Hilda H. Slunt

9.9k total citations · 4 hit papers
39 papers, 8.0k citations indexed

About

Hilda H. Slunt is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Hilda H. Slunt has authored 39 papers receiving a total of 8.0k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 24 papers in Physiology and 11 papers in Cellular and Molecular Neuroscience. Recurrent topics in Hilda H. Slunt's work include Alzheimer's disease research and treatments (23 papers), Prion Diseases and Protein Misfolding (11 papers) and Genetic Neurodegenerative Diseases (5 papers). Hilda H. Slunt is often cited by papers focused on Alzheimer's disease research and treatments (23 papers), Prion Diseases and Protein Misfolding (11 papers) and Genetic Neurodegenerative Diseases (5 papers). Hilda H. Slunt collaborates with scholars based in United States, Japan and France. Hilda H. Slunt's co-authors include David Borchelt, Sangram S. Sisodia, Donald L. Price, Neal G. Copeland, Nancy A. Jenkins, Gopal Thinakaran, Michael K. Lee, Joanna L. Jankowsky, Tamara Ratovitski and Sam Gandy and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Hilda H. Slunt

39 papers receiving 7.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In Vivo

1996 1.2k citations David Borchelt, Gopal Thinakaran et al. Neuron profile →
Endoproteolysis of Presenilin 1 and Accumulation of Processed Derivatives In Vivo

1996 863 citations Gopal Thinakaran, David Borchelt et al. Neuron profile →
Co-expression of multiple transgenes in mouse CNS: a comparison of strategies

2001 680 citations Joanna L. Jankowsky, Hilda H. Slunt et al. Biomolecular Engineering profile →
β-amyloid precursor protein-deficient mice show reactive gliosis and decreased locomotor activity

1995 566 citations Hui Zheng, Minghao Jiang et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hilda H. Slunt United States	31	5.5k	3.9k	2.3k	1.5k	1.1k	39	8.0k
Stephen D. Schmidt United States	30	5.6k 1.0×	2.8k 0.7×	1.2k 0.5×	1.3k 0.9×	1.1k 1.0×	38	7.4k
Lisa McConlogue United States	31	6.0k 1.1×	3.6k 0.9×	2.2k 1.0×	1.7k 1.1×	803 0.7×	41	8.2k
Nikolaos K. Robakis United States	51	4.3k 0.8×	4.1k 1.0×	1.6k 0.7×	1.1k 0.7×	1.1k 0.9×	133	7.3k
Steven L. Wagner United States	37	4.4k 0.8×	3.7k 0.9×	1.9k 0.8×	879 0.6×	689 0.6×	78	8.4k
Albert Y. Hung United States	24	4.7k 0.9×	3.6k 0.9×	1.4k 0.6×	1.3k 0.9×	1.0k 0.9×	35	6.8k
Chris Eckman United States	11	6.7k 1.2×	3.0k 0.8×	2.2k 1.0×	1.8k 1.2×	432 0.4×	13	8.0k
Christopher Janus United States	30	4.3k 0.8×	2.4k 0.6×	2.4k 1.0×	1.1k 0.7×	551 0.5×	53	6.9k
Wilma Wasco United States	32	4.2k 0.8×	3.3k 0.8×	1.5k 0.7×	1.1k 0.7×	892 0.8×	59	6.0k
Gopal Thinakaran United States	57	8.9k 1.6×	6.0k 1.5×	2.5k 1.1×	2.2k 1.4×	3.0k 2.6×	121	12.1k
Anne M. Cataldo United States	45	5.2k 0.9×	3.8k 1.0×	1.7k 0.7×	852 0.6×	2.8k 2.5×	66	9.3k

Countries citing papers authored by Hilda H. Slunt

Since Specialization

Citations

This map shows the geographic impact of Hilda H. Slunt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hilda H. Slunt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hilda H. Slunt more than expected).

Fields of papers citing papers by Hilda H. Slunt

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hilda H. Slunt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hilda H. Slunt. The network helps show where Hilda H. Slunt may publish in the future.

Co-authorship network of co-authors of Hilda H. Slunt

This figure shows the co-authorship network connecting the top 25 collaborators of Hilda H. Slunt. A scholar is included among the top collaborators of Hilda H. Slunt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hilda H. Slunt. Hilda H. Slunt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gary, Devin S., et al.. (2007). Investigation of RNA interference to suppress expression of full-length and fragment human huntingtin. NeuroMolecular Medicine. 9(2). 145–155. 3 indexed citations

Jankowsky, Joanna L., Hilda H. Slunt, Victoria Gonzales, et al.. (2005). Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease. PLoS Medicine. 2(12). e355–e355. 177 indexed citations

Wang, Jiou, Guilian Xu, Hilda H. Slunt, et al.. (2005). Coincident thresholds of mutant protein for paralytic disease and protein aggregation caused by restrictively expressed superoxide dismutase cDNA. Neurobiology of Disease. 20(3). 943–952. 91 indexed citations

Jankowsky, Joanna L., Hilda H. Slunt, Victoria Gonzales, et al.. (2004). APP processing and amyloid deposition in mice haplo-insufficient for presenilin 1. Neurobiology of Aging. 25(7). 885–892. 136 indexed citations

Schilling, Gabriele, Alena Savonenko, Alexandra M. Klevytska, et al.. (2004). Nuclear-targeting of mutant huntingtin fragments produces Huntington's disease-like phenotypes in transgenic mice. Human Molecular Genetics. 13(15). 1599–1610. 78 indexed citations

Kratz, John, Duncan Stearns, David L. Huso, et al.. (2002). Expression of stabilized β-catenin in differentiated neurons of transgenic mice does not result in tumor formation. BMC Cancer. 2(1). 33–33. 19 indexed citations

Borchelt, David, Michael K. Lee, Victoria Gonzales, et al.. (2002). Accumulation of proteolytic fragments of mutant presenilin 1 and accelerated amyloid deposition are co-regulated in transgenic mice. Neurobiology of Aging. 23(2). 171–177. 16 indexed citations

Jankowsky, Joanna L., Hilda H. Slunt, Tamara Ratovitski, et al.. (2001). Co-expression of multiple transgenes in mouse CNS: a comparison of strategies. Biomolecular Engineering. 17(6). 157–165. 680 indexed citations breakdown →

Schilling, Gabriele, Jonathan Wood, Kui Duan, et al.. (1999). Nuclear Accumulation of Truncated Atrophin-1 Fragments in a Transgenic Mouse Model of DRPLA. Neuron. 24(1). 275–286. 143 indexed citations

10.

Buxbaum, Joseph D., Gopal Thinakaran, Vassilis E. Koliatsos, et al.. (1998). Alzheimer Amyloid Protein Precursor in the Rat Hippocampus: Transport and Processing through the Perforant Path. Journal of Neuroscience. 18(23). 9629–9637. 212 indexed citations

11.

Thinakaran, Gopal, Tamara Ratovitski, Hilda H. Slunt, et al.. (1997). Evidence That Levels of Presenilins (PS1 and PS2) Are Coordinately Regulated by Competition for Limiting Cellular Factors. Journal of Biological Chemistry. 272(45). 28415–28422. 275 indexed citations

12.

Ratovitski, Tamara, Hilda H. Slunt, Gopal Thinakaran, et al.. (1997). Endoproteolytic Processing and Stabilization of Wild-type and Mutant Presenilin. Journal of Biological Chemistry. 272(39). 24536–24541. 177 indexed citations

13.

Sisodia, Sangram S., Gopal Thinakaran, Hilda H. Slunt, et al.. (1996). Studies on the Metabolism and Biological Function of APLP2a. Annals of the New York Academy of Sciences. 777(1). 77–81. 10 indexed citations

14.

Lee, Michael K., Hilda H. Slunt, Lee J. Martin, et al.. (1996). Expression of Presenilin 1 and 2 (PS1 and PS2) in Human and Murine Tissues. Journal of Neuroscience. 16(23). 7513–7525. 244 indexed citations

15.

Zheng, Hui, Minghao Jiang, Myrna E. Trumbauer, et al.. (1996). Mice Deficient for the Amyloid Precursor Protein Gene. Annals of the New York Academy of Sciences. 777(1). 421–426. 61 indexed citations

16.

Borchelt, David, Gopal Thinakaran, Christopher B. Eckman, et al.. (1996). Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In Vivo. Neuron. 17(5). 1005–1013. 1224 indexed citations breakdown →

17.

Thinakaran, Gopal, Hilda H. Slunt, & Sangram S. Sisodia. (1995). Novel Regulation of Chondroitin Sulfate Glycosaminoglycan Modification of Amyloid Precursor Protein and Its Homologue, APLP2. Journal of Biological Chemistry. 270(28). 16522–16525. 42 indexed citations

18.

Lo, Acy, Gopal Thinakaran, Hilda H. Slunt, & Sangram S. Sisodia. (1995). Metabolism of the Amyloid Precursor-like Protein 2 in MDCK Cells.. Journal of Biological Chemistry. 270(21). 12641–12645. 28 indexed citations

19.

Zheng, Hui, Minghao Jiang, Myrna E. Trumbauer, et al.. (1995). β-amyloid precursor protein-deficient mice show reactive gliosis and decreased locomotor activity. Cell. 81(4). 525–531. 566 indexed citations breakdown →

20.

Lamb, Bruce T., Sangram S. Sisodia, Ann M. Lawler, et al.. (1993). Introduction and expression of the 400 kilobase precursor amyloid protein gene in transgenic mice. Nature Genetics. 5(1). 22–30. 229 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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