Ito Kawakami

708 total citations
27 papers, 428 citations indexed

About

Ito Kawakami is a scholar working on Neurology, Physiology and Neurology. According to data from OpenAlex, Ito Kawakami has authored 27 papers receiving a total of 428 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Neurology, 13 papers in Physiology and 7 papers in Neurology. Recurrent topics in Ito Kawakami's work include Alzheimer's disease research and treatments (12 papers), Parkinson's Disease Mechanisms and Treatments (10 papers) and Amyotrophic Lateral Sclerosis Research (9 papers). Ito Kawakami is often cited by papers focused on Alzheimer's disease research and treatments (12 papers), Parkinson's Disease Mechanisms and Treatments (10 papers) and Amyotrophic Lateral Sclerosis Research (9 papers). Ito Kawakami collaborates with scholars based in Japan, United States and United Kingdom. Ito Kawakami's co-authors include Masato Hasegawa, Tetsuaki Arai, Haruhiko Akiyama, Shinji Higashi, Kenichi Oshima, Kazuhiro Niizato, Yuko Saito, Shigeo Murayama, Tomoyasu Matsubara and Masato Hosokawa and has published in prestigious journals such as Nature, Brain and Acta Neuropathologica.

In The Last Decade

Ito Kawakami

25 papers receiving 422 citations

Peers

Ito Kawakami
Mehtap Bacioglu United States
Karissa Arthur United States
Timo Eninger Germany
Rosie Freer United Kingdom
Ito Kawakami
Citations per year, relative to Ito Kawakami Ito Kawakami (= 1×) peers Rajka M. Liščić

Countries citing papers authored by Ito Kawakami

Since Specialization
Citations

This map shows the geographic impact of Ito Kawakami's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ito Kawakami with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ito Kawakami more than expected).

Fields of papers citing papers by Ito Kawakami

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ito Kawakami. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ito Kawakami. The network helps show where Ito Kawakami may publish in the future.

Co-authorship network of co-authors of Ito Kawakami

This figure shows the co-authorship network connecting the top 25 collaborators of Ito Kawakami. A scholar is included among the top collaborators of Ito Kawakami based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ito Kawakami. Ito Kawakami is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kawakami, Ito, et al.. (2025). Increased granulovacuolar degeneration in the thalamus and higher neurofibrillary tangle Braak stages in bipolar disorder. Psychiatry and Clinical Neurosciences. 79(12). 801–807.
2.
Arseni, Diana, Takashi Nonaka, Max Jacobsen, et al.. (2024). Heteromeric amyloid filaments of ANXA11 and TDP-43 in FTLD-TDP type C. Nature. 634(8034). 662–668. 38 indexed citations
3.
Ishida, Hiroaki, Mitsuhiro Miyashita, Kenichi Oshima, et al.. (2022). Carbonyl stress-sensitive brain regions in the patient with treatment-resistant schizophrenia with a glyoxalase 1 frameshift mutation: Autopsy study. 1(2). 100064–100064. 1 indexed citations
4.
Matsubara, Tomoyasu, Zen‐ichi Tanei, Ito Kawakami, et al.. (2022). Morphological study of the phrenic nerve to determine a reference value for the myelinated fiber density in elderly individuals. Neuropathology. 43(2). 129–134. 2 indexed citations
5.
Kawakami, Ito, Shuji Iritani, Yuichi Riku, et al.. (2022). Neuropathological investigation of patients with prolonged anorexia nervosa. Psychiatry and Clinical Neurosciences. 76(5). 187–194. 6 indexed citations
6.
Matsubara, Tomoyasu, Zen‐ichi Tanei, Ito Kawakami, et al.. (2021). Lewy pathology of the submandibular gland in Lewy body disease: A report of autopsy cases. Neuropathology. 41(6). 476–483. 8 indexed citations
7.
Watanabe, Ryohei, Ito Kawakami, Takeshi Ikeuchi, et al.. (2021). An autopsied FTDP-17 case with MAPT IVS 10 + 14C > T mutation presenting with frontotemporal dementia. eNeurologicalSci. 24. 100363–100363. 1 indexed citations
8.
Watanabe, Ryohei, Shinji Higashi, Takashi Nonaka, et al.. (2020). Intracellular dynamics of Ataxin-2 in the human brains with normal and frontotemporal lobar degeneration with TDP-43 inclusions. Acta Neuropathologica Communications. 8(1). 176–176. 16 indexed citations
9.
Tanei, Zen‐ichi, Yuko Saito, Shinji Ito, et al.. (2020). Lewy pathology of the esophagus correlates with the progression of Lewy body disease: a Japanese cohort study of autopsy cases. Acta Neuropathologica. 141(1). 25–37. 51 indexed citations
10.
Torii, Youta, Shuji Iritani, Hirotaka Sekiguchi, et al.. (2020). The accumulation of advanced glycation end-products in a schizophrenic patient with a glyoxalase 1 frameshift mutation: An autopsy study. Schizophrenia Research. 223. 356–358. 2 indexed citations
11.
Kametani, Fuyuki, Mari Yoshida, Tomoyasu Matsubara, et al.. (2020). Comparison of Common and Disease-Specific Post-translational Modifications of Pathological Tau Associated With a Wide Range of Tauopathies. Frontiers in Neuroscience. 14. 581936–581936. 52 indexed citations
12.
Ishigaki, Shinsuke, Yuichi Riku, Yusuke Fujioka, et al.. (2020). Aberrant interaction between FUS and SFPQ in neurons in a wide range of FTLD spectrum diseases. Brain. 143(8). 2398–2405. 22 indexed citations
13.
Kawakami, Ito, Tetsuaki Arai, & Masato Hasegawa. (2019). The basis of clinicopathological heterogeneity in TDP-43 proteinopathy. Acta Neuropathologica. 138(5). 751–770. 76 indexed citations
14.
Kawakami, Ito, Katsushige Watanabe, Kenichi Oshima, et al.. (2019). Tau progression in single severe frontal traumatic brain injury in human brains. Journal of the Neurological Sciences. 407. 116495–116495. 13 indexed citations
15.
Watanabe, Ryohei, Ito Kawakami, Mitsumoto Onaya, et al.. (2017). Frontotemporal dementia with trans‐activation response DNA‐binding protein 43 presenting with catatonic syndrome. Neuropathology. 38(3). 281–287. 10 indexed citations
16.
Kawakami, Ito, Zen Kobayashi, Tetsuaki Arai, et al.. (2016). Chorea as a clinical feature of the basophilic inclusion body disease subtype of fused-in-sarcoma-associated frontotemporal lobar degeneration. Acta Neuropathologica Communications. 4(1). 36–36. 6 indexed citations
17.
Fujishiro, Hiroshige, Ito Kawakami, Kenichi Oshima, Kazuhiro Niizato, & Shuji Iritani. (2016). Delirium prior to dementia as a clinical phenotype of Lewy body disease: an autopsied case report. International Psychogeriatrics. 29(4). 687–689. 10 indexed citations
18.
Kawakami, Ito, Eizo Iseki, Koji Kasanuki, et al.. (2016). A family with hereditary diffuse leukoencephalopathy with spheroids caused by a novel c.2442 + 2T > C mutation in the CSF1R gene. Journal of the Neurological Sciences. 367. 349–355. 14 indexed citations
19.
Kawakami, Ito, Masato Hasegawa, Tetsuaki Arai, et al.. (2014). Tau accumulation in the nucleus accumbens in tangle-predominant dementia. Acta Neuropathologica Communications. 2(1). 40–40. 32 indexed citations
20.
Aoki, Naoya, Shinji Higashi, Ito Kawakami, et al.. (2012). Localization of fused in sarcoma (FUS) protein to the post-synaptic density in the brain. Acta Neuropathologica. 124(3). 383–394. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026