Robert S. Mann
- Molecular Biology top 10%
- Developmental Biology and Gene Regulation 4
- Epigenetics and DNA Methylation 3
- Histone Deacetylase Inhibitors Research 2
- Hedgehog Signaling Pathway Studies 1
- Ubiquitin and proteasome pathways 1
- Hematology top 10%
- Cell Biology top 10%
- Zebrafish Biomedical Research Applications 2
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- Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities 1
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- Peptidase Inhibition and Analysis 1
- Co-authors
- V. G. AllfreyG. VidaliL.C. BoffaSpyros Artavanis‐TsakonasGrace GrayThimios A. MitsiadisDavid FlowersSteven J. Staats
- Partner nations
- United StatesUnited KingdomSweden
In The Last Decade
Robert S. Mann
9 papers receiving 1.3k citations
Hit Papers
Peers
Comparison fields: 5 of 94
- Molecular Biology 1.0k
- Hematology 111
- Immunology 182
- Cell Biology 120
- Cancer Research 109
Countries citing papers authored by Robert S. Mann
This map shows the geographic impact of Robert S. Mann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert S. Mann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert S. Mann more than expected).
Fields of papers citing papers by Robert S. Mann
This network shows the impact of papers produced by Robert S. Mann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert S. Mann. The network helps show where Robert S. Mann may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Robert S. Mann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2000 | 82 | |
| 2 | 1999 | 165 | |
| 3 | 1999 | 156 | |
| 4 | 1999 | 132 | |
| 5 | 1998 | 285 | |
| 6 | 1998 | 22 | |
| 7 | 1984 | 15 | |
| 8 | Suppression of histone deacetylation in vivo and in vitro by sodium butyrate.breakdown → | 1978 | 406 |
| 9 | 1978 | 51 |
About Robert S. Mann
Robert S. Mann is a scholar working on Cell Biology, Reproductive Medicine, Molecular Biology, Oncology and Cancer Research, having authored 9 papers that have together received 1.3k indexed citations. Recurring topics across this work include Developmental Biology and Gene Regulation (4 papers), Epigenetics and DNA Methylation (3 papers), Histone Deacetylase Inhibitors Research (2 papers), Zebrafish Biomedical Research Applications (2 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (1 paper), Hedgehog Signaling Pathway Studies (1 paper), Peptidase Inhibition and Analysis (1 paper) and Ubiquitin and proteasome pathways (1 paper). The work is most often cited by research in Molecular Biology (1.0k citations), Hematology (111 citations), Immunology (182 citations), Cell Biology (120 citations) and Cancer Research (109 citations). Robert S. Mann has collaborated with scholars based in United States, United Kingdom and Sweden. Frequent co-authors include V. G. Allfrey, G. Vidali, L.C. Boffa, Spyros Artavanis‐Tsakonas, Grace Gray, Thimios A. Mitsiadis, David Flowers, Steven J. Staats, Kateri Moore and Barbara Varnum‐Finney. Their work appears in journals such as Blood, Genetics, Journal of Biological Chemistry, Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids and American Journal Of Pathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.