James G. Moe

798 total citations
25 papers, 456 citations indexed

About

James G. Moe is a scholar working on Physiology, Pharmacology and Molecular Biology. According to data from OpenAlex, James G. Moe has authored 25 papers receiving a total of 456 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 12 papers in Pharmacology and 8 papers in Molecular Biology. Recurrent topics in James G. Moe's work include Alzheimer's disease research and treatments (15 papers), Cholinesterase and Neurodegenerative Diseases (11 papers) and Computational Drug Discovery Methods (6 papers). James G. Moe is often cited by papers focused on Alzheimer's disease research and treatments (15 papers), Cholinesterase and Neurodegenerative Diseases (11 papers) and Computational Drug Discovery Methods (6 papers). James G. Moe collaborates with scholars based in United States and Russia. James G. Moe's co-authors include Irina M. Russu, Eliot J. Davidowitz, G. Ramachandra Reddy, Michael P. Stone, Michael R. Sierks, Huilai Tian, Patrícia Luciana da Costa Lopez, Sharareh Emadi, Lawrence J. Marnett and Lawrence J. Marnett and has published in prestigious journals such as Nucleic Acids Research, PLoS ONE and Biochemistry.

In The Last Decade

James G. Moe

23 papers receiving 450 citations

Peers

James G. Moe
James G. Moe
Citations per year, relative to James G. Moe James G. Moe (= 1×) peers S. Fabio Falsone

Countries citing papers authored by James G. Moe

Since Specialization
Citations

This map shows the geographic impact of James G. Moe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James G. Moe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James G. Moe more than expected).

Fields of papers citing papers by James G. Moe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James G. Moe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James G. Moe. The network helps show where James G. Moe may publish in the future.

Co-authorship network of co-authors of James G. Moe

This figure shows the co-authorship network connecting the top 25 collaborators of James G. Moe. A scholar is included among the top collaborators of James G. Moe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James G. Moe. James G. Moe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Davidowitz, Eliot J., Pedro F. Lopez, Dilip R. Patel, et al.. (2025). Therapeutic Treatment With OLX‐07010 Inhibited Tau Aggregation and Ameliorated Motor Deficits in an Aged Mouse Model of Tauopathy. Journal of Neurochemistry. 169(3). e70025–e70025. 2 indexed citations
2.
Davidowitz, Eliot J., Patrícia Luciana da Costa Lopez, Heidy Jimenez, et al.. (2023). Small molecule inhibitor of tau self-association in a mouse model of tauopathy: A preventive study in P301L tau JNPL3 mice. PLoS ONE. 18(8). e0286523–e0286523. 6 indexed citations
3.
Davidowitz, Eliot J., Pavan Krishnamurthy, Patricia López, et al.. (2019). In Vivo Validation of a Small Molecule Inhibitor of Tau Self-Association in htau Mice. Journal of Alzheimer s Disease. 73(1). 147–161. 13 indexed citations
4.
Moe, James G., Patricia López, Heidy Jimenez, et al.. (2018). P4‐222: IN VIVO EFFICACY OF A SMALL MOLECULE INHIBITOR OF TAU OLIGOMER FORMATION IN HTAU MICE. Alzheimer s & Dementia. 14(7S_Part_29). 1 indexed citations
5.
Moe, James G., et al.. (2015). P3‐329: Identification of tau oligomer proteolytic activity as a novel target for drug discovery. Alzheimer s & Dementia. 11(7S_Part_16).
6.
Tian, Huilai, Eliot J. Davidowitz, Patricia López, et al.. (2014). Isolation and characterization of antibody fragments selective for toxic oligomeric tau. Neurobiology of Aging. 36(3). 1342–1355. 26 indexed citations
7.
Moe, James G., et al.. (2014). P4‐212: DRUG DEVELOPMENT OF INHIBITORS OF TAU OLIGOMER FORMATION FOR ALZHEIMER'S DISEASE AND TAUOPATHIES. Alzheimer s & Dementia. 10(4S_Part_23). 1 indexed citations
8.
Tian, Huilai, Eliot J. Davidowitz, Patrícia Luciana da Costa Lopez, et al.. (2013). Trimeric Tau Is Toxic to Human Neuronal Cells at Low Nanomolar Concentrations. International Journal of Cell Biology. 2013. 1–9. 74 indexed citations
9.
Moe, James G., et al.. (2013). P4–423: Small‐molecule lead identification for inhibition of tau oligomer formation. Alzheimer s & Dementia. 9(4S_Part_22). 2 indexed citations
10.
Davidowitz, Eliot J. & James G. Moe. (2011). P3‐161: Tau autoproteolytic activity is induced by tau oligomerization. Alzheimer s & Dementia. 7(4S_Part_16). 1 indexed citations
11.
Савватеева-Попова, Е. В., А. В. Попов, Abraham Grossman, et al.. (2007). Pathogenic chaperone-like RNA induces congophilic aggregates and facilitates neurodegeneration in Drosophila. Cell Stress and Chaperones. 12(1). 9–9. 10 indexed citations
12.
Nietupski, Raymond, Dale A. Pelletier, Mark J. Fiandaca, et al.. (1996). Detection of rRNA from four respiratory pathogens using an automated Qβ replicase assay. Molecular and Cellular Probes. 10(5). 359–370. 9 indexed citations
14.
Moe, James G., Ewa Folta‐Stogniew, & Irina M. Russu. (1995). Energetics of base pair opening in a DNA dodecamer containing an A3T3tract. Nucleic Acids Research. 23(11). 1984–1989. 16 indexed citations
15.
Moe, James G., G. Ramachandra Reddy, Lawrence J. Marnett, & Michael P. Stone. (1994). 1H NMR characterization of a duplex oligodeoxynucleotide containing propanodeoxyguanosine opposite a two-base deletion in the (CpG)3 frameshift hotspot of Salmonella typhimurium hisD3052. Chemical Research in Toxicology. 7(3). 319–328. 16 indexed citations
17.
Moe, James G. & Irina M. Russu. (1992). Kinetics and energetics of base-pair opening in 5'-d(CGCGAATTCGCG)-3' and a substituted dodecamer containing G.cntdot.T mismatches. Biochemistry. 31(36). 8421–8428. 110 indexed citations
18.
Moe, James G. & Irina M. Russu. (1990). Proton exchange and base-pair opening kinetics in 5′-d(CGCGAATTCGCG)-3′ and related dodecamers. Nucleic Acids Research. 18(4). 821–827. 57 indexed citations
19.
Allen, Todd M., et al.. (1987). Effects on the murine mononuclear phagocyte system of chronic administration of liposomes containing cytotoxic drug or lipid A compared with empty liposomes. Canadian Journal of Physiology and Pharmacology. 65(2). 185–190. 16 indexed citations
20.
Chernick, S. S., James G. Moe, & Klaus Schwarz. (1955). Dietary Necrotic Liver Degeneration and Coenzyme A. Experimental Biology and Medicine. 89(4). 520–523. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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