James G. Ryall

5.7k total citations · 1 hit paper
58 papers, 3.8k citations indexed

About

James G. Ryall is a scholar working on Molecular Biology, Physiology and Rehabilitation. According to data from OpenAlex, James G. Ryall has authored 58 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 42 papers in Molecular Biology, 28 papers in Physiology and 19 papers in Rehabilitation. Recurrent topics in James G. Ryall's work include Muscle Physiology and Disorders (30 papers), Adipose Tissue and Metabolism (24 papers) and Exercise and Physiological Responses (19 papers). James G. Ryall is often cited by papers focused on Muscle Physiology and Disorders (30 papers), Adipose Tissue and Metabolism (24 papers) and Exercise and Physiological Responses (19 papers). James G. Ryall collaborates with scholars based in Australia, United States and Canada. James G. Ryall's co-authors include Gordon S. Lynch, Jonathan D. Schertzer, Vittorio Sartorelli, Martin N. Sillence, Stephen Dalton, David R. Plant, Xuesong Feng, Aster H. Juan, Assia Derfoul and Hossein Zare and has published in prestigious journals such as Physiological Reviews, Genes & Development and SHILAP Revista de lepidopterología.

In The Last Decade

James G. Ryall

58 papers receiving 3.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The NAD+-Dependent SIRT1 Deacetylase Translates a Metabolic Switch into Regulatory Epigenetics in Skeletal Muscle Stem Cells

2015 422 citations James G. Ryall, Xuesong Feng et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
James G. Ryall Australia	34	2.6k	1.5k	794	438	358	58	3.8k
Angus G. Scrimgeour United States	23	2.2k 0.9×	990 0.6×	954 1.2×	495 1.1×	89 0.2×	38	3.5k
William O. Kline United States	11	3.8k 1.5×	1.7k 1.1×	1.3k 1.7×	772 1.8×	109 0.3×	11	4.8k
Michael Gonzalez United States	6	3.4k 1.3×	1.5k 1.0×	1.2k 1.6×	706 1.6×	71 0.2×	6	4.2k
Erqian Na United States	15	3.1k 1.2×	1.3k 0.9×	935 1.2×	576 1.3×	49 0.1×	20	4.1k
Elisa Calabria Italy	20	3.3k 1.3×	1.5k 1.0×	819 1.0×	615 1.4×	48 0.1×	32	4.2k
Thorkil Ploug Denmark	46	2.6k 1.0×	3.2k 2.1×	1.8k 2.3×	392 0.9×	104 0.3×	112	5.7k
Christopher Cardozo United States	39	3.0k 1.1×	1.0k 0.7×	685 0.9×	396 0.9×	37 0.1×	153	5.0k
Marcelo D. Gomes Brazil	17	2.9k 1.1×	1.2k 0.8×	870 1.1×	511 1.2×	39 0.1×	39	3.6k
Anne Picard Italy	15	2.9k 1.1×	1.1k 0.7×	630 0.8×	447 1.0×	31 0.1×	32	3.6k
Keith Baar United States	27	1.7k 0.7×	1.5k 1.0×	1.4k 1.8×	666 1.5×	35 0.1×	60	3.3k

Countries citing papers authored by James G. Ryall

Since Specialization

Citations

This map shows the geographic impact of James G. Ryall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James G. Ryall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James G. Ryall more than expected).

Fields of papers citing papers by James G. Ryall

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James G. Ryall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James G. Ryall. The network helps show where James G. Ryall may publish in the future.

Co-authorship network of co-authors of James G. Ryall

This figure shows the co-authorship network connecting the top 25 collaborators of James G. Ryall. A scholar is included among the top collaborators of James G. Ryall based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James G. Ryall. James G. Ryall is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Swiderski, Kristy, et al.. (2020). HSP70 drives myoblast fusion during C2C12 myogenic differentiation. Biology Open. 9(7). 6 indexed citations

Johnstone, Cameron N., et al.. (2018). Altered energy metabolism and metabolic gene expression associated with increased metastatic capacity identified in MDA-MB-231 cell line variants. Journal of Cancer Metastasis and Treatment. 2018. 5 indexed citations

Ryall, James G. & Gordon S. Lynch. (2018). The molecular signature of muscle stem cells is driven by nutrient availability and innate cell metabolism. Current Opinion in Clinical Nutrition & Metabolic Care. 21(4). 240–245. 8 indexed citations

Park, Sung Jun, Faiyaz Ahmad, Alexandra L. Brown, et al.. (2017). Specific Sirt1 Activator-mediated Improvement in Glucose Homeostasis Requires Sirt1-Independent Activation of AMPK. EBioMedicine. 18. 128–138. 31 indexed citations

Ryall, James G., et al.. (2017). Measuring Mitochondrial Substrate Utilization in Skeletal Muscle Stem Cells. Methods in molecular biology. 1668. 61–73. 5 indexed citations

Davey, Jonathan R., Kevin I. Watt, Benjamin L. Parker, et al.. (2016). Integrated expression analysis of muscle hypertrophy identifies Asb2 as a negative regulator of muscle mass. JCI Insight. 1(5). 43 indexed citations

Mølck, Christina, James G. Ryall, Jean‐Marc Pascussi, et al.. (2016). The A 2b adenosine receptor antagonist PSB-603 promotes oxidative phosphorylation and ROS production in colorectal cancer cells via adenosine receptor-independent mechanism. Cancer Letters. 383(1). 135–143. 26 indexed citations

Proserpio, Valentina, Raffaella Fittipaldi, James G. Ryall, Vittorio Sartorelli, & Giuseppina Caretti. (2013). The methyltransferase SMYD3 mediates the recruitment of transcriptional cofactors at the myostatin and c-Met genes and regulates skeletal muscle atrophy. Genes & Development. 27(11). 1299–1312. 65 indexed citations

Poel, Chris van der, Luc E. Gosselin, Jonathan D. Schertzer, et al.. (2011). Ageing prolongs inflammatory marker expression in regenerating rat skeletal muscles after injury. Journal of Inflammation. 8(1). 41–41. 27 indexed citations

10.

Murphy, Kate T., James G. Ryall, Lawrence Nair, et al.. (2010). Antibody-Directed Myostatin Inhibition Improves Diaphragm Pathology in Young but not Adult Dystrophic mdx Mice. American Journal Of Pathology. 176(5). 2425–2434. 46 indexed citations

11.

Ryall, James G., Jarrod E. Church, & Gordon S. Lynch. (2009). Novel role for β‐adrenergic signalling in skeletal muscle growth, development and regeneration. Clinical and Experimental Pharmacology and Physiology. 37(3). 397–401. 27 indexed citations

12.

Turpin-Nolan, Sarah M., James G. Ryall, Robert J. Southgate, et al.. (2009). Examination of ‘lipotoxicity’ in skeletal muscle of high‐fat fed and ob / ob mice. The Journal of Physiology. 587(7). 1593–1605. 92 indexed citations

13.

Ryall, James G., Jonathan D. Schertzer, Kate T. Murphy, Andrew M. Allen, & Gordon S. Lynch. (2008). Chronic β₂-adrenoceptor stimulation impairs cardiac relaxation via reduced SR Ca²⁺-ATPase protein and activity. American Journal of Physiology-Heart and Circulatory Physiology. 294(6). H2587–H2595. 22 indexed citations

14.

Ryall, James G. & Gordon S. Lynch. (2008). The potential and the pitfalls of β-adrenoceptor agonists for the management of skeletal muscle wasting. Pharmacology & Therapeutics. 120(3). 219–232. 77 indexed citations

15.

Gehrig, Stefan M., James G. Ryall, Jonathan D. Schertzer, & Gordon S. Lynch. (2008). Insulin‐like growth factor‐I analogue protects muscles of dystrophic mdx mice from contraction‐mediated damage. Experimental Physiology. 93(11). 1190–1198. 37 indexed citations

16.

Pearen, Michael A., James G. Ryall, Megan Maxwell, et al.. (2006). The Orphan Nuclear Receptor, NOR-1, Is a Target of β-Adrenergic Signaling in Skeletal Muscle. Endocrinology. 147(11). 5217–5227. 105 indexed citations

17.

Schertzer, Jonathan D., James G. Ryall, & Gordon S. Lynch. (2006). Systemic administration of IGF-I enhances oxidative status and reduces contraction-induced injury in skeletal muscles of mdx dystrophic mice. American Journal of Physiology-Endocrinology and Metabolism. 291(3). E499–E505. 58 indexed citations

18.

Lynch, Gordon S., Jonathan D. Schertzer, & James G. Ryall. (2006). Therapeutic approaches for muscle wasting disorders. Pharmacology & Therapeutics. 113(3). 461–487. 119 indexed citations

19.

Ryall, James G., Martin N. Sillence, & Gordon S. Lynch. (2006). Systemic administration of β₂‐adrenoceptor agonists, formoterol and salmeterol, elicit skeletal muscle hypertrophy in rats at micromolar doses. British Journal of Pharmacology. 147(6). 587–595. 93 indexed citations

20.

Gregorevic, Paul, James G. Ryall, David R. Plant, Martin N. Sillence, & Gordon S. Lynch. (2005). Chronic β-agonist administration affects cardiac function of adult but not old rats, independent of β-adrenoceptor density. American Journal of Physiology-Heart and Circulatory Physiology. 289(1). H344–H349. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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