J. N. Walton

2.7k total citations
50 papers, 2.1k citations indexed

About

J. N. Walton is a scholar working on Molecular Biology, Neurology and Surgery. According to data from OpenAlex, J. N. Walton has authored 50 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 8 papers in Neurology and 7 papers in Surgery. Recurrent topics in J. N. Walton's work include Muscle Physiology and Disorders (13 papers), Neurogenetic and Muscular Disorders Research (7 papers) and Genetic Neurodegenerative Diseases (5 papers). J. N. Walton is often cited by papers focused on Muscle Physiology and Disorders (13 papers), Neurogenetic and Muscular Disorders Research (7 papers) and Genetic Neurodegenerative Diseases (5 papers). J. N. Walton collaborates with scholars based in United Kingdom, United States and India. J. N. Walton's co-authors include E. F. Fincham, D. Gardner‐Medwin, B. E. Tomlinson, G. W. Pearce, R. J. Pennington, F.G.I. Jennekens, Walter G. Bradley, P. Hudgson, L.P. Lassman and John Pearce and has published in prestigious journals such as Nature, The Lancet and Brain.

In The Last Decade

J. N. Walton

49 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
J. N. Walton United Kingdom 26 711 401 352 337 299 50 2.1k
H. G. Lenard Germany 29 512 0.7× 286 0.7× 313 0.9× 253 0.8× 96 0.3× 91 2.1k
Martin S. Schwartz United Kingdom 28 376 0.5× 704 1.8× 216 0.6× 370 1.1× 281 0.9× 83 2.0k
Don M. Long United States 27 432 0.6× 611 1.5× 264 0.8× 449 1.3× 137 0.5× 86 2.8k
P. Fawcett United Kingdom 23 592 0.8× 825 2.1× 286 0.8× 538 1.6× 584 2.0× 58 2.2k
Byron A. Kakulas Australia 28 878 1.2× 370 0.9× 222 0.6× 522 1.5× 138 0.5× 134 2.6k
Francis Mastaglia Australia 26 436 0.6× 478 1.2× 139 0.4× 293 0.9× 391 1.3× 79 2.3k
D A Ingram United Kingdom 26 466 0.7× 640 1.6× 143 0.4× 442 1.3× 391 1.3× 43 2.3k
W. G. P. Mair United Kingdom 18 273 0.4× 431 1.1× 143 0.4× 480 1.4× 64 0.2× 33 1.7k
D. G. F. Harriman United Kingdom 24 587 0.8× 271 0.7× 132 0.4× 370 1.1× 88 0.3× 53 1.6k
P. Tonali Italy 32 1.2k 1.7× 624 1.6× 182 0.5× 640 1.9× 85 0.3× 103 3.0k

Countries citing papers authored by J. N. Walton

Since Specialization
Citations

This map shows the geographic impact of J. N. Walton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. N. Walton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. N. Walton more than expected).

Fields of papers citing papers by J. N. Walton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. N. Walton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. N. Walton. The network helps show where J. N. Walton may publish in the future.

Co-authorship network of co-authors of J. N. Walton

This figure shows the co-authorship network connecting the top 25 collaborators of J. N. Walton. A scholar is included among the top collaborators of J. N. Walton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. N. Walton. J. N. Walton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nicholson, Garth A., R. Lane, D. Gardner‐Medwin, & J. N. Walton. (1981). Carrier testing in families of isolated cases of duchenne muscular dystrophy. Journal of the Neurological Sciences. 51(1). 29–42. 4 indexed citations
2.
Nicholson, Garth A., R. J. Pennington, D. Gardner‐Medwin, & J. N. Walton. (1979). CARRIER DETECTION IN DUCHENNE MUSCULAR DYSTROPHY: ASSESSMENT OF THE EFFECT OF AGE ON DETECTION-RATE WITH SERUM-CREATINE-KINASE-ACTIVITY. The Lancet. 313(8118). 692–694. 32 indexed citations
3.
Pearn, John, et al.. (1978). A genetic study of subacute and chronic spinal muscular atrophy in childhood. Journal of the Neurological Sciences. 37(3). 227–248. 27 indexed citations
4.
Pearn, John, P. Hudgson, & J. N. Walton. (1978). A Clinical and Genetic Study of Spinal Muscular Atrophy of Adult Onset: The Autosomal Recessive Form as a Discrete Disease Entity. Brain. 101(4). 591–606. 53 indexed citations
5.
Tomlinson, B. E., J. N. Walton, & Dorothy Irving. (1974). Spinal cord limb motor neurones in muscular dystrophy. Journal of the Neurological Sciences. 22(3). 305–327. 28 indexed citations
6.
Romero-Herrera, A.E., H. Lehmann, B. E. Tomlinson, & J. N. Walton. (1973). Myoglobin in Primary Muscular Disease: I. Duchenne Muscular Dystrophy: and: II. Muscular Dystrophy of Distal Type. Journal of Medical Genetics. 10(4). 309–322. 7 indexed citations
7.
Walton, J. N.. (1972). Birth and brain damage. Journal of the Neurological Sciences. 15(1). 123–123. 19 indexed citations
8.
Mastaglia, Frank & J. N. Walton. (1971). An electron microscopic study of skeletal muscle from cases of the Kugelberg-Welander syndrome. Acta Neuropathologica. 17(3). 201–219. 10 indexed citations
9.
Jennekens, F.G.I., B. E. Tomlinson, & J. N. Walton. (1971). The sizes of the two main histochemical fibre types in five limb muscles in man. Journal of the Neurological Sciences. 13(3). 281–292. 37 indexed citations
10.
Aherne, W., D. R. Ayyar, Pamela A.G. Clarke, & J. N. Walton. (1971). Muscle fibre size in normal infants, children and adolescents. Journal of the Neurological Sciences. 14(2). 171–182. 40 indexed citations
11.
Bradley, Walter G., L.P. Lassman, G. W. Pearce, & J. N. Walton. (1970). The neuromyopathy of vincristine in man. Journal of the Neurological Sciences. 10(2). 107–131. 193 indexed citations
12.
Walton, J. N.. (1970). Motor neuron diseases: Research on amyotrophic lateral sclerosis and related disorders. Journal of the Neurological Sciences. 11(2). 201–202. 19 indexed citations
13.
Walton, J. N.. (1969). Muscular dystrophies and their management.. BMJ. 3(5671). 639–642. 2 indexed citations
14.
Fulthorpe, J. J., D. Gardner‐Medwin, P. Hudgson, & J. N. Walton. (1969). Nemaline myopathy. Neurology. 19(8). 735–735. 19 indexed citations
15.
Knowles, M., M Saunders, S. Currie, J. N. Walton, & E. J. Field. (1969). LYMPHOCYTE TRANSFORMATION IN THE GUILLAIN-BARRE SYNDROME. The Lancet. 294(7631). 1168–1170. 34 indexed citations
16.
Pearce, John, S. S. Gubbay, & J. N. Walton. (1965). LONG-TERM ANTICOAGULANT THERAPY IN TRANSIENT CEREBRAL ISCHÆMIC ATTACKS. The Lancet. 285(7375). 6–9. 60 indexed citations
17.
Pearce, John, S. S. Gubbay, John Hardy, et al.. (1964). Laevadosin in Treatment of the Duchenne Type of Muscular Dystrophy: Preliminary Results of a Double-blind Controlled Trial. BMJ. 2(5414). 915–917. 6 indexed citations
18.
Walton, J. N., et al.. (1961). BENIGN MYALGIC ENCEPHALOMYELITIS IN NEWCASTLE UPON TYNE. The Lancet. 277(7180). 733–737. 9 indexed citations
19.
Walton, J. N.. (1956). AMYOTONIA CONGENITA. The Lancet. 267(6931). 1023–1028. 58 indexed citations
20.
Walton, J. N.. (1952). THE ELECTROMYOGRAM IN MYOPATHY: ANALYSIS WITH THE AUDIO-FREQUENCY SPECTROMETER. Journal of Neurology Neurosurgery & Psychiatry. 15(4). 219–226. 76 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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