Héctor Perkal
- Pathology and Forensic Medicine top 2%
- Neurology top 5%
- Rheumatology top 5%
- Molecular Biology
- Oncology
- Co-authors
- Xavier MontalbánManuel ComabellaÀlex RoviraMar TintoréJordi RíoCarlos NosJaume Sastre‐GarrigaCarmen Tur
- Topics
- Multiple Sclerosis Research Studies (10 papers)Dementia and Cognitive Impairment Research (4 papers)Peripheral Neuropathies and Disorders (3 papers)
- Partner nations
- SpainUnited StatesSwitzerland
In The Last Decade
Héctor Perkal
19 papers receiving 858 citations
Peers
Comparison fields: 5 of 68
- Pathology and Forensic Medicine 617
- Neurology 286
- Rheumatology 205
- Molecular Biology 171
- Oncology 163
Countries citing papers authored by Héctor Perkal
This map shows the geographic impact of Héctor Perkal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Héctor Perkal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Héctor Perkal more than expected).
Fields of papers citing papers by Héctor Perkal
This network shows the impact of papers produced by Héctor Perkal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Héctor Perkal. The network helps show where Héctor Perkal may publish in the future.
Co-authorship network of co-authors of Héctor Perkal
This figure shows the co-authorship network connecting the top 25 collaborators of Héctor Perkal. A scholar is included among the top collaborators of Héctor Perkal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Héctor Perkal. Héctor Perkal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 15 | |
| 2 | 63 | |
| 3 | 57 | |
| 4 | 8 | |
| 5 | 55 | |
| 6 | 7 | |
| 7 | 79 | |
| 8 | 5 | |
| 9 | 10 | |
| 10 | 14 | |
| 11 | 173 | |
| 12 | 19 | |
| 13 | 50 | |
| 14 | [Acute disseminated encephalomyelitis: study of factors involved in a possible development towards multiple sclerosis]. | 2 |
| 15 | 18 | |
| 16 | 244 | |
| 17 | 15 | |
| 18 | [Spanish adaptation of the disease-specific questionnaire MSQOL-54 in multiple sclerosis patients]. | 20 |
| 19 | 19 |
About Héctor Perkal
Héctor Perkal is a scholar working on Pathology and Forensic Medicine, Rheumatology and Cancer Research, having authored 19 papers that have together received 873 indexed citations. Recurring topics across this work include Multiple Sclerosis Research Studies (10 papers), Dementia and Cognitive Impairment Research (4 papers) and Peripheral Neuropathies and Disorders (3 papers). The work is most often cited by research in Pathology and Forensic Medicine (617 citations), Neurology (286 citations) and Rheumatology (205 citations). Héctor Perkal has collaborated with scholars based in Spain, United States and Switzerland. Frequent co-authors include Xavier Montalbán, Manuel Comabella, Àlex Rovira, Mar Tintoré, Jordi Río, Carlos Nos, Jaume Sastre‐Garriga, Carmen Tur, R. Pelayo and N. Téllez. Their work appears in journals such as Neurology, Annals of Neurology and Journal of the Neurological Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.