Heitaroh Iwata

1.6k total citations
101 papers, 1.3k citations indexed

About

Heitaroh Iwata is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Heitaroh Iwata has authored 101 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 25 papers in Cellular and Molecular Neuroscience and 18 papers in Physiology. Recurrent topics in Heitaroh Iwata's work include Neuroscience and Neuropharmacology Research (20 papers), Aldose Reductase and Taurine (13 papers) and Alcoholism and Thiamine Deficiency (13 papers). Heitaroh Iwata is often cited by papers focused on Neuroscience and Neuropharmacology Research (20 papers), Aldose Reductase and Taurine (13 papers) and Alcoholism and Thiamine Deficiency (13 papers). Heitaroh Iwata collaborates with scholars based in Japan, United States and South Korea. Heitaroh Iwata's co-authors include Akemichi Baba, Toshio Matsuda, Tohru Masukawa, Satoru Yamagami, Yutaka Kōyama, Itaru Yamamoto, Eibai Lee, Tadahiro Shikimi, Akira Ohta and Tohru Tatsuno and has published in prestigious journals such as Brain Research, Biochemical and Biophysical Research Communications and Journal of Neurochemistry.

In The Last Decade

Heitaroh Iwata

100 papers receiving 1.2k citations

Peers

Heitaroh Iwata

CMN

BIOCH

PHYSI

M. Ledig France

Jörg Lindenau Germany

J. E. Cremer United Kingdom

Rainer Fried United States

Carlo De Marco Italy

Mohammad I. Sabri United States

Pierre‐Marie Sinet France

Jill E. Cremer United Kingdom

K.‐F. R. Sheu United States

Palur G. Gunasekar United States

M. Ledig France

Heitaroh Iwata

447 ×0.9

341 ×0.9

CMN

134 ×0.5

BIOCH

273 ×1.2

PHYSI

160 ×0.8

Citations per year, relative to Heitaroh Iwata Heitaroh Iwata (= 1×) peers M. Ledig

Countries citing papers authored by Heitaroh Iwata

Since Specialization

Citations

This map shows the geographic impact of Heitaroh Iwata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heitaroh Iwata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heitaroh Iwata more than expected).

Fields of papers citing papers by Heitaroh Iwata

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Heitaroh Iwata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heitaroh Iwata. The network helps show where Heitaroh Iwata may publish in the future.

Co-authorship network of co-authors of Heitaroh Iwata

This figure shows the co-authorship network connecting the top 25 collaborators of Heitaroh Iwata. A scholar is included among the top collaborators of Heitaroh Iwata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Heitaroh Iwata. Heitaroh Iwata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kōyama, Yutaka, Tadashi Ishibashi, Heitaroh Iwata, & Akemichi Baba. (1993). Pretreatment of rat brain slices with ouabain decreases chloride-dependent l-glutamate transport in synaptic membrane. Neurochemistry International. 23(2). 149–155. 2 indexed citations

Baba, Akemichi, et al.. (1991). Membrane-associated thiamine triphosphatase in rat skeletal muscle. International Journal of Biochemistry. 23(10). 1111–1114. 9 indexed citations

Kōyama, Yutaka, Tomosada Sugimoto, Yoshio Shigenaga, Akemichi Baba, & Heitaroh Iwata. (1991). A morphological study on glutamate-induced swelling of cultured astrocytes: Involvement of calcium and chloride ion mechanisms. Neuroscience Letters. 124(2). 235–238. 28 indexed citations

Baba, Akemichi, et al.. (1991). NMDA induces protein kinase C translocation in hippocampal slices of immature rat brain. Neuroscience Letters. 126(2). 119–122. 22 indexed citations

Kōyama, Yutaka, Akemichi Baba, & Heitaroh Iwata. (1991). l-Glutamate-induced swelling of cultured astrocytes is dependent on extracellular Ca2+. Neuroscience Letters. 122(2). 210–212. 37 indexed citations

Baba, Akemichi, et al.. (1991). Inhibition of NMDA-induced protein kinase C translocation by a Zn2+ chelator: Implication of intracellular Zn2+. Brain Research. 557(1-2). 103–108. 28 indexed citations

Baba, Akemichi, et al.. (1991). NMDA Induces Protein Kinase C Translocation in Guinea Pig Cerebral Synaptoneurosomes.. The Japanese Journal of Pharmacology. 56(3). 287–296. 8 indexed citations

Ishihara, Takafumi, et al.. (1990). Effect of diphenylthiocarbazone (dithizone) on glutamate level in hippocampus preparation in vitro and in vivo.. Journal of Pharmacobio-Dynamics. 13(4). 225–230. 2 indexed citations

Baba, Akemichi, et al.. (1990). Inhibition of (3H)glutamate release by Zn2+ in rat hippocampal slices.. Journal of Pharmacobio-Dynamics. 13(5). 321–326. 4 indexed citations

10.

Kōyama, Yutaka, Akemichi Baba, & Heitaroh Iwata. (1990). Characteristics of Cl?-dependentl-[35S]cysteic acid transport into rat brain synaptic membrane vesicles. Neurochemical Research. 15(12). 1153–1158. 7 indexed citations

11.

Matsuda, Toshio, Tomoyuki Kanda, Yeon Hee Seong, Akemichi Baba, & Heitaroh Iwata. (1990). p-Chlorophenylalanine attenuates the pituitary-adrenocorticol response to 5-HT1A receptor agonists in mice. European Journal of Pharmacology. 181(3). 295–297. 22 indexed citations

12.

Iwata, Heitaroh, Yutaka Kōyama, & Akemichi Baba. (1989). Depolarization Increases Chloride‐Dependent Glutamate Sequestration in Synaptic Membranes of Rat Cerebral Cortex. Journal of Neurochemistry. 52(2). 354–359. 10 indexed citations

13.

Matsuda, Toshio, et al.. (1989). Tissue difference in cellular localization of thiamine phosphate esters. Comparative Biochemistry and Physiology Part B Comparative Biochemistry. 94(2). 405–409. 15 indexed citations

14.

Matsuda, Toshio, et al.. (1989). Postnatal Development of Thiamine Metabolism in Rat Brain. Journal of Neurochemistry. 52(3). 842–846. 18 indexed citations

15.

Iwata, Heitaroh, Chuzo Iwata, & Toshio Matsuda. (1988). Difference between two isozymes of (Na++K+)-ATPase in the interaction with omeprazole.. The Japanese Journal of Pharmacology. 46(1). 35–42. 9 indexed citations

16.

Iwata, Heitaroh, et al.. (1987). Neural Regulation of Taurine Transport in Skeletal Muscle. Advances in experimental medicine and biology. 217. 199–205. 3 indexed citations

17.

Iwata, Heitaroh, et al.. (1986). Regulation of Taurine Transport in Rat Skeletal Muscle. Journal of Neurochemistry. 47(1). 158–163. 38 indexed citations

18.

Iwata, Heitaroh, et al.. (1981). Involvement of tissue sulfhydryls in the formation of a complex of methylmercury with selenium. Biochemical Pharmacology. 30(23). 3159–3163. 36 indexed citations

19.

Baba, Akemichi, et al.. (1978). Reversible inhibition of adenylate cyclase activity of rat brain caudate nucleus by oxidized glutathione. Biochemical and Biophysical Research Communications. 85(3). 1204–1210. 32 indexed citations

20.

Fujimoto, Seigo & Heitaroh Iwata. (1975). Studies on the prevention of cardiotoxic effect of ouabain by taurine. Folia Pharmacologica Japonica. 71(1). 53–59. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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