Iolanda Lázaro
Impact in
-
- Diet, Metabolism, and Disease
- Biochemistry top 5%
Papers in
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- Fatty Acid Research and Health 11
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- Diet, Metabolism, and Disease 7
- Co-authors
- L. MasanaAnna CabréCarmen Gómez‐GuerreroJesús EgidoJosefa GironaNúria PlanaAinhoa OguizaCarlota Recio
- Journals
- Atherosclerosis (8 papers)Cardiovascular Diabetology (3 papers)Nutrients (3 papers)Journal of Lipid Research (3 papers)Journal of Nutrition (2 papers)
- Partner nations
- SpainUnited StatesChile
In The Last Decade
Iolanda Lázaro
55 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 114
- Endocrinology, Diabetes and Metabolism 272
- Biochemistry 92
- Nephrology 87
- Molecular Biology 749
- Immunology 212
Countries citing papers authored by Iolanda Lázaro
This map shows the geographic impact of Iolanda Lázaro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Iolanda Lázaro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Iolanda Lázaro more than expected).
Fields of papers citing papers by Iolanda Lázaro
This network shows the impact of papers produced by Iolanda Lázaro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Iolanda Lázaro. The network helps show where Iolanda Lázaro may publish in the future.
Co-authors
The 25 scholars most cited alongside Iolanda Lázaro, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2025 | 0 | |
| 3 | 2025 | 0 | |
| 4 | 2024 | 0 | |
| 5 | 2024 | 1 | |
| 6 | 2023 | 7 | |
| 7 | 2023 | 1 | |
| 8 | 2023 | 3 | |
| 9 | 2022 | 16 | |
| 10 | 2020 | 34 | |
| 11 | 2020 | 17 | |
| 12 | 2019 | 39 | |
| 13 | 2016 | 49 | |
| 14 | 2015 | 37 | |
| 15 | 2015 | 39 | |
| 16 | 2013 | 15 | |
| 17 | 2011 | 11 | |
| 18 | 2009 | 8 | |
| 19 | 2009 | 34 | |
| 20 | 2007 | 130 |
About Iolanda Lázaro
Iolanda Lázaro is a scholar working on Nutrition and Dietetics, Endocrinology, Diabetes and Metabolism, Biochemistry, Epidemiology and Cardiology and Cardiovascular Medicine, having authored 58 papers that have together received 1.5k indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (14 papers), Fatty Acid Research and Health (11 papers), Liver Disease Diagnosis and Treatment (10 papers), Adipokines, Inflammation, and Metabolic Diseases (9 papers), Lipid metabolism and disorders (7 papers), Diet, Metabolism, and Disease (7 papers), Cytokine Signaling Pathways and Interactions (5 papers) and Atherosclerosis and Cardiovascular Diseases (5 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (272 citations), Biochemistry (92 citations), Nephrology (87 citations), Molecular Biology (749 citations) and Immunology (212 citations). Iolanda Lázaro has collaborated with scholars based in Spain, United States and Chile. Frequent co-authors include L. Masana, Anna Cabré, Carmen Gómez‐Guerrero, Jesús Egido, Josefa Girona, Núria Plana, Ainhoa Oguiza, Carlota Recio, Mercedes Heras and Francesc Marimón. Their work appears in journals such as Atherosclerosis, Cardiovascular Diabetology, Nutrients, Journal of Lipid Research and Journal of Nutrition.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.