Eliot J. Davidowitz

1.1k total citations
27 papers, 689 citations indexed

About

Eliot J. Davidowitz is a scholar working on Physiology, Pharmacology and Molecular Biology. According to data from OpenAlex, Eliot J. Davidowitz has authored 27 papers receiving a total of 689 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 11 papers in Pharmacology and 10 papers in Molecular Biology. Recurrent topics in Eliot J. Davidowitz's work include Alzheimer's disease research and treatments (15 papers), Cholinesterase and Neurodegenerative Diseases (11 papers) and Cancer, Hypoxia, and Metabolism (6 papers). Eliot J. Davidowitz is often cited by papers focused on Alzheimer's disease research and treatments (15 papers), Cholinesterase and Neurodegenerative Diseases (11 papers) and Cancer, Hypoxia, and Metabolism (6 papers). Eliot J. Davidowitz collaborates with scholars based in United States, Russia and Hungary. Eliot J. Davidowitz's co-authors include Robert D. Burk, Alan R. Schoenfeld, James G. Moe, Michael R. Sierks, Huilai Tian, Patrícia Luciana da Costa Lopez, Prasad Devarajan, Sharareh Emadi, Andrew Eisenberger and Tchaiko Parris and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Eliot J. Davidowitz

25 papers receiving 680 citations

Peers

Eliot J. Davidowitz
Xinchun Pi United States
Anna Smith United States
Xin Mei China
Mark V. Stevens United States
Xinchun Pi United States
Eliot J. Davidowitz
Citations per year, relative to Eliot J. Davidowitz Eliot J. Davidowitz (= 1×) peers Xinchun Pi

Countries citing papers authored by Eliot J. Davidowitz

Since Specialization
Citations

This map shows the geographic impact of Eliot J. Davidowitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eliot J. Davidowitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eliot J. Davidowitz more than expected).

Fields of papers citing papers by Eliot J. Davidowitz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eliot J. Davidowitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eliot J. Davidowitz. The network helps show where Eliot J. Davidowitz may publish in the future.

Co-authorship network of co-authors of Eliot J. Davidowitz

This figure shows the co-authorship network connecting the top 25 collaborators of Eliot J. Davidowitz. A scholar is included among the top collaborators of Eliot J. Davidowitz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eliot J. Davidowitz. Eliot J. Davidowitz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Davidowitz, Eliot J., Pedro F. Lopez, Dilip R. Patel, et al.. (2025). Therapeutic Treatment With OLX‐07010 Inhibited Tau Aggregation and Ameliorated Motor Deficits in an Aged Mouse Model of Tauopathy. Journal of Neurochemistry. 169(3). e70025–e70025. 2 indexed citations
2.
Davidowitz, Eliot J., Patrícia Luciana da Costa Lopez, Heidy Jimenez, et al.. (2023). Small molecule inhibitor of tau self-association in a mouse model of tauopathy: A preventive study in P301L tau JNPL3 mice. PLoS ONE. 18(8). e0286523–e0286523. 6 indexed citations
3.
Davidowitz, Eliot J., Pavan Krishnamurthy, Patricia López, et al.. (2019). In Vivo Validation of a Small Molecule Inhibitor of Tau Self-Association in htau Mice. Journal of Alzheimer s Disease. 73(1). 147–161. 13 indexed citations
4.
Moe, James G., et al.. (2015). P3‐329: Identification of tau oligomer proteolytic activity as a novel target for drug discovery. Alzheimer s & Dementia. 11(7S_Part_16).
5.
Tian, Huilai, Eliot J. Davidowitz, Patricia López, et al.. (2014). Isolation and characterization of antibody fragments selective for toxic oligomeric tau. Neurobiology of Aging. 36(3). 1342–1355. 26 indexed citations
6.
Moe, James G., et al.. (2014). P4‐212: DRUG DEVELOPMENT OF INHIBITORS OF TAU OLIGOMER FORMATION FOR ALZHEIMER'S DISEASE AND TAUOPATHIES. Alzheimer s & Dementia. 10(4S_Part_23). 1 indexed citations
7.
Tian, Huilai, Eliot J. Davidowitz, Patrícia Luciana da Costa Lopez, et al.. (2013). Trimeric Tau Is Toxic to Human Neuronal Cells at Low Nanomolar Concentrations. International Journal of Cell Biology. 2013. 1–9. 74 indexed citations
8.
Moe, James G., et al.. (2013). P4–423: Small‐molecule lead identification for inhibition of tau oligomer formation. Alzheimer s & Dementia. 9(4S_Part_22). 2 indexed citations
9.
Davidowitz, Eliot J. & James G. Moe. (2011). P3‐161: Tau autoproteolytic activity is induced by tau oligomerization. Alzheimer s & Dementia. 7(4S_Part_16). 1 indexed citations
10.
Adler, Victor, et al.. (2007). α2-Macroglobulin is a potential facilitator of prion protein transformation. Amyloid. 14(1). 1–10. 8 indexed citations
11.
Савватеева-Попова, Е. В., А. В. Попов, Abraham Grossman, et al.. (2007). Pathogenic chaperone-like RNA induces congophilic aggregates and facilitates neurodegeneration in Drosophila. Cell Stress and Chaperones. 12(1). 9–9. 10 indexed citations
12.
Davidowitz, Eliot J., et al.. (2005). Concentration of prion protein from biological samples to increase the limits of detection by immunoassay. Biotechnology and Applied Biochemistry. 41(3). 247–253. 6 indexed citations
13.
Devarajan, Prasad, et al.. (2001). The von Hippel-Lindau Gene Product Inhibits Renal Cell Apoptosis via Bcl-2-dependent Pathways. Journal of Biological Chemistry. 276(44). 40599–40605. 43 indexed citations
14.
Davidowitz, Eliot J., Alan R. Schoenfeld, & Robert D. Burk. (2001). VHL Induces Renal Cell Differentiation and Growth Arrest through Integration of Cell-Cell and Cell-Extracellular Matrix Signaling. Molecular and Cellular Biology. 21(3). 865–874. 89 indexed citations
15.
Schoenfeld, Alan R., Eliot J. Davidowitz, & Robert D. Burk. (2001). Endoplasmic reticulum/cytosolic localization of von Hippel-Lindau gene products is mediated by a 64-amino acid region. International Journal of Cancer. 91(4). 457–467. 31 indexed citations
16.
Schoenfeld, Alan R., Tchaiko Parris, Andrew Eisenberger, et al.. (2000). The von Hippel-Lindau tumor suppressor gene protects cells from UV-mediated apoptosis. Oncogene. 19(51). 5851–5857. 55 indexed citations
17.
Schoenfeld, Alan R., Eliot J. Davidowitz, & Robert D. Burk. (2000). Elongin BC complex prevents degradation of von Hippel-Lindau tumor suppressor gene products. Proceedings of the National Academy of Sciences. 97(15). 8507–8512. 104 indexed citations
18.
Davidowitz, Eliot J., Christopher A. Cullis, & Naomi Lang-Unnasch. (1994). Messenger RNA from diverse classes of alfalfa leghemoglobin genes show a similar pattern of spatial expression in symbiotic root nodules. Plant and Soil. 162(2). 303–307. 2 indexed citations
19.
Davidowitz, Eliot J., et al.. (1991). Sequence analysis of alfalfa (Medicago sativa) leghemoglobin cDNA and genomic clones. Plant Molecular Biology. 16(1). 161–165. 11 indexed citations
20.
Davidowitz, Eliot J., et al.. (1989). Nucleotide sequence of a cDNA clone encoding a leghemoglobin fromMedicago sativa. Nucleic Acids Research. 17(8). 3307–3307. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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