary, Nicole Ginanni, K. V. Laughlin, et al.. (2000). The Transient Increase of Tight Junction Permeability Induced by Bryostatin 1 Correlates with Rapid Downregulation of Protein Kinase C-α. Experimental Cell Research. 261(1). 239–249. 21 indexed citations

9.

Mullin, James M., Nicole Ginanni, & K. V. Laughlin. (1998). Protein kinase C activation increases transepithelial transport of biologically active insulin.. PubMed. 58(8). 1641–5. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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