J. Kint

1.8k citations
41 papers · 1.4k indexed · 1 hit paper · h-index 18

J. Kint

40 papers receiving 1.2k citations

Hit Papers

Fabry's Disease: Alpha-Galactosidase Deficiency3751970202619882007100200300

Peers

J. Kint
Comparison fields: 5 of 118
  • Physiology 606
  • Clinical Biochemistry 89
  • Rheumatology 196
  • Cell Biology 174
  • Organic Chemistry 259
Replace Ilkka Mononen with:
Ilkka Mononen Finland
Emmanuel Shapira United States
Fabrizio Barbetti Italy
Wayne L. Ryan United States
Axel Périanin France
Y. S. Shin Germany
Sylvie Marleau Canada
Catherine Figarella France
Mohamed Lamkanfi Belgium
Amiram Ravid Israel
J. Kint relative to Ilkka Mononen Finland Ilkka Mononen's profile →
Citations per field
00.5×1.6×
Ilkka Mononen · 1×
Citations per year

Countries citing papers authored by J. Kint

Since Specialization
Citations

This map shows the geographic impact of J. Kint's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Kint with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Kint more than expected).

Fields of papers citing papers by J. Kint

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Kint. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Kint. The network helps show where J. Kint may publish in the future.

Co-authorship network

The 25 scholars most cited alongside J. Kint, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with J. Kint Line = papers co-authored together J. Kint links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20134
2 20119
3 200916
4 20029
5 199885
6 19963
7 199622
8 19949
9 199410
10 199335
11 199152
12 198823
13 19887
14 198719
15 1979163
16 197839
17 197413
18
La mucolipidose de type II (I-cell disease)
19737
19
Biochemical studies in relation to the possible germination regulatory role of naturally occurring coumarin and phenolics
197254
20 196920

About J. Kint

J. Kint is a scholar working on Physiology, Physiology, Equine, Small Animals and Clinical Biochemistry, having authored 41 papers that have together received 1.4k indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (16 papers), Glycosylation and Glycoproteins Research (9 papers), Carbohydrate Chemistry and Synthesis (5 papers), Erythrocyte Function and Pathophysiology (4 papers), Biomedical Research and Pathophysiology (4 papers), Animal health and immunology (4 papers), Glycogen Storage Diseases and Myoclonus (4 papers) and Neonatal Health and Biochemistry (3 papers). The work is most often cited by research in Physiology (606 citations), Clinical Biochemistry (89 citations), Rheumatology (196 citations), Cell Biology (174 citations) and Organic Chemistry (259 citations). J. Kint has collaborated with scholars based in Belgium, Netherlands and Sweden. Frequent co-authors include G. Dacremont, M. Mareel, C. F. van Sumere, D. Carton, C Hooft, H. Teuchy, G Wolf, Jaak Jaeken, Helena Stibler and V. Ramaekers. Their work appears in journals such as Clinica Chimica Acta, Nature, Science, Journal of Inherited Metabolic Disease and Pediatric Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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