István Fűri

1.1k total citations
18 papers, 779 citations indexed

About

István Fűri is a scholar working on Molecular Biology, Immunology and Epidemiology. According to data from OpenAlex, István Fűri has authored 18 papers receiving a total of 779 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Immunology and 5 papers in Epidemiology. Recurrent topics in István Fűri's work include Immune Response and Inflammation (5 papers), Epigenetics and DNA Methylation (3 papers) and RNA modifications and cancer (3 papers). István Fűri is often cited by papers focused on Immune Response and Inflammation (5 papers), Epigenetics and DNA Methylation (3 papers) and RNA modifications and cancer (3 papers). István Fűri collaborates with scholars based in Hungary, United States and Canada. István Fűri's co-authors include Gyöngyi Szabó, Donna Catalano, Karen Kodys, Patrick Lowe, Arvin Iracheta‐Vellve, Terence N. Bukong, Shashi Bala, Aditya Ambade, Yeonhee Cho and Adeyinka Adejumo and has published in prestigious journals such as PLoS ONE, Hepatology and Journal of Hepatology.

In The Last Decade

István Fűri

18 papers receiving 770 citations

Peers

István Fűri

EPIDE

IMMUN

PFM

Xue‐Yin Pan China

Lingling He China

Georgia Dalagiorgou Greece

Xiaoxiang Mao China

Xiang Lin China

Zhuo-Wei Hu China

Núria Tarrats United Kingdom

Sadatsugu Sakane Japan

Xiaolin Zhang China

Xue‐Yin Pan China

István Fűri

395 ×1.1

233 ×0.9

EPIDE

83 ×0.4

IMMUN

194 ×1.0

101 ×0.6

PFM

Citations per year, relative to István Fűri István Fűri (= 1×) peers Xue‐Yin Pan

Countries citing papers authored by István Fűri

Since Specialization

Citations

This map shows the geographic impact of István Fűri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by István Fűri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites István Fűri more than expected).

Fields of papers citing papers by István Fűri

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by István Fűri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by István Fűri. The network helps show where István Fűri may publish in the future.

Co-authorship network of co-authors of István Fűri

This figure shows the co-authorship network connecting the top 25 collaborators of István Fűri. A scholar is included among the top collaborators of István Fűri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with István Fűri. István Fűri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Galamb, Orsolya, Alexandra Kalmár, Anna Sebestyén, et al.. (2020). Promoter Hypomethylation and Increased Expression of the Long Non-coding RNA LINC00152 Support Colorectal Carcinogenesis. Pathology & Oncology Research. 26(4). 2209–2223. 14 indexed citations

Lowe, Patrick, Caroline Morel, Aditya Ambade, et al.. (2020). Chronic alcohol-induced neuroinflammation involves CCR2/5-dependent peripheral macrophage infiltration and microglia alterations. Journal of Neuroinflammation. 17(1). 296–296. 68 indexed citations

Kiszner, Gergő, Péter Balla, Gábor Barna, et al.. (2019). Exploring Differential Connexin Expression across Melanocytic Tumor Progression Involving the Tumor Microenvironment. Cancers. 11(2). 165–165. 11 indexed citations

Babuta, Mrigya, István Fűri, Shashi Bala, et al.. (2019). Dysregulated Autophagy and Lysosome Function Are Linked to Exosome Production by Micro‐RNA 155 in Alcoholic Liver Disease. Hepatology. 70(6). 2123–2141. 167 indexed citations

Bukong, Terence N., Yeonhee Cho, Arvin Iracheta‐Vellve, et al.. (2018). Abnormal neutrophil traps and impaired efferocytosis contribute to liver injury and sepsis severity after binge alcohol use. Journal of Hepatology. 69(5). 1145–1154. 136 indexed citations

Tornai, Dávid, István Fűri, Zu T. Shen, et al.. (2018). Inhibition of Triggering Receptor Expressed on Myeloid Cells 1 Ameliorates Inflammation and Macrophage and Neutrophil Activation in Alcoholic Liver Disease in Mice. Hepatology Communications. 3(1). 99–115. 23 indexed citations

Bala, Shashi, Tímea Csák, Karen Kodys, et al.. (2017). Alcohol-induced miR-155 and HDAC11 inhibit negative regulators of the TLR4 pathway and lead to increased LPS responsiveness of Kupffer cells in alcoholic liver disease. Journal of Leukocyte Biology. 102(2). 487–498. 74 indexed citations

Saha, Banishree, Fatemeh Momen‐Heravi, István Fűri, et al.. (2017). Extracellular vesicles from mice with alcoholic liver disease carry a distinct protein cargo and induce macrophage activation through heat shock protein 90. Hepatology. 67(5). 1986–2000. 104 indexed citations

Fűri, István, Alexandra Kalmár, Barnabás Wichmann, et al.. (2015). Cell Free DNA of Tumor Origin Induces a ‘Metastatic’ Expression Profile in HT-29 Cancer Cell Line. PLoS ONE. 10(7). e0131699–e0131699. 28 indexed citations

10.

Leiszter, Katalin, Ferenc Sípos, Orsolya Galamb, et al.. (2015). Promoter Hypermethylation-Related Reduced Somatostatin Production Promotes Uncontrolled Cell Proliferation in Colorectal Cancer. PLoS ONE. 10(2). e0118332–e0118332. 25 indexed citations

11.

Kalmár, Alexandra, Bálint Péterfia, Barnabás Wichmann, et al.. (2015). Comparison of Automated and Manual DNA Isolation Methods for DNA Methylation Analysis of Biopsy, Fresh Frozen, and Formalin-Fixed, Paraffin-Embedded Colorectal Cancer Samples. SLAS TECHNOLOGY. 20(6). 642–651. 17 indexed citations

12.

Műzes, Györgyi, et al.. (2014). Interaction of Autophagy and Toll-Like Receptors: A Regulatory Cross- Talk - Even in Cancer Cells?. Current Drug Targets. 15(8). 743–752. 9 indexed citations

13.

Műzes, Györgyi, Ferenc Sípos, István Fűri, et al.. (2014). Preconditioning with Intravenous Colitic Cell-Free DNA Prevents DSS-Colitis by Altering TLR9-Associated Gene Expression Profile. Digestive Diseases and Sciences. 59(12). 2935–2946. 9 indexed citations

14.

Sípos, Ferenc, Györgyi Műzes, István Fűri, et al.. (2014). Intravenous Administration of a Single-Dose Free-Circulating DNA of Colitic Origin Improves Severe Murine DSS-Colitis. Pathology & Oncology Research. 20(4). 867–877. 9 indexed citations

15.

Valcz, Gábor, Árpád V. Patai, Alexandra Kalmár, et al.. (2014). Myofibroblast-Derived SFRP1 as Potential Inhibitor of Colorectal Carcinoma Field Effect. PLoS ONE. 9(11). e106143–e106143. 35 indexed citations

16.

Fűri, István. (2013). Epithelial toll-like receptor 9 signaling in colorectal inflammation and cancer: Clinico-pathogenic aspects. World Journal of Gastroenterology. 19(26). 4119–4119. 30 indexed citations

17.

Fűri, István, Ferenc Sípos, Sándor Spisák, et al.. (2013). Association of Self‐DNA Mediated TLR9‐Related Gene, DNA Methyltransferase, and Cytokeratin Protein Expression Alterations in HT29‐Cells to DNA Fragment Length and Methylation Status. The Scientific World JOURNAL. 2013(1). 293296–293296. 12 indexed citations

18.

Sípos, Ferenc, Györgyi Műzes, Árpád V. Patai, et al.. (2013). Genome-Wide Screening for Understanding the Role of DNA Methylation in Colorectal Cancer. Epigenomics. 5(5). 569–581. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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