Hiromi Koike

1.4k total citations
30 papers, 1.1k citations indexed

About

Hiromi Koike is a scholar working on Hepatology, Epidemiology and Molecular Biology. According to data from OpenAlex, Hiromi Koike has authored 30 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Hepatology, 10 papers in Epidemiology and 7 papers in Molecular Biology. Recurrent topics in Hiromi Koike's work include Hepatitis C virus research (9 papers), Hepatitis B Virus Studies (7 papers) and Liver Disease Diagnosis and Treatment (6 papers). Hiromi Koike is often cited by papers focused on Hepatitis C virus research (9 papers), Hepatitis B Virus Studies (7 papers) and Liver Disease Diagnosis and Treatment (6 papers). Hiromi Koike collaborates with scholars based in Japan, Malaysia and Brazil. Hiromi Koike's co-authors include Ryuichi Morishita, Toshio Ogihara, Kazuaki Chayama, Yasufumi Kaneda, Akihito Tsubota, Isao Koida, Naruya Tomita, Motokuni Aoki, Masahiro Kobayashi and Michie Hashimoto and has published in prestigious journals such as Circulation, Hepatology and The FASEB Journal.

In The Last Decade

Hiromi Koike

29 papers receiving 1.1k citations

Peers

Hiromi Koike
Comparison fields: 5 of 81
  • Hepatology 583
  • Epidemiology 521
  • Molecular Biology 270
  • Infectious Diseases 205
  • Surgery 127
Replace A. Cappon with:
A. Cappon Italy
John Cardone United Kingdom
Kelvin W. Li United States
Toshiyuki Shinji Japan
Robert Tacke United States
Amber Thompson United Kingdom
Hesham Basma United States
Jun Dong Germany
Zhenzhen Pan China
A. Cappon Italy View profile →
Citations per field, relative to Hiromi Koike
Hiromi Koike · 1×
Citations per year, relative to Hiromi Koike
Hiromi Koike · 1×

Countries citing papers authored by Hiromi Koike

Since Specialization
Citations

This map shows the geographic impact of Hiromi Koike's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiromi Koike with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiromi Koike more than expected).

Fields of papers citing papers by Hiromi Koike

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiromi Koike. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiromi Koike. The network helps show where Hiromi Koike may publish in the future.

Co-authorship network of co-authors of Hiromi Koike

This figure shows the co-authorship network connecting the top 25 collaborators of Hiromi Koike. A scholar is included among the top collaborators of Hiromi Koike based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiromi Koike. Hiromi Koike is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 3
2 38
3 2
4 87
5 69
6 26
7 94
8
IFN-γ誘導可能なタンパク質-10(CXCL10)の、Thy1.1の糸球体腎炎における有足細胞を保護する新規な潜在能力
1
9 4
10 11
11 21
12 8
13 16
14 317
15 1
16 4
17 15
18 170
19 6
20 2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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