James G. Moe

798 citations
25 papers · 456 indexed · h-index 11
Topics
Alzheimer's disease research and treatments (15 papers)Cholinesterase and Neurodegenerative Diseases (11 papers)Computational Drug Discovery Methods (6 papers)
Partner nations
United StatesRussia

In The Last Decade

James G. Moe

23 papers receiving 450 citations

Peers

James G. Moe
Comparison fields: 5 of 66
  • Molecular Biology 353
  • Physiology 117
  • Cellular and Molecular Neuroscience 37
  • Neurology 36
  • Genetics 34
Replace Eckhard Mandelkow with:
Eckhard Mandelkow Germany
Victoria A. Assimon United States
M. Schwalbe Germany
Natsuko Goda Japan
Anisa Scott United States
Maxim Igaev Germany
S. Fabio Falsone Austria
Ivan Haralampiev Germany
Karin Wieligmann Germany
Saravanakumar Narayanan Germany
James G. Moe relative to Eckhard Mandelkow Germany Eckhard Mandelkow's profile →
Citations per field
00.5×7.7×
Eckhard Mandelkow · 1×
Citations per year

Countries citing papers authored by James G. Moe

Since Specialization
Citations

This map shows the geographic impact of James G. Moe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James G. Moe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James G. Moe more than expected).

Fields of papers citing papers by James G. Moe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James G. Moe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James G. Moe. The network helps show where James G. Moe may publish in the future.

Co-authorship network of co-authors of James G. Moe

This figure shows the co-authorship network connecting the top 25 collaborators of James G. Moe. A scholar is included among the top collaborators of James G. Moe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James G. Moe. James G. Moe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 2
2 6
3 13
4 1
5 0
6 26
7 1
8 74
9 2
10 1
11 10
12 9
13 28
14 16
15 16
16 53
17 110
18 57
19 16
20 2

About James G. Moe

James G. Moe is a scholar working on Pharmacology, Physiology and Neurology, having authored 25 papers that have together received 456 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (15 papers), Cholinesterase and Neurodegenerative Diseases (11 papers) and Computational Drug Discovery Methods (6 papers). The work is most often cited by research in Molecular Biology (353 citations), Physiology (117 citations) and Neurology (36 citations). James G. Moe has collaborated with scholars based in United States and Russia. Frequent co-authors include Irina M. Russu, Eliot J. Davidowitz, G. Ramachandra Reddy, Michael P. Stone, Michael R. Sierks, Huilai Tian, Patrícia Luciana da Costa Lopez, Sharareh Emadi, Lawrence J. Marnett and Lawrence J. Marnett. Their work appears in journals such as Nucleic Acids Research, PLoS ONE and Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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