Hung‐Ying Kao
- Molecular Biology top 1%
- Genetics top 1%
- Oncology top 5%
- Immunology top 5%
- Cellular and Molecular Neuroscience top 5%
- Co-authors
- Ronald M. EvansPeter OrdentlichMichael DownesDebabrata ChakravartiLászló NagyRichard J. LinStuart L. SchreiberDonald E. Ayer
- Topics
- Retinoids in leukemia and cellular processes (28 papers)Histone Deacetylase Inhibitors Research (21 papers)Estrogen and related hormone effects (16 papers)
- Cited by
- Molecular BiologyGeneticsImmunology
- Partner nations
- United StatesTaiwanFrance
In The Last Decade
Hung‐Ying Kao
78 papers receiving 6.3k citations
Hit Papers
Peers
Comparison fields: 5 of 122
- Molecular Biology 5.3k
- Genetics 1.5k
- Oncology 954
- Immunology 740
- Cellular and Molecular Neuroscience 595
Countries citing papers authored by Hung‐Ying Kao
This map shows the geographic impact of Hung‐Ying Kao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hung‐Ying Kao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hung‐Ying Kao more than expected).
Fields of papers citing papers by Hung‐Ying Kao
This network shows the impact of papers produced by Hung‐Ying Kao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hung‐Ying Kao. The network helps show where Hung‐Ying Kao may publish in the future.
Co-authorship network of co-authors of Hung‐Ying Kao
This figure shows the co-authorship network connecting the top 25 collaborators of Hung‐Ying Kao. A scholar is included among the top collaborators of Hung‐Ying Kao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hung‐Ying Kao. Hung‐Ying Kao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 12 | |
| 3 | 5 | |
| 4 | 1 | |
| 5 | 14 | |
| 6 | 45 | |
| 7 | 22 | |
| 8 | 22 | |
| 9 | 21 | |
| 10 | 53 | |
| 11 | 29 | |
| 12 | 13 | |
| 13 | 38 | |
| 14 | 32 | |
| 15 | 22 | |
| 16 | 112 | |
| 17 | 266 | |
| 18 | 177 | |
| 19 | 304 | |
| 20 | Nuclear Receptor Repression Mediated by a Complex Containing SMRT, mSin3A, and Histone Deacetylasebreakdown → | 1070 |
About Hung‐Ying Kao
Hung‐Ying Kao is a scholar working on Molecular Biology, Nephrology and Biochemistry, having authored 78 papers that have together received 6.3k indexed citations. Recurring topics across this work include Retinoids in leukemia and cellular processes (28 papers), Histone Deacetylase Inhibitors Research (21 papers) and Estrogen and related hormone effects (16 papers). The work is most often cited by research in Molecular Biology (5.3k citations), Genetics (1.5k citations) and Immunology (740 citations). Hung‐Ying Kao has collaborated with scholars based in United States, Taiwan and France. Frequent co-authors include Ronald M. Evans, Peter Ordentlich, Michael Downes, Debabrata Chakravarti, László Nagy, Richard J. Lin, Stuart L. Schreiber, Donald E. Ayer, Christian A. Hassig and Yu Liu. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.