J. Keith McClung

1.3k total citations
25 papers, 1.1k citations indexed

About

J. Keith McClung is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, J. Keith McClung has authored 25 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Genetics. Recurrent topics in J. Keith McClung's work include Nuclear Structure and Function (8 papers), RNA Research and Splicing (7 papers) and Cancer-related Molecular Pathways (5 papers). J. Keith McClung is often cited by papers focused on Nuclear Structure and Function (8 papers), RNA Research and Splicing (7 papers) and Cancer-related Molecular Pathways (5 papers). J. Keith McClung collaborates with scholars based in United States. J. Keith McClung's co-authors include Robert T. Dell’Orco, James R. Smith, Charles K. Lumpkin, Eldon R. Jupe, David B. Danner, Olivia M. Pereira‐Smith, Mark J. Nuell, David A. Stewart, Edward L. Schneider and Michael R. Miller and has published in prestigious journals such as Science, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

J. Keith McClung

25 papers receiving 1.1k citations

Peers

J. Keith McClung

PHYSI

ONCOL

Nicholas R. Helps United Kingdom

Michiko Minami Japan

Jack O. Hensold United States

Robert S. Mann United States

J.J. Keusch Switzerland

David G. Campbell United Kingdom

Jennifer D. Luethy United States

Kohsuke Nonoguchi Japan

Elena Zelin United States

Mamoru Aoto Japan

Nicholas R. Helps United Kingdom

J. Keith McClung

1.4k ×1.6

115 ×0.7

PHYSI

190 ×1.2

ONCOL

448 ×2.9

98 ×0.9

Citations per year, relative to J. Keith McClung J. Keith McClung (= 1×) peers Nicholas R. Helps

Countries citing papers authored by J. Keith McClung

Since Specialization

Citations

This map shows the geographic impact of J. Keith McClung's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Keith McClung with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Keith McClung more than expected).

Fields of papers citing papers by J. Keith McClung

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Keith McClung. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Keith McClung. The network helps show where J. Keith McClung may publish in the future.

Co-authorship network of co-authors of J. Keith McClung

This figure shows the co-authorship network connecting the top 25 collaborators of J. Keith McClung. A scholar is included among the top collaborators of J. Keith McClung based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. Keith McClung. J. Keith McClung is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Narasimhan, Sukanya, M Armstrong, J. Keith McClung, F F Richards, & Eleanor K. Spicer. (1997). Prohibitin, a putative negative control element present in Pneumocystis carinii. Infection and Immunity. 65(12). 5125–5130. 12 indexed citations

Jupe, Eldon R., et al.. (1996). The 3′ Untranslated Region of Prohibitin and Cellular Immortalization. Experimental Cell Research. 224(1). 128–135. 63 indexed citations

Dell’Orco, Robert T., et al.. (1996). Prohibitin and the senescent phenotype. Experimental Gerontology. 31(1-2). 245–252. 52 indexed citations

Altus, Michael S., Carla M. Wood, David A. Stewart, et al.. (1995). Regions of evolutionary conservation between the rat and human prohibitin-encoding genes. Gene. 158(2). 291–294. 19 indexed citations

Lumpkin, Charles K., et al.. (1995). Acute ethanol and selected growth suppressor transcripts in regenerating rat liver. Alcohol. 12(4). 357–362. 10 indexed citations

Jupe, Eldon R., et al.. (1995). Prohibitin Antiproliferative Activity and Lack of Heterozygosity in Immortalized Cell Lines. Experimental Cell Research. 218(2). 577–580. 31 indexed citations

McClung, J. Keith, et al.. (1995). Prohibitin: Potential role in senescence, development, and tumor suppression. Experimental Gerontology. 30(2). 99–124. 163 indexed citations

Stewart, Charles A., et al.. (1994). Prohibitin Expression during Cellular Senescence of Human Diploid Fibroblasts. Biochemical and Biophysical Research Communications. 201(1). 409–414. 40 indexed citations

Roskams, A. Jane, Varda Friedman, Carla M. Wood, et al.. (1993). Cell cycle activity and expression of prohibitin mRNA. Journal of Cellular Physiology. 157(2). 289–295. 72 indexed citations

10.

Lumpkin, Charles K., et al.. (1992). The effects of acute ethanol on growth in rat liver: Steady state c-myc transcripts. Alcohol. 9(3). 279–282. 3 indexed citations

11.

McClung, J. Keith, Robert L. King, David B. Danner, et al.. (1992). Expression of prohibitin, an antiproliferative protein. Experimental Gerontology. 27(4). 413–417. 28 indexed citations

12.

White, John J., Roger L. Eddy, T.B. Shows, et al.. (1991). Assignment of the human prohibition gene (PHB) to chromosome 17 and identification of a DNA polymorphism. Genomics. 11(1). 228–230. 36 indexed citations

13.

Nuell, Mark J., David A. Stewart, Lisa Walker, et al.. (1991). Prohibitin, an Evolutionarily Conserved Intracellular Protein That Blocks DNA Synthesis in Normal Fibroblasts and HeLa Cells. Molecular and Cellular Biology. 11(3). 1372–1381. 56 indexed citations

14.

Hammond, Russell A., J. Keith McClung, & Michael R. Miller. (1990). Effect of DNA polymerase inhibitors on DNA repair in intact and permeable human fibroblasts: evidence that DNA polymerases .delta. and .beta. are involved in DNA repair synthesis induced by N-methyl-N'-nitro-N-nitrosoguanidine. Biochemistry. 29(1). 286–291. 59 indexed citations

15.

McClung, J. Keith & Robert A. Gonzales. (1989). Purification of plasmid DNA by fast protein liquid chromatography on superose 6 preparative grade. Analytical Biochemistry. 177(2). 378–382. 26 indexed citations

16.

McClung, J. Keith, David B. Danner, David A. Stewart, et al.. (1989). Isolation of a cDNA that hybrid selects antiproliferative mRNA from rat liver. Biochemical and Biophysical Research Communications. 164(3). 1316–1322. 145 indexed citations

17.

Nuell, Mark J., J. Keith McClung, James R. Smith, & David B. Danner. (1989). Approach to the isolation of antiproliferative genes. Experimental Gerontology. 24(5-6). 469–476. 14 indexed citations

18.

Lumpkin, Charles K., J. Keith McClung, Olivia M. Pereira‐Smith, & James R. Smith. (1986). Existence of High Abundance Antiproliferative mRNA's in Senescent Human Diploid Fibroblasts. Science. 232(4748). 393–395. 165 indexed citations

19.

Lumpkin, Charles K., J. Keith McClung, & James R. Smith. (1985). Entry into S phase is inhibited in human fibroblasts by rat liver poly(A) + RNA. Experimental Cell Research. 160(2). 544–549. 30 indexed citations

20.

McClung, J. Keith & Rolf F. Kletzien. (1984). Analysis of BHK Cell Growth Kinetics After Microinjection of Catalytic Subunit of Cyclic AMP-Dependent Protein Kinase. Molecular and Cellular Biology. 4(6). 1079–1085. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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