Péter Hollósi

508 total citations
17 papers, 407 citations indexed

About

Péter Hollósi is a scholar working on Molecular Biology, Cell Biology and Cancer Research. According to data from OpenAlex, Péter Hollósi has authored 17 papers receiving a total of 407 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 5 papers in Cell Biology and 5 papers in Cancer Research. Recurrent topics in Péter Hollósi's work include Epigenetics and DNA Methylation (6 papers), RNA modifications and cancer (5 papers) and Proteoglycans and glycosaminoglycans research (5 papers). Péter Hollósi is often cited by papers focused on Epigenetics and DNA Methylation (6 papers), RNA modifications and cancer (5 papers) and Proteoglycans and glycosaminoglycans research (5 papers). Péter Hollósi collaborates with scholars based in Hungary, Germany and Austria. Péter Hollósi's co-authors include Ilona Kovalszky, Sándor Paku, Kornélia Baghy, Alexandra Fullár, József Dudás, Bálint Péterfia, Katalin Karászi, Gábor Sobel, Árpád V. Patai and Zoltán Papp and has published in prestigious journals such as Gastroenterology, PLoS ONE and Oncotarget.

In The Last Decade

Péter Hollósi

16 papers receiving 402 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Péter Hollósi Hungary 9 221 118 112 106 54 17 407
Carolina Cavalcante Bitu Finland 11 321 1.5× 154 1.3× 65 0.6× 158 1.5× 90 1.7× 14 592
Ana Belén Martínez‐Cruz Spain 12 398 1.8× 128 1.1× 60 0.5× 195 1.8× 18 0.3× 15 519
Rebecca A. Wagenaar-Miller United States 8 253 1.1× 257 2.2× 49 0.4× 212 2.0× 43 0.8× 8 584
Wei-Chieh Huang Taiwan 12 393 1.8× 239 2.0× 54 0.5× 135 1.3× 28 0.5× 22 578
Sumitaka Hagiwara Japan 11 276 1.2× 81 0.7× 31 0.3× 185 1.7× 43 0.8× 21 469
Tomomi Hirosaki Japan 10 144 0.7× 103 0.9× 155 1.4× 53 0.5× 24 0.4× 11 455
Alexandra Fullár Hungary 10 257 1.2× 136 1.2× 153 1.4× 168 1.6× 28 0.5× 15 520
Y Matsumura Japan 10 214 1.0× 70 0.6× 129 1.2× 164 1.5× 95 1.8× 27 438
Shu Abe Japan 3 116 0.5× 166 1.4× 59 0.5× 122 1.2× 63 1.2× 8 409
Chao Zeng China 11 244 1.1× 199 1.7× 29 0.3× 92 0.9× 34 0.6× 23 466

Countries citing papers authored by Péter Hollósi

Since Specialization
Citations

This map shows the geographic impact of Péter Hollósi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Péter Hollósi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Péter Hollósi more than expected).

Fields of papers citing papers by Péter Hollósi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Péter Hollósi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Péter Hollósi. The network helps show where Péter Hollósi may publish in the future.

Co-authorship network of co-authors of Péter Hollósi

This figure shows the co-authorship network connecting the top 25 collaborators of Péter Hollósi. A scholar is included among the top collaborators of Péter Hollósi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Péter Hollósi. Péter Hollósi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Galamb, Orsolya, Alexandra Kalmár, Anna Sebestyén, et al.. (2020). Promoter Hypomethylation and Increased Expression of the Long Non-coding RNA LINC00152 Support Colorectal Carcinogenesis. Pathology & Oncology Research. 26(4). 2209–2223. 14 indexed citations
2.
Hollósi, Péter, Katalin Karászi, Bálint Péterfia, et al.. (2020). Syndecan-1 Promotes Hepatocyte-Like Differentiation of Hepatoma Cells Targeting Ets-1 and AP-1. Biomolecules. 10(10). 1356–1356. 6 indexed citations
3.
Fullár, Alexandra, Katalin Karászi, Péter Hollósi, et al.. (2020). Two ways of epigenetic silencing of TFPI2 in cervical cancer. PLoS ONE. 15(6). e0234873–e0234873. 13 indexed citations
4.
Péterfia, Bálint, Péter Hollósi, Irén Haltrich, et al.. (2018). Elsődleges genetikai vizsgálat Prader–Willi-szindróma igazolására. Orvosi Hetilap. 159(2). 64–69. 1 indexed citations
5.
Péterfia, Bálint, Alexandra Kalmár, Árpád V. Patai, et al.. (2017). Construction of a multiplex mutation hot spot PCR panel: the first step towards colorectal cancer genotyping on the GS Junior platform. Journal of Cancer. 8(2). 162–173. 5 indexed citations
6.
Módos, Orsolya, Henning Reis, Christian Niedworok, et al.. (2016). Mutations of KRAS, NRAS, BRAF, EGFR, and PIK3CA genes in urachal carcinoma: Occurence and prognostic significance. Oncotarget. 7(26). 39293–39301. 35 indexed citations
7.
Patai, Árpád V., Árpád V. Patai, Gábor Valcz, et al.. (2015). Comprehensive DNA Methylation Analysis Reveals a Common Ten-Gene Methylation Signature in Colorectal Adenomas and Carcinomas. PLoS ONE. 10(8). e0133836–e0133836. 39 indexed citations
8.
Módos, Orsolya, Tibor Szarvas, Henning Reis, et al.. (2015). Mutation analysis of EGFR signal transduction pathway in urachal carcinoma. European Urology Supplements. 14(6). e1215–e1215. 3 indexed citations
9.
Kalmár, Alexandra, Bálint Péterfia, Péter Hollósi, et al.. (2015). DNA hypermethylation and decreased mRNA expression of MAL, PRIMA1, PTGDR and SFRP1 in colorectal adenoma and cancer. BMC Cancer. 15(1). 736–736. 53 indexed citations
10.
Fullár, Alexandra, József Dudás, Péter Hollósi, et al.. (2015). Remodeling of extracellular matrix by normal and tumor-associated fibroblasts promotes cervical cancer progression. BMC Cancer. 15(1). 256–256. 111 indexed citations
11.
Kalmár, Alexandra, Bálint Péterfia, Péter Hollósi, et al.. (2015). Bisulfite-Based DNA Methylation Analysis from Recent and Archived Formalin-Fixed, Paraffin Embedded Colorectal Tissue Samples. Pathology & Oncology Research. 21(4). 1149–1156. 9 indexed citations
12.
Kalmár, Alexandra, Bálint Péterfia, Péter Hollósi, et al.. (2013). Su1998 Gene Expression-Based High-Throughput Screening Reveals COL12, PTGDR, SFRP2 and SOCS3 As Potential Novel Methylation Markers of Left-Sided Colorectal Cancer. Gastroenterology. 144(5). S–527.
13.
Sípos, Ferenc, Györgyi Műzes, Árpád V. Patai, et al.. (2013). Genome-Wide Screening for Understanding the Role of DNA Methylation in Colorectal Cancer. Epigenomics. 5(5). 569–581. 8 indexed citations
14.
Péterfia, Bálint, Tibor Füle, Kornélia Baghy, et al.. (2012). Syndecan-1 Enhances Proliferation, Migration and Metastasis of HT-1080 Cells in Cooperation with Syndecan-2. PLoS ONE. 7(6). e39474–e39474. 34 indexed citations
15.
Fthenou, Eleni, Péter Hollósi, Ilona Kovalszky, et al.. (2009). Syndecan-1 and FGF-2, but Not FGF Receptor-1, Share a Common Transport Route and Co-Localize with Heparanase in the Nuclei of Mesenchymal Tumor Cells. PLoS ONE. 4(10). e7346–e7346. 64 indexed citations
16.
Péterfia, Bálint, Péter Hollósi, László Szilák, et al.. (2006). [Role of syndecan-1 proteoglycan in the invasiveness of HT-1080 fibrosarcoma].. PubMed. 50(2). 115–20. 8 indexed citations
17.
Kovalszky, Ilona, József Dudás, Mónika Gallai, et al.. (2004). [Proteoglycans in the liver].. PubMed. 48(3). 207–13. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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