James D. Schmelzer

5.1k total citations
81 papers, 4.2k citations indexed

About

James D. Schmelzer is a scholar working on Physiology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, James D. Schmelzer has authored 81 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Physiology, 26 papers in Cellular and Molecular Neuroscience and 25 papers in Neurology. Recurrent topics in James D. Schmelzer's work include Pain Mechanisms and Treatments (35 papers), Botulinum Toxin and Related Neurological Disorders (15 papers) and Cardiac Ischemia and Reperfusion (13 papers). James D. Schmelzer is often cited by papers focused on Pain Mechanisms and Treatments (35 papers), Botulinum Toxin and Related Neurological Disorders (15 papers) and Cardiac Ischemia and Reperfusion (13 papers). James D. Schmelzer collaborates with scholars based in United States, Japan and Spain. James D. Schmelzer's co-authors include Phillip A. Low, Phillip A. Low, Ann M. Schmeichel, R R Tuck, Kim K. Nickander, Paula J. Zollman, Mikihiro Kihara, Kevin R. Ward, Yoshiyuki Mitsui and Hans Tritschler and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Medicine and Neuron.

In The Last Decade

James D. Schmelzer

79 papers receiving 4.1k citations

Peers

James D. Schmelzer
D. A. Greene United States
William G. Mayhan United States
S. A. Lattimer United States
James Palacino United States
Hwa Kyoung Shin South Korea
Takashi Taniguchi Japan
Masayasu Matsumoto Japan
Earl F. Ellis United States
Karen Horsburgh United Kingdom
Hiroyuki Kinouchi Japan
D. A. Greene United States
James D. Schmelzer
Citations per year, relative to James D. Schmelzer James D. Schmelzer (= 1×) peers D. A. Greene

Countries citing papers authored by James D. Schmelzer

Since Specialization
Citations

This map shows the geographic impact of James D. Schmelzer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James D. Schmelzer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James D. Schmelzer more than expected).

Fields of papers citing papers by James D. Schmelzer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James D. Schmelzer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James D. Schmelzer. The network helps show where James D. Schmelzer may publish in the future.

Co-authorship network of co-authors of James D. Schmelzer

This figure shows the co-authorship network connecting the top 25 collaborators of James D. Schmelzer. A scholar is included among the top collaborators of James D. Schmelzer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James D. Schmelzer. James D. Schmelzer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Singer, Wolfgang, Allan B. Dietz, Anita Zeller, et al.. (2017). Intrathecal Administration of Autologous Mesenchymal Stem Cells in Multiple System Atrophy – A Phase I/II Dose-Escalation Trial (S11.002). Neurology. 88(16_supplement). 1 indexed citations
2.
Moriya, Tamami, Chunfeng Zhao, S. Stephen, et al.. (2011). Tendon Injury Produces Changes in SSCT and Nerve Physiology Similar to Carpal Tunnel Syndrome in an in Vivo Rabbit Model. Hand. 6(4). 399–407. 17 indexed citations
3.
Lipp, Axel, James D. Schmelzer, Phillip A. Low, Bruce D. Johnson, & Eduardo E. Benarroch. (2010). Ventilatory and Cardiovascular Responses to Hypercapnia and Hypoxia in Multiple-System Atrophy. Archives of Neurology. 67(2). 211–6. 16 indexed citations
4.
Yoshii, Yuichi, Chunfeng Zhao, James D. Schmelzer, et al.. (2009). The Effects of Hypertonic Dextrose Injection on Connective Tissue and Nerve Conduction Through the Rabbit Carpal Tunnel. Archives of Physical Medicine and Rehabilitation. 90(2). 333–339. 47 indexed citations
5.
Schmeichel, Ann M., et al.. (2007). Orchestration of the inflammatory response in ischemia‐reperfusion injury. Journal of the Peripheral Nervous System. 12(2). 131–138. 10 indexed citations
6.
Wang, Yanping, Nobutoshi Kawamura, James D. Schmelzer, Ann M. Schmeichel, & Phillip A. Low. (2007). Decreased peripheral nerve damage after ischemia–reperfusion injury in mice lacking TNF-alpha. Journal of the Neurological Sciences. 267(1-2). 107–111. 27 indexed citations
7.
Wang, Yanping, et al.. (2006). Enhanced inflammatory response via activation of NF-κB in acute experimental diabetic neuropathy subjected to ischemia–reperfusion injury. Journal of the Neurological Sciences. 247(1). 47–52. 80 indexed citations
8.
Kawamura, Natsumi, Ann M. Schmeichel, Ying-Hong Wang, James D. Schmelzer, & Phillip A. Low. (2006). Multiple effects of hypothermia on inflammatory response following ischemia–reperfusion injury in experimental ischemic neuropathy. Experimental Neurology. 202(2). 487–496. 30 indexed citations
9.
Kawamura, Nobutoshi, James D. Schmelzer, Yanping Wang, Ann M. Schmeichel, & Phillip A. Low. (2005). The therapeutic window of hypothermic neuroprotection in experimental ischemic neuropathy: Protection in ischemic phase and potential deterioration in later reperfusion phase. Experimental Neurology. 195(2). 305–312. 15 indexed citations
10.
Wang, Yanping, et al.. (2004). Ischemia–reperfusion injury of peripheral nerve in experimental diabetic neuropathy. Journal of the Neurological Sciences. 227(1). 101–107. 18 indexed citations
11.
Saito, Fumiaki, Steven A. Moore, Rita Barresi, et al.. (2003). Unique Role of Dystroglycan in Peripheral Nerve Myelination, Nodal Structure, and Sodium Channel Stabilization. Neuron. 38(5). 747–758. 212 indexed citations
12.
Kihara, Mikihiro, et al.. (2001). A small dose of the immunosuppressive agent FK506 (tacrolimus) protects peripheral nerve from ischemic fiber degeneration. Muscle & Nerve. 24(12). 1601–1606. 13 indexed citations
13.
Mitsui, Yoshiyuki, et al.. (1999). Alpha-lipoic acid provides neuroprotection from ischemia-reperfusion injury of peripheral nerve. Journal of the Neurological Sciences. 163(1). 11–16. 80 indexed citations
14.
Kihara, Mikihiro, James D. Schmelzer, & Phillip A. Low. (1995). Effect of cilostazol on experimental diabetic neuropathy in the rat. Diabetologia. 38(8). 914–918. 36 indexed citations
15.
Nickander, Kim K., et al.. (1994). Effect of α-tocopherol deficiency on indices of oxidative stress in normal and diabetic peripheral nerve. Journal of the Neurological Sciences. 126(1). 6–14. 74 indexed citations
16.
Kihara, Mikihiro, Paula J. Zollman, James D. Schmelzer, & Phillip A. Low. (1993). The influence of dose of microspheres on nerve blood flow, electrophysiology, and fiber degeneration of rat peripheral nerve. Muscle & Nerve. 16(12). 1383–1389. 34 indexed citations
17.
18.
Low, Phillip A., et al.. (1988). Effect of hyperbaric oxygenation on normal and chronic streptozotocin diabetic peripheral nerves. Experimental Neurology. 99(1). 201–212. 41 indexed citations
19.
Schmelzer, James D. & Phillip A. Low. (1988). The effect of hyperbaric oxygenation and hypoxia on the blood-nerve barrier. Brain Research. 473(2). 321–326. 10 indexed citations
20.
Nagata, Hiroshi, Stephen Brimijoin, Phillip A. Low, & James D. Schmelzer. (1987). Slow axonal transport in experimental hypoxia and in neuropathy induced by p-bromophenylacetylurea. Brain Research. 422(2). 319–326. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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