Eva Petermann

8.1k total citations · 3 hit papers
47 papers, 6.2k citations indexed

About

Eva Petermann is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Eva Petermann has authored 47 papers receiving a total of 6.2k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 21 papers in Oncology and 12 papers in Cell Biology. Recurrent topics in Eva Petermann's work include DNA Repair Mechanisms (37 papers), Cancer-related Molecular Pathways (13 papers) and Microtubule and mitosis dynamics (12 papers). Eva Petermann is often cited by papers focused on DNA Repair Mechanisms (37 papers), Cancer-related Molecular Pathways (13 papers) and Microtubule and mitosis dynamics (12 papers). Eva Petermann collaborates with scholars based in United Kingdom, Sweden and United States. Eva Petermann's co-authors include Thomas Helleday, Ricky A. Sharma, Ben Hodgson, Cecilia Lundin, Niklas Schultz, Natalia Issaeva, Keith W. Caldecott, Panagiotis Kotsantis, Rebecca M. Jones and Manuel Luís Orta and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Nucleic Acids Research.

In The Last Decade

Eva Petermann

46 papers receiving 6.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

DNA repair pathways as targets for cancer therapy

2008 1.3k citations Thomas Helleday, Eva Petermann et al. profile →
Hydroxyurea-Stalled Replication Forks Become Progressively Inactivated and Require Two Different RAD51-Mediated Pathways for Restart and Repair

2010 643 citations Eva Petermann, Thomas Helleday et al. profile →
Sources, resolution and physiological relevance of R-loops and RNA–DNA hybrids

2022 223 citations Eva Petermann, Li Lan et al. Nature Reviews Molecular Cell Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Eva Petermann United Kingdom	33	5.5k	2.7k	982	838	475	47	6.2k
Bin‐Bing S. Zhou United States	19	4.5k 0.8×	2.4k 0.9×	1.0k 1.0×	1.1k 1.3×	391 0.8×	42	5.6k
Christopher J. Bakkenist United States	34	5.5k 1.0×	2.6k 1.0×	1.5k 1.5×	656 0.8×	392 0.8×	82	6.6k
Claus Storgaard Sørensen Denmark	40	5.3k 1.0×	2.4k 0.9×	839 0.9×	1.4k 1.7×	516 1.1×	65	6.0k
Lisa Wiesmüller Germany	42	4.6k 0.8×	2.4k 0.9×	824 0.8×	668 0.8×	487 1.0×	136	5.8k
Randi G. Syljuåsen Norway	26	4.7k 0.8×	2.5k 0.9×	950 1.0×	1.4k 1.7×	340 0.7×	55	5.3k
Sheau-Yann Shieh Taiwan	28	6.2k 1.1×	4.6k 1.7×	1.3k 1.3×	1.1k 1.3×	530 1.1×	39	7.4k
Steven P. Linke United States	26	3.5k 0.6×	2.2k 0.8×	965 1.0×	638 0.8×	686 1.4×	41	4.7k
Ji‐Hoon Lee United States	30	4.6k 0.8×	1.8k 0.7×	1.2k 1.2×	550 0.7×	359 0.8×	56	5.2k
Brian Gabrielli Australia	47	4.6k 0.8×	2.1k 0.8×	887 0.9×	1.6k 1.9×	346 0.7×	129	6.0k
Christine E. Canman United States	34	6.0k 1.1×	4.1k 1.5×	1.6k 1.7×	910 1.1×	368 0.8×	49	7.2k

Countries citing papers authored by Eva Petermann

Since Specialization

Citations

This map shows the geographic impact of Eva Petermann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Petermann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Petermann more than expected).

Fields of papers citing papers by Eva Petermann

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Petermann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Petermann. The network helps show where Eva Petermann may publish in the future.

Co-authorship network of co-authors of Eva Petermann

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Petermann. A scholar is included among the top collaborators of Eva Petermann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Petermann. Eva Petermann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wilson, Claire, Richard D. Kelly, Panagiotis Kotsantis, et al.. (2025). Human RNase H2 upregulation counteracts oncogene- and chemotherapy-induced replication stress. Oncogene. 44(35). 3255–3271.

Petermann, Eva, Li Lan, & Lee Zou. (2022). Sources, resolution and physiological relevance of R-loops and RNA–DNA hybrids. Nature Reviews Molecular Cell Biology. 23(8). 521–540. 223 indexed citations breakdown →

Petermann, Eva, Li Lan, & Lee Zou. (2022). Author Correction: Sources, resolution and physiological relevance of R-loops and RNA–DNA hybrids. Nature Reviews Molecular Cell Biology. 23(6). 444–444. 4 indexed citations

González, Elena, Carl Ward, George J. Murphy, et al.. (2021). MYBL2 and ATM suppress replication stress in pluripotent stem cells. EMBO Reports. 22(5). e51120–e51120. 14 indexed citations

Piberger, Ann Liza, Richard D. Kelly, Alexandra K. Walker, et al.. (2020). PrimPol-dependent single-stranded gap formation mediates homologous recombination at bulky DNA adducts. Nature Communications. 11(1). 5863–5863. 81 indexed citations

Bayley, Rachel, Stéphanie Dumon, Giacomo Volpe, et al.. (2018). MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells. Cancer Research. 78(20). 5767–5779. 18 indexed citations

Benedict, Bente, Marleen Dekker, Aslι Küçükosmanoğlu, et al.. (2018). Loss of p53 suppresses replication-stress-induced DNA breakage in G1/S checkpoint deficient cells. eLife. 7. 39 indexed citations

Ronson, George E., Ann Liza Piberger, Martin R. Higgs, et al.. (2018). PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation. Nature Communications. 9(1). 746–746. 171 indexed citations

Piberger, Ann Liza, et al.. (2018). BET Inhibition Induces HEXIM1- and RAD51-Dependent Conflicts between Transcription and Replication. Cell Reports. 25(8). 2061–2069.e4. 39 indexed citations

10.

Kotsantis, Panagiotis, Rebecca M. Jones, Martin R. Higgs, & Eva Petermann. (2015). Cancer Therapy and Replication Stress. Advances in clinical chemistry. 69. 91–138. 21 indexed citations

11.

Jones, Rebecca M., Panagiotis Kotsantis, Grant S. Stewart, Petra Groth, & Eva Petermann. (2014). BRCA2 and RAD51 Promote Double-Strand Break Formation and Cell Death in Response to Gemcitabine. Molecular Cancer Therapeutics. 13(10). 2412–2421. 38 indexed citations

12.

Kwok, Marwan, Nicholas Davies, Angelo Agathanggelou, et al.. (2014). ATR Inhibition Exacerbates Replication Stress in TP53 or ATM Deficient CLL Cells and Enhances Sensitivity to Chemotherapy and Targeted Therapy. Blood. 124(21). 3340–3340. 1 indexed citations

13.

Savill, Kristin M. Zimmerman, Rebecca M. Jones, Eva Petermann, & Penelope A. Jeggo. (2013). Diminished Origin-Licensing Capacity Specifically Sensitizes Tumor Cells to Replication Stress. Molecular Cancer Research. 11(4). 370–380. 33 indexed citations

14.

Jones, Rebecca M. & Eva Petermann. (2012). Replication fork dynamics and the DNA damage response. Biochemical Journal. 443(1). 13–26. 93 indexed citations

15.

Wilsker, Deborah, Jon Chung, Iván Pradilla, et al.. (2011). Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival. Molecular Cancer Therapeutics. 11(1). 98–107. 4 indexed citations

16.

Petermann, Eva, Mick Woodcock, & Thomas Helleday. (2010). Chk1 promotes replication fork progression by controlling replication initiation. Proceedings of the National Academy of Sciences. 107(37). 16090–16095. 221 indexed citations

17.

Petermann, Eva & Thomas Helleday. (2007). DNA replication-associated lesions: importance in early tumorigenesis and cancer therapy. Biochemical Society Transactions. 35(5). 1352–1354. 3 indexed citations

18.

Petermann, Eva, Apolinar Maya‐Mendoza, George Zachos, et al.. (2006). Chk1 Requirement for High Global Rates of Replication Fork Progression during Normal Vertebrate S Phase. Molecular and Cellular Biology. 26(8). 3319–3326. 148 indexed citations

19.

Breslin, Claire, Paula Clements, Sherif F. El‐Khamisy, et al.. (2006). Measurement of Chromosomal DNA Single‐Strand Breaks and Replication Fork Progression Rates. Methods in enzymology on CD-ROM/Methods in enzymology. 409. 410–425. 39 indexed citations

20.

Keil, Claudia, Eva Petermann, & Shiao Li Oei. (2004). Tannins elevate the level of poly(ADP–ribose) in HeLa cell extracts. Archives of Biochemistry and Biophysics. 425(1). 115–121. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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