Jacob Falck

8.6k total citations · 4 hit papers
28 papers, 6.9k citations indexed

About

Jacob Falck is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Jacob Falck has authored 28 papers receiving a total of 6.9k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 18 papers in Oncology and 9 papers in Cancer Research. Recurrent topics in Jacob Falck's work include DNA Repair Mechanisms (22 papers), Cancer-related Molecular Pathways (15 papers) and Carcinogens and Genotoxicity Assessment (9 papers). Jacob Falck is often cited by papers focused on DNA Repair Mechanisms (22 papers), Cancer-related Molecular Pathways (15 papers) and Carcinogens and Genotoxicity Assessment (9 papers). Jacob Falck collaborates with scholars based in Denmark, United Kingdom and United States. Jacob Falck's co-authors include Jiří Bártek, Stephen P. Jackson, Julia Coates, Claudia Lukas, Niels Mailand, Randi G. Syljuåsen, Graeme C.M. Smith, Ali Jazayeri, Jiřina Bártková and Markus Welcker and has published in prestigious journals such as Nature, Science and Journal of Biological Chemistry.

In The Last Decade

Jacob Falck

28 papers receiving 6.7k citations

Hit Papers

Conserved modes of recruitment of ATM, ATR and DNA-PKcs t... 2000 2026 2008 2017 2005 2005 2001 2000 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jacob Falck Denmark 26 6.2k 3.2k 1.6k 1.4k 634 28 6.9k
Shuhei Matsuoka Japan 12 6.6k 1.1× 3.1k 1.0× 1.4k 0.9× 1.3k 1.0× 695 1.1× 14 7.3k
Bin‐Bing S. Zhou United States 19 4.5k 0.7× 2.4k 0.7× 1.0k 0.6× 1.1k 0.8× 391 0.6× 42 5.6k
Christine E. Canman United States 34 6.0k 1.0× 4.1k 1.3× 1.6k 1.0× 910 0.7× 368 0.6× 49 7.2k
Sheau-Yann Shieh Taiwan 28 6.2k 1.0× 4.6k 1.4× 1.3k 0.8× 1.1k 0.8× 530 0.8× 39 7.4k
Larry M. Karnitz United States 45 5.9k 0.9× 2.8k 0.9× 1.1k 0.7× 1.0k 0.7× 343 0.5× 87 7.6k
Eva Petermann United Kingdom 33 5.5k 0.9× 2.7k 0.8× 982 0.6× 838 0.6× 475 0.7× 47 6.2k
Claus Storgaard Sørensen Denmark 40 5.3k 0.8× 2.4k 0.8× 839 0.5× 1.4k 1.0× 516 0.8× 65 6.0k
Xiaochun Yu United States 44 7.1k 1.1× 2.8k 0.9× 943 0.6× 914 0.7× 1.3k 2.1× 101 8.0k
Alwin Krämer Germany 39 4.0k 0.6× 2.2k 0.7× 814 0.5× 1.8k 1.3× 522 0.8× 124 5.9k
Randi G. Syljuåsen Norway 26 4.7k 0.7× 2.5k 0.8× 950 0.6× 1.4k 1.0× 340 0.5× 55 5.3k

Countries citing papers authored by Jacob Falck

Since Specialization
Citations

This map shows the geographic impact of Jacob Falck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jacob Falck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jacob Falck more than expected).

Fields of papers citing papers by Jacob Falck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jacob Falck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jacob Falck. The network helps show where Jacob Falck may publish in the future.

Co-authorship network of co-authors of Jacob Falck

This figure shows the co-authorship network connecting the top 25 collaborators of Jacob Falck. A scholar is included among the top collaborators of Jacob Falck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jacob Falck. Jacob Falck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Falck, Jacob, Josep V. Forment, Julia Coates, et al.. (2012). CDK targeting of NBS1 promotes DNA‐end resection, replication restart and homologous recombination. EMBO Reports. 13(6). 561–568. 86 indexed citations
2.
Fugger, Kasper, Martin Mistrík, Christoffel Dinant, et al.. (2009). Human Fbh1 helicase contributes to genome maintenance via pro- and anti-recombinase activities. The Journal of Cell Biology. 186(5). 655–663. 81 indexed citations
3.
Danielsen, Jannie M. Rendtlew, et al.. (2008). HCLK2 Is Required for Activity of the DNA Damage Response Kinase ATR. Journal of Biological Chemistry. 284(7). 4140–4147. 36 indexed citations
4.
Falck, Jacob, Julia Coates, & Stephen P. Jackson. (2005). Conserved modes of recruitment of ATM, ATR and DNA-PKcs to sites of DNA damage. Nature. 434(7033). 605–611. 983 indexed citations breakdown →
5.
Jazayeri, Ali, Jacob Falck, Claudia Lukas, et al.. (2005). ATM- and cell cycle-dependent regulation of ATR in response to DNA double-strand breaks. Nature Cell Biology. 8(1). 37–45. 832 indexed citations breakdown →
6.
Falck, Jacob, et al.. (2004). Differential impact of diverse anticancer chemotherapeutics on the Cdc25A-degradation checkpoint pathway. Experimental Cell Research. 302(2). 162–169. 26 indexed citations
7.
Kilpivaara, Outi, Pia Vahteristo, Jacob Falck, et al.. (2004). CHEK2 variant I157T may be associated with increased breast cancer risk. International Journal of Cancer. 111(4). 543–547. 110 indexed citations
8.
Lukas, Claudia, Fredrik Melander, Manuel Stucki, et al.. (2004). Mdc1 couples DNA double‐strand break recognition by Nbs1 with its H2AX‐dependent chromatin retention. The EMBO Journal. 23(13). 2674–2683. 313 indexed citations
9.
Lukas, Claudia, et al.. (2003). Distinct spatiotemporal dynamics of mammalian checkpoint regulators induced by DNA damage. Nature Cell Biology. 5(3). 255–260. 375 indexed citations
10.
Gatei, Magtouf, Claus Storgaard Sørensen, Randi G. Syljuåsen, et al.. (2003). Ataxia-telangiectasia-mutated (ATM) and NBS1-dependent Phosphorylation of Chk1 on Ser-317 in Response to Ionizing Radiation. Journal of Biological Chemistry. 278(17). 14806–14811. 238 indexed citations
11.
Sørensen, Claus Storgaard, Randi G. Syljuåsen, Jacob Falck, et al.. (2003). Chk1 regulates the S phase checkpoint by coupling the physiological turnover and ionizing radiation-induced accelerated proteolysis of Cdc25A. Cancer Cell. 3(3). 247–258. 416 indexed citations
12.
Goldberg, Michal, Manuel Stucki, Jacob Falck, et al.. (2003). MDC1 is required for the intra-S-phase DNA damage checkpoint. Nature. 421(6926). 952–956. 411 indexed citations
13.
Falck, Jacob, et al.. (2002). The DNA damage-dependent intra–S phase checkpoint is regulated by parallel pathways. Nature Genetics. 30(3). 290–294. 303 indexed citations
14.
Bártek, Jiří, et al.. (2001). Chk2 kinase — a busy messenger. Nature Reviews Molecular Cell Biology. 2(12). 877–886. 314 indexed citations
15.
Falck, Jacob, et al.. (2001). Functional impact of concomitant versus alternative defects in the Chk2-p53 tumour suppressor pathway. Oncogene. 20(39). 5503–5510. 93 indexed citations
16.
Bártková, Jiřina, et al.. (2001). Chk2 tumour suppressor protein in human spermatogenesis and testicular germ-cell tumours. Oncogene. 20(41). 5897–5902. 57 indexed citations
17.
Santoni‐Rugiu, Eric, et al.. (2000). Involvement of Myc Activity in a G 1 /S-Promoting Mechanism Parallel to the pRb/E2F Pathway. Molecular and Cellular Biology. 20(10). 3497–3509. 142 indexed citations
18.
Mailand, Niels, Jacob Falck, Claudia Lukas, et al.. (2000). Rapid Destruction of Human Cdc25A in Response to DNA Damage. Science. 288(5470). 1425–1429. 612 indexed citations breakdown →
19.
Falck, Jacob, Peter Buhl Jensen, & Maxwell Sehested. (1999). Evidence for Repressional Role of an Inverted CCAAT Box in Cell Cycle-dependent Transcription of the Human DNA Topoisomerase IIα Gene. Journal of Biological Chemistry. 274(26). 18753–18758. 27 indexed citations
20.
Wessel, Irene, et al.. (1997). Loss of amino acids 1490Lys-Ser-Lys1492 in the COOH-terminal region of topoisomerase IIalpha in human small cell lung cancer cells selected for resistance to etoposide results in an extranuclear enzyme localization.. PubMed. 57(20). 4451–4. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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