Ikuyo Kou

4.6k total citations
25 papers, 1.8k citations indexed

About

Ikuyo Kou is a scholar working on Molecular Biology, Rheumatology and Genetics. According to data from OpenAlex, Ikuyo Kou has authored 25 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 8 papers in Rheumatology and 8 papers in Genetics. Recurrent topics in Ikuyo Kou's work include Osteoarthritis Treatment and Mechanisms (7 papers), TGF-β signaling in diseases (5 papers) and Genetic factors in colorectal cancer (4 papers). Ikuyo Kou is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (7 papers), TGF-β signaling in diseases (5 papers) and Genetic factors in colorectal cancer (4 papers). Ikuyo Kou collaborates with scholars based in Japan, Italy and United Kingdom. Ikuyo Kou's co-authors include Shiro Ikegawa, Masahiro Nakajima, Akihiko Mabuchi, Kozo Nakamura, Hideki Kizawa, Satoru Kamekura, Toshiyuki Ikeda, Shoji Seki, Hiroshi Kawaguchi and Tsuyoshi Takato and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Nature Genetics.

In The Last Decade

Ikuyo Kou

25 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ikuyo Kou Japan 19 874 714 391 383 272 25 1.8k
Yangli Xie China 25 1.1k 1.3× 468 0.7× 342 0.9× 257 0.7× 196 0.7× 53 2.0k
Motomi Enomoto‐Iwamoto United States 18 786 0.9× 1.2k 1.7× 290 0.7× 332 0.9× 162 0.6× 21 1.9k
Ken‐ichi Katsube Japan 28 1.4k 1.6× 467 0.7× 194 0.5× 454 1.2× 305 1.1× 68 2.4k
Yoshinari Miyamoto Japan 16 970 1.1× 787 1.1× 351 0.9× 357 0.9× 101 0.4× 25 2.2k
Kaneyuki Tsuchimochi United States 15 792 0.9× 1.0k 1.5× 157 0.4× 268 0.7× 356 1.3× 17 1.9k
Masahiro Iwamoto Japan 27 1.9k 2.2× 1.1k 1.6× 584 1.5× 247 0.6× 350 1.3× 57 2.9k
David Pfander Germany 21 585 0.7× 757 1.1× 167 0.4× 293 0.8× 125 0.5× 34 1.6k
Tatsuya Furuichi Japan 23 2.1k 2.4× 695 1.0× 482 1.2× 282 0.7× 443 1.6× 40 3.2k
Qiuqian Wu United States 13 747 0.9× 855 1.2× 170 0.4× 176 0.5× 137 0.5× 15 1.4k
Jennifer H. Jonason United States 23 803 0.9× 439 0.6× 140 0.4× 250 0.7× 111 0.4× 38 1.5k

Countries citing papers authored by Ikuyo Kou

Since Specialization
Citations

This map shows the geographic impact of Ikuyo Kou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ikuyo Kou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ikuyo Kou more than expected).

Fields of papers citing papers by Ikuyo Kou

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ikuyo Kou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ikuyo Kou. The network helps show where Ikuyo Kou may publish in the future.

Co-authorship network of co-authors of Ikuyo Kou

This figure shows the co-authorship network connecting the top 25 collaborators of Ikuyo Kou. A scholar is included among the top collaborators of Ikuyo Kou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ikuyo Kou. Ikuyo Kou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Takeda, Kazuki, Ikuyo Kou, Noriaki Kawakami, et al.. (2017). Compound Heterozygosity for Null Mutations and a Common Hypomorphic Risk Haplotype inTBX6Causes Congenital Scoliosis. Human Mutation. 38(3). 317–323. 30 indexed citations
2.
Einarsdóttir, Elísabet, Anna Grauers, Hong Jiao, et al.. (2017). CELSR2 is a candidate susceptibility gene in idiopathic scoliosis. PLoS ONE. 12(12). e0189591–e0189591. 15 indexed citations
3.
4.
Nakajima, Masahiro, Ikuyo Kou, Hirofumi Ohashi, & Shiro Ikegawa. (2016). Identification and Functional Characterization of RSPO2 as a Susceptibility Gene for Ossification of the Posterior Longitudinal Ligament of the Spine. The American Journal of Human Genetics. 99(1). 202–207. 37 indexed citations
5.
Ogura, Yoji, Shunsuke Fujibayashi, Aritoshi Iida, et al.. (2015). A novel FOXC2 mutation in spinal extradural arachnoid cyst. Human Genome Variation. 2(1). 15032–15032. 8 indexed citations
6.
Ogura, Yoji, Noriko Miyake, Ikuyo Kou, et al.. (2015). Identification of HOXD4 Mutations in Spinal Extradural Arachnoid Cyst. PLoS ONE. 10(11). e0142126–e0142126. 14 indexed citations
7.
Pagnamenta, Alistair T., Moira Blyth, Sahar Mansour, et al.. (2015). Deletions and de novo mutations of SOX11 are associated with a neurodevelopmental disorder with features of Coffin–Siris syndrome. Journal of Medical Genetics. 53(3). 152–162. 53 indexed citations
8.
Ogura, Yoji, Shoji Yabuki, Aritoshi Iida, et al.. (2013). FOXC2 Mutations in Familial and Sporadic Spinal Extradural Arachnoid Cyst. PLoS ONE. 8(11). e80548–e80548. 21 indexed citations
9.
Fan, Yanhui, You‐Qiang Song, Danny Chan, et al.. (2012). SNP rs11190870 near LBX1 is associated with adolescent idiopathic scoliosis in southern Chinese. Journal of Human Genetics. 57(4). 244–246. 48 indexed citations
10.
Kou, Ikuyo, Atsushi Takahashi, Tomohiko Urano, et al.. (2011). Common Variants in a Novel Gene, FONG on Chromosome 2q33.1 Confer Risk of Osteoporosis in Japanese. PLoS ONE. 6(5). e19641–e19641. 31 indexed citations
11.
Duval, Elise, Nicolas Bigot, Magalie Hervieu, et al.. (2011). Asporin Expression Is Highly Regulated in Human Chondrocytes. Molecular Medicine. 17(7-8). 816–823. 36 indexed citations
12.
Kou, Ikuyo, Masahiro Nakajima, & Shiro Ikegawa. (2010). Binding characteristics of the osteoarthritis-associated protein asporin. Journal of Bone and Mineral Metabolism. 28(4). 395–402. 32 indexed citations
13.
Nakajima, Masahiro, Atsushi Takahashi, Ikuyo Kou, et al.. (2010). New Sequence Variants in HLA Class II/III Region Associated with Susceptibility to Knee Osteoarthritis Identified by Genome-Wide Association Study. PLoS ONE. 5(3). e9723–e9723. 80 indexed citations
14.
Murakami, Tomohiko, Atsushi Saito, Shin‐ichiro Hino, et al.. (2009). Signalling mediated by the endoplasmic reticulum stress transducer OASIS is involved in bone formation. Nature Cell Biology. 11(10). 1205–1211. 312 indexed citations
15.
Cheung, Ching‐Lung, Vivian Chan, Shiro Ikegawa, et al.. (2008). Pre-B-cell leukemia homeobox 1 (PBX1) shows functional and possible genetic association with bone mineral density variation. Human Molecular Genetics. 18(4). 679–687. 26 indexed citations
16.
Mori, Seijiro, Ikuyo Kou, Hidenori Sato, et al.. (2008). Association of genetic variations of genes encoding thrombospondin, type 1, domain-containing 4 and 7A with low bone mineral density in Japanese women with osteoporosis. Journal of Human Genetics. 53(8). 694–697. 19 indexed citations
17.
Kou, Ikuyo, Masahiro Nakajima, & Shiro Ikegawa. (2007). Expression and Regulation of the Osteoarthritis-associated Protein Asporin. Journal of Biological Chemistry. 282(44). 32193–32199. 45 indexed citations
18.
Nakajima, Masahiro, Hideki Kizawa, Masao Saitoh, et al.. (2007). Mechanisms for Asporin Function and Regulation in Articular Cartilage. Journal of Biological Chemistry. 282(44). 32185–32192. 131 indexed citations
19.
Kizawa, Hideki, Ikuyo Kou, Aritoshi Iida, et al.. (2005). An aspartic acid repeat polymorphism in asporin inhibits chondrogenesis and increases susceptibility to osteoarthritis. Nature Genetics. 37(2). 138–144. 353 indexed citations
20.
Ikeda, Toshiyuki, Satoru Kamekura, Akihiko Mabuchi, et al.. (2004). The combination of SOX5, SOX6, and SOX9 (the SOX trio) provides signals sufficient for induction of permanent cartilage. Arthritis & Rheumatism. 50(11). 3561–3573. 295 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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