Hsiang-Lin Chang
Impact in
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- Research in Cotton Cultivation
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- Cell death mechanisms and regulation
- Protein Tyrosine Phosphatases
- Fibroblast Growth Factor Research
Papers in
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- Protein Tyrosine Phosphatases 3
- Fibroblast Growth Factor Research 2
- Cell death mechanisms and regulation 1
- Retinoids in leukemia and cellular processes 1
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- Research in Cotton Cultivation 6
- Co-authors
- Young C. Lin (17 shared papers)Y. Sugimoto (16 shared papers)Suling Liu (9 shared papers)Yi‐Wen Huang (11 shared papers)Weiping Ye (11 shared papers)Li‐Shu Wang (11 shared papers)Jiahua Jiang (6 shared papers)Michael K. Dowd (5 shared papers)
- Journals
- Anticancer Research (1 paper)Breast Cancer Research and Treatment (1 paper)Cancer Research (1 paper)The Journal of Steroid Biochemistry and Molecular Biology (1 paper)PubMed (13 papers)
- Partner nations
- United States
In The Last Decade
Hsiang-Lin Chang
17 papers receiving 417 citations
Peers
Comparison fields: 5 of 65
- Plant Science 137
- Molecular Biology 215
- Genetics 31
- Horticulture 3
- Cancer Research 42
Countries citing papers authored by Hsiang-Lin Chang
This map shows the geographic impact of Hsiang-Lin Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hsiang-Lin Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hsiang-Lin Chang more than expected).
Fields of papers citing papers by Hsiang-Lin Chang
This network shows the impact of papers produced by Hsiang-Lin Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hsiang-Lin Chang. The network helps show where Hsiang-Lin Chang may publish in the future.
Co-authors
The 16 scholars most cited alongside Hsiang-Lin Chang, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | The (-)-enantiomer of gossypol possesses higher anticancer potency than racemic gossypol in human breast cancer. | 2002 | 82 |
| 2 | Molecular mechanisms of (-)-gossypol-induced apoptosis in human prostate cancer cells. | 2006 | 57 |
| 3 | The inhibitory effects of gossypol on human prostate cancer cells-PC3 are associated with transforming growth factor beta1 (TGFbeta1) signal transduction pathway. | 2004 | 52 |
| 4 | Modulation of multidrug resistance gene expression in human breast cancer cells by (-)-gossypol-enriched cottonseed oil. | 2007 | 27 |
| 5 | Function and regulatory mechanisms of the candidate tumor suppressor receptor protein tyrosine phosphatase gamma (PTPRG) in breast cancer cells. | 2010 | 27 |
| 6 | Estrogenic down-regulation of protein tyrosine phosphatase gamma (PTP gamma) in human breast is associated with estrogen receptor alpha. | 2003 | 23 |
| 7 | Keratinocyte growth factor (KGF) regulates estrogen receptor-alpha (ER-alpha) expression and cell apoptosis via phosphatidylinositol 3-kinase (PI3K)/Akt pathway in human breast cancer cells. | 2009 | 21 |
| 8 | Induction of apoptosis by (-)-gossypol-enriched cottonseed oil in human breast cancer cells. | 2010 | 21 |
| 9 | Conjugated linoleic acid (CLA) up-regulates the estrogen-regulated cancer suppressor gene, protein tyrosine phosphatase gamma (PTPgama), in human breast cells. | 2006 | 18 |
| 10 | Effects of serum on (-)-gossypol-suppressed growth in human prostate cancer cells. | 2006 | 18 |
| 11 | Keratinocyte growth factor (KGF) induces tamoxifen (Tam) resistance in human breast cancer MCF-7 cells. | 2006 | 18 |
| 12 | 2002 | 18 | |
| 13 | 2006 | 16 | |
| 14 | Effects of human breast stromal cells on conjugated linoleic acid (CLA) modulated vascular endothelial growth factor-A (VEGF-A) expression in MCF-7 cells. | 2006 | 14 |
| 15 | Effects of age on growth and ERbeta mRNA expression of canine prostatic cells. | 2006 | 11 |
| 16 | Effects of gossypol on O2 consumption and CO2 production in human prostate cancer cells. | 2002 | 5 |
| 17 | Anti-angiogenesis effects of conjugated linoleic acid in human breast cancer cell line MCF-7 | 2004 | 2 |
About Hsiang-Lin Chang
Hsiang-Lin Chang is a scholar working on Molecular Biology, Plant Science, Pulmonary and Respiratory Medicine, Oncology and Nutrition and Dietetics, having authored 17 papers that have together received 430 indexed citations. Recurring topics across this work include Research in Cotton Cultivation (6 papers), Protein Tyrosine Phosphatases (3 papers), Fibroblast Growth Factor Research (2 papers), Prostate Cancer Treatment and Research (1 paper), Fatty Acid Research and Health (1 paper), Cell death mechanisms and regulation (1 paper), Retinoids in leukemia and cellular processes (1 paper) and Cancer, Lipids, and Metabolism (1 paper). The work is most often cited by research in Plant Science (137 citations), Molecular Biology (215 citations), Genetics (31 citations), Horticulture (3 citations) and Cancer Research (42 citations). Hsiang-Lin Chang has collaborated with scholars based in United States. Frequent co-authors include Young C. Lin, Y. Sugimoto, Suling Liu, Yi‐Wen Huang, Weiping Ye, Li‐Shu Wang, Jiahua Jiang, Michael K. Dowd, Peter J. Wan and Samuel K. Kulp. Their work appears in journals such as Anticancer Research, Breast Cancer Research and Treatment, Cancer Research, The Journal of Steroid Biochemistry and Molecular Biology and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.