Iwona Gumper
- Molecular Biology top 10%
- Cell Biology top 5%
- Epidemiology
- Physiology
- Surgery
- Co-authors
- David D. SabatiniEdward M. De RobertisRadek DobrowolskiLuis C. FuentealbaVincent TaelmanJean-Louis PlouhinecDavid M. SabatiniRoberto Zoncu
- Topics
- Wnt/β-catenin signaling in development and cancer (4 papers)Cellular transport and secretion (4 papers)Skin and Cellular Biology Research (3 papers)
- Cited by
- Cell BiologyPhysiologyAging
- Partner nations
- United StatesSwitzerlandJapan
In The Last Decade
Iwona Gumper
10 papers receiving 1.2k citations
Hit Papers
Peers
Comparison fields: 5 of 80
- Molecular Biology 880
- Cell Biology 362
- Epidemiology 184
- Physiology 119
- Surgery 113
Countries citing papers authored by Iwona Gumper
This map shows the geographic impact of Iwona Gumper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Iwona Gumper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Iwona Gumper more than expected).
Fields of papers citing papers by Iwona Gumper
This network shows the impact of papers produced by Iwona Gumper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Iwona Gumper. The network helps show where Iwona Gumper may publish in the future.
Co-authorship network of co-authors of Iwona Gumper
This figure shows the co-authorship network connecting the top 25 collaborators of Iwona Gumper. A scholar is included among the top collaborators of Iwona Gumper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Iwona Gumper. Iwona Gumper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 40 | |
| 2 | Rag GTPase-mediated regulation of mTORC1 by nutrients is necessary for neonatal autophagy and survival | 1 |
| 3 | 355 | |
| 4 | 69 | |
| 5 | Wnt Signaling Requires Sequestration of Glycogen Synthase Kinase 3 inside Multivesicular Endosomesbreakdown → | 568 |
| 6 | 31 | |
| 7 | 46 | |
| 8 | 11 | |
| 9 | 37 | |
| 10 | A Golgi-related structure remains after the brefeldin A-induced formation of an ER-Golgi hybrid compartment. | 24 |
About Iwona Gumper
Iwona Gumper is a scholar working on Cell Biology, Immunology and Allergy and Molecular Biology, having authored 10 papers that have together received 1.2k indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (4 papers), Cellular transport and secretion (4 papers) and Skin and Cellular Biology Research (3 papers). The work is most often cited by research in Cell Biology (362 citations), Physiology (80 citations) and Aging (27 citations). Iwona Gumper has collaborated with scholars based in United States, Switzerland and Japan. Frequent co-authors include David D. Sabatini, Edward M. De Robertis, Radek Dobrowolski, Luis C. Fuentealba, Vincent Taelman, Jean-Louis Plouhinec, David M. Sabatini, Roberto Zoncu, Rachel L. Wolfson and Alejo Efeyan. Their work appears in journals such as Nature, Cell and Journal of Investigative Dermatology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.