Hong‐Hee Kim

6.7k total citations
126 papers, 5.2k citations indexed

About

Hong‐Hee Kim is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Hong‐Hee Kim has authored 126 papers receiving a total of 5.2k indexed citations (citations by other indexed papers that have themselves been cited), including 104 papers in Molecular Biology, 59 papers in Oncology and 34 papers in Cancer Research. Recurrent topics in Hong‐Hee Kim's work include Bone Metabolism and Diseases (80 papers), Bone health and treatments (42 papers) and NF-κB Signaling Pathways (31 papers). Hong‐Hee Kim is often cited by papers focused on Bone Metabolism and Diseases (80 papers), Bone health and treatments (42 papers) and NF-κB Signaling Pathways (31 papers). Hong‐Hee Kim collaborates with scholars based in South Korea, United States and Puerto Rico. Hong‐Hee Kim's co-authors include Zang Hee Lee, Youngkyun Lee, Hyung Joon Kim, Han Bok Kwak, Hyunil Ha, Hao Huang, Eun-Ju Chang, Jeongim Ha, Hyun‐Man Kim and Eun‐Ju Chang and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and Nature Communications.

In The Last Decade

Hong‐Hee Kim

124 papers receiving 5.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hong‐Hee Kim South Korea 41 3.6k 1.8k 896 840 538 126 5.2k
Jaerang Rho South Korea 29 3.7k 1.0× 2.0k 1.1× 962 1.1× 1.1k 1.3× 596 1.1× 74 5.3k
Zang Hee Lee South Korea 39 3.2k 0.9× 1.5k 0.8× 821 0.9× 641 0.8× 465 0.9× 102 4.7k
Xu Feng United States 33 4.0k 1.1× 2.1k 1.2× 835 0.9× 802 1.0× 943 1.8× 80 5.9k
Masamichi Takami Japan 33 3.4k 1.0× 2.0k 1.1× 820 0.9× 879 1.0× 647 1.2× 110 4.9k
Soo Young Lee South Korea 29 3.6k 1.0× 2.0k 1.1× 1.0k 1.1× 775 0.9× 682 1.3× 68 5.1k
Haibo Zhao United States 39 2.9k 0.8× 1.3k 0.7× 654 0.7× 458 0.5× 563 1.0× 94 4.4k
Masaki Inada Japan 31 2.1k 0.6× 1.3k 0.7× 1.3k 1.4× 423 0.5× 456 0.8× 90 4.8k
Wei Zou United States 34 2.5k 0.7× 1.2k 0.7× 625 0.7× 775 0.9× 376 0.7× 77 3.8k
Yoshiyuki Hakeda Japan 43 3.4k 0.9× 2.0k 1.1× 739 0.8× 389 0.5× 735 1.4× 89 5.4k
Kurt Redlich Austria 41 3.6k 1.0× 1.9k 1.0× 910 1.0× 1.4k 1.6× 1.1k 2.0× 91 6.8k

Countries citing papers authored by Hong‐Hee Kim

Since Specialization
Citations

This map shows the geographic impact of Hong‐Hee Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hong‐Hee Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hong‐Hee Kim more than expected).

Fields of papers citing papers by Hong‐Hee Kim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hong‐Hee Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hong‐Hee Kim. The network helps show where Hong‐Hee Kim may publish in the future.

Co-authorship network of co-authors of Hong‐Hee Kim

This figure shows the co-authorship network connecting the top 25 collaborators of Hong‐Hee Kim. A scholar is included among the top collaborators of Hong‐Hee Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hong‐Hee Kim. Hong‐Hee Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kim, Hong‐Hee, et al.. (2024). TMEM175 plays a crucial role in osteoblast differentiation by regulating lysosomal function and autophagy. Molecules and Cells. 47(11). 100127–100127. 2 indexed citations
2.
Kim, Min Kyung, et al.. (2022). PARK2 Induces Osteoclastogenesis through Activation of the NF-κB Pathway. Molecules and Cells. 45(10). 749–760. 10 indexed citations
3.
Song, Min‐Kyoung, et al.. (2021). Effects of the Lysine Methyltransferase Inhibitor AZ505 on Bone Metabolism. Journal of Bone Metabolism. 28(4). 297–305. 2 indexed citations
4.
Kim, Min Kyung, et al.. (2019). Mitofusin 2, a mitochondria-ER tethering protein, facilitates osteoclastogenesis by regulating the calcium-calcineurin-NFATc1 axis. Biochemical and Biophysical Research Communications. 516(1). 202–208. 13 indexed citations
5.
Kim, Bong-Jun, Jong-Ho Lee, Won Jong Jin, et al.. (2018). Trapidil induces osteogenesis by upregulating the signaling of bone morphogenetic proteins. Cellular Signalling. 49. 68–78. 3 indexed citations
6.
Kim, Haemin, et al.. (2016). Tetraspanin 7 regulates sealing zone formation and the bone-resorbing activity of osteoclasts. Biochemical and Biophysical Research Communications. 477(4). 1078–1084. 15 indexed citations
7.
Mah, Su‐Jung, Jiyeon Lee, Haemin Kim, et al.. (2015). Induction of S100A4 in periodontal ligament cells enhances osteoclast formation. Archives of Oral Biology. 60(9). 1215–1221. 14 indexed citations
8.
Song, Min‐Kyoung, Zang Hee Lee, & Hong‐Hee Kim. (2015). Adseverin mediates RANKL-induced osteoclastogenesis by regulating NFATc1. Experimental & Molecular Medicine. 47(12). e199–e199. 12 indexed citations
9.
Park, Cheol‐Kyu, Youngkyun Lee, Kyun Ha Kim, et al.. (2014). Nrf2 is a novel regulator of bone acquisition. Bone. 63. 36–46. 84 indexed citations
10.
11.
12.
Lee, Seung Hun, Beom‐Jun Kim, Hyung Jin Choi, et al.. (2012). (–)-Epigallocathechin-3-Gallate, an AMPK Activator, Decreases Ovariectomy-Induced Bone Loss by Suppression of Bone Resorption. Calcified Tissue International. 90(5). 404–410. 22 indexed citations
13.
Rim, Hong-Kun, et al.. (2011). Alcohol-Fermented Soybean Increases the Expression of Receptor-Interacting Protein 2 and IκB Kinase β in Mouse Peritoneal Macrophages. Journal of Medicinal Food. 14(10). 1181–1189. 2 indexed citations
14.
Ha, Hyunil, Jong-Ho Lee, Ha‐Neui Kim, et al.. (2006). α-Lipoic Acid Inhibits Inflammatory Bone Resorption by Suppressing Prostaglandin E2 Synthesis. The Journal of Immunology. 176(1). 111–117. 75 indexed citations
15.
Lee, Hyeon‐Woo, et al.. (2005). CD137 induces adhesion and cytokine production in human monocytic THP-1 cells. Experimental & Molecular Medicine. 37(2). 78–85. 6 indexed citations
16.
Kwak, Han Bok, Soo Woong Lee, Young‐Ah Kim, et al.. (2004). Inhibition of osteoclast differentiation and bone resorption by a novel lysophosphatidylcholine derivative, SCOH. Biochemical Pharmacology. 67(7). 1239–1248. 26 indexed citations
17.
Lee, Zang Hee & Hong‐Hee Kim. (2003). Signal transduction by receptor activator of nuclear factor kappa B in osteoclasts. Biochemical and Biophysical Research Communications. 305(2). 211–214. 345 indexed citations
18.
Ha, Hyunil, et al.. (2003). Lipid rafts are important for the association of RANK and TRAF6. Experimental & Molecular Medicine. 35(4). 279–284. 32 indexed citations
19.
Chae, Han–Jung, Sang Chul Kim, Soo‐Wan Chae, et al.. (2001). Blockade of the p38 Mitogen-activated Protein Kinase Pathway Inhibits Inducible Nitric Oxide Synthase and Interleukin-6 Expression in MC3T3E-1 Osteoblasts. Pharmacological Research. 43(3). 275–282. 16 indexed citations
20.
Kim, Hong‐Hee. (1999). Characterization of the NF-κB activation induced by TR8, an osteoclastogenic tumor necrosis factor receptor family member. Archives of Pharmacal Research. 22(5). 454–458. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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